packet 6 Flashcards

1
Q

Overall function of cardiovascular system and blood flow in general is affected by

A

Blood Pressure
Resistance
Venous Return
Velocity and Volume

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2
Q

force exerted by circulating blood on the walls of blood vessels

A

blood pressure

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3
Q

systolic - diastolic

important measure of stress exerted on small arteries

A

pulse pressure

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4
Q

Factors that determine systemic vascular resistance

A

Blood viscosity
Vessel length
Radius
Laminar flow

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5
Q

7-13 mm Hg venous pressure towards heart

A

Pressure gradient

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6
Q

inhalation - thoracic cavity expands (pressure ) abdominal pressure , forcing blood upward
central venous pressure fluctuates

A

thoracic pump

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7
Q

Exercise increase venous return in many ways

A

heart beats faster, harder - increase cardiac output (CO) and BP
vessels of skeletal muscles, lungs and heart dilate increase flow
increase respiratory rate increase action of thoracic pump
increase skeletal muscle pump

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8
Q

venous pressure not enough force blood upward

with prolonged standing, CO may be low enough to cause dizziness or syncope the following cause

A

venous pooling

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9
Q

Vasoconstriction of blood vessel smooth muscle is controlled both by

A

the autonomic nervous system and at the local level (sympathetic increase parasympathetic decrease)

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10
Q

Four factors control arterial flow at the organ level (includes body position):

A

change in metabolic activity

  • changes in blood flow
  • stretch of arterial smooth muscle
  • local chemical messengers
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11
Q

Blood pressure is a key factor for providing blood (oxygen and energy)

A

to organs

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12
Q

Systolic pressure must be at least 70 to sustain

A

kidney

filtration and adequate blood flow to the brain

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13
Q

Role of Cardiovascular center

A

Collection of nuclei that affect rate, force, vessel diameter

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14
Q

Signals sent out through vagus (parasympathetic)

A

lowers heart rate

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15
Q

help regulate heart rate & stroke volume

specific neurons regulate blood vessel diameter - sympathetic vasomotor nerves

A

nuclei

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16
Q

carotid and aortic sinuses sense the blood pressure in the aortic arch and internal carotid  send signal to the vasomotor center in the medulla oblongata
Other information are sent from the hypothalamus, cortex

A

Baroreceptors

17
Q

Effects help maintain the blood flow to the brain at arterial pressure too low to activate the baroreceptors.

activated by hypoxia, hypocapnia, and acidosis are located in the aortic arch and carotid sinus.
Stimulated during asphyxia and severe hypotension.
Changes breathing, and include sympathetic constriction of (mainly skeletal muscle) arterioles and tachycardia resulting indirectly from the increased lung inflation.

A

chemoreceptors

18
Q

Monitor blood pressure and take corrective action if it should drop.
Leads to a cascade of events eventually producing angiotensin II

A

kidney

19
Q

constricts the walls of arterioles closing down capillary beds;
stimulates the proximal tubules in the kidney to reabsorb sodium ions;
stimulates the adrenal cortex to release aldosterone. Aldosterone causes the kidneys to reclaim still more sodium and thus water
stimulates the pituitary to release the ADH

A

Angiotensin II

20
Q

A rise in blood pressure stretches the atria of the heart, triggers release of atrial natriuretic peptide (ANP).
ANP lowers blood pressure
The effects on the kidney reduce the reabsorption of water = increase flow of urine - the net effect = reduce blood pressure by reducing the volume of blood in the system.

A

heart

21
Q

increases heart rate & force of contraction
causes vasoconstriction in skin & abdominal organs
vasodilation in cardiac & skeletal muscle

A

Epinephrine & norepinephrine

22
Q

is ability to make these changes as needed by demand for O2 & waste removal

A

autoregulation

23
Q

released from cells alter vessel diameter
(K+, H+, lactic acid, NO)
systemic vessels dilate in response to low levels of O2
pulmonary vessels constrict in response to low levels of O2

A

vasoactive substance

24
Q

cause changes in each capillary bed
autoregulation is ability to make these changes as needed by demand for O2 & waste removal
important for tissues that have major increases in activity (brain, cardiac & skeletal muscle)

A

local factors

25
Q

Constriction of arterioles leading to resistance in blood flow and strain on the heart
chronic resting BP > 140/90

A

hypertension

26
Q

chronic low resting BP

caused by blood loss, dehydration, anemia

A

hypotension

27
Q

Any state where cardiac output insufficient to meet metabolic needs (inadequate cardiac output = not enough O2 to meet metabolic needs)

A

shock and homeostasis

28
Q

inadequate pumping of heart

A

cardiogenic shock

29
Q

low venous return (LVR) shock - 3 principle forms

A
  1. hypovolemic shock
  2. obstructed shock
  3. venous pooling
30
Q

most common

loss of blood volume: trauma, burns, dehydration

A

hypovolemic shock

31
Q

tumor or aneurysm

A

obstructed venous return shock

32
Q

long periods of standing, sitting or widespread vasodilation

A

venous pooling

33
Q

loss of vasomotor tone, vasodilation, causes from emotional shock to brainstem injury

A

neurogenic shock

34
Q

bacterial toxins trigger vasodilation and increase capillary permeability

A

septic shock

35
Q

severe immune reaction to antigen, histamine release, generalized vasodilation, increase capillary permeability

A

anaphylactic shock

36
Q
An extremely low blood pressure
Fast but weak pulse
Dizziness, faintness or 
      light-headedness
Moist, clammy skin
Profuse sweating
Unconsciousness
Rapid, shallow breathing 
Chest pain
Blue lips and fingernails
A

signs and symptoms of shock