Lecture 25 and 26: Poultry 2 and 3 Flashcards

1
Q

What type of virus is fowl pox

A

poxvirus, aviopoxvirus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

what happens with the wet form of fowl pox

A

proliferative ulcerative stomatitis, laryngitis, esophagitis, tracheitis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

what are some signs of wet form of fowl pox

A

Dysphagia, aspiration, suffocation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

what are some ddx for wet form of fowl pox

A

candidiasis, trichomoniasis, hypovitaminosis A, capillariasis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

what wrong with dry form of fowl pox n

A

proliferative crusting dermatitis, blepharitis of unfeathered regions

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

what are some signs of dry form of fowl pox

A

impede vision, smell and oral function

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

notice the following lesions and Bolinger bodies on histo. What is likely dx

A

wet and dry forms of fowl pox, avipoxvirus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

what type of virus is infectious laryngotracheitis

A

alphaherpesvirus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What are some signs of infectious laryngotracheitis

A

dyspnea, fibrinonecrohemorrhagic tracheitis +/- pseudo membranes, tracheal and glottis occlusion by exudate

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

T or f: infectious laryngotracheitis is reportable

A

true

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

what wrong

A

Infectious laryngotracheitis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

what type of virus is infectious bronchitis

A

coronavirus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

what are some signs of infectious bronchitis

A
  1. Respiratory- serous tracheitis, airsacculitis, pneumonia
  2. Reproduction- thin albumen, wrinkled eggs
  3. Renal: pallor/swelling leading to urolithiasis and visceral gout
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

what wrong

A

Infectious bronchitis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

t or f: infectious bronchitis is reportable

A

true

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

what type of virus is infectious bursal disease

A

birnavirus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

infectious bursal disease is highly contagious in __week old birds, __morbidity, __ mortality

A

3-6 weeks old, high morbidity, moderate- high mortality

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

what is MOA of infectious bursal disease

A

destroys B lymphocytes of bursa of fabricous—> immunosuppression

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

what are some signs of infectious bursal disease

A

necrohemorrhagic edematous bursitis, hemorrhages of thighs and pectoral muscles

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

what is Subclinical form of infectious bursal disease characterized by

A

atrophied bursa= immunosuppression

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

what type of virus is Newcastle disease

A

paramyxovirus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

what are the 3 virulent strains of Newcastle disease and relative mortality

A

lentogenic- negligible mortality
Mesogenic <10% mortality
Velogenic 90-100% mortality

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

what are some signs of Velogenic Newcastle disease

A

neurologic signs, hemorrhages, and edema, hemorrhage and necrosis of Cecal tonsils

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

t of f: new castle disease is zoonotic and reportable

A

true (Velogenic- reportable)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

what wrong and what caused

A

Cecal tonsil hemorrhages
Cause: new castle disease

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

what is cause of avian cholera: fowl cholera

A

pasturella multocida

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

t or f: avian cholera is not reportable

A

false

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
28
Q

avian cholera may cause __ and __ in large numbers

A

septicemia and death

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
29
Q

what are the per acute, acute, and chronic signs of avian cholera/ fowl cholera

A

per acute: no gross lesions
Acute: necrohemorrhagic hepatitis, hemorrhages, salpingitis, pleuropneumonia
Chronic: fibrinoheterophilic exudate in wattles, joints, hocks, egg yolk peritonitis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
30
Q

Tons of birds died on property, necropsies showed the following signs- what wrong and what likely cause

A

hepatic necrosis, exudate in wattles, pleuropneumonia
Cause: avian cholera: fowl cholera (Pasturella multocida)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
31
Q

what does FESSE stand for in colibacillosis

A

F: fibrinoheterophilic inflammation
E: environmental disease
S: secondary: look for primary cause or husbandry issue
S: septicemia: polyserositis, omphalitis, salpingitis, hypopyon
E: E. Coli

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
32
Q

what could cause the following signs

A

E. Coli

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
33
Q

clostridial diseases effect __ or __ and cause __ inflammation

A

skin or intestines, necrotizing inflammation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
34
Q

C. Septicum and C. Perfringes in chickens causes what

A

necrotizing dermatitis (skin)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
35
Q

what is pathogenesis of C. Septicum and C. Perfringes causing necrotizing dermatitis in chickens

A

broilers overcrowded, wet little—> skin trauma—> environmental bacteria invade

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
36
Q

C. Septicum and C. Perfringes causes what in turkeys

A

gangrenous dermatitis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
37
Q

what is pathogenesis of C. Septicum and C. Perfringes causing gangrenous dermatitis in turkeys

A

predisposed by large clostridia loads—> wet environments and host gut

Cannibalism may spread

38
Q

what does C. Colinum cause in bobwhite quail

A

ulcerative enteritis
SI ulcers
Hepatitis

39
Q

what is pathogenesis of C. Colinum causing ulcerative enteritis in bobwhite quail

A

horizontal transmission via feces or flies

40
Q

what is mortality rate of C. Colinum in bobwhite quail

A

100% mortality in 2 weeks

41
Q

what does C. Perfringes cause in chickens and turkeys

A

necrotic enteritis, fibrinonecrotizing enteritis

42
Q

which clostridial disease causes “Turkish towel”

A

C. Perfringes

43
Q

what wrong and what could have caused

A

necrotizing dermatitis
C. Perfringes or C. Septicum

44
Q

turkey- what wrong and what could have caused

A

Gangrenous dermatitis, C. Perfringes and C. Septicum

45
Q

bobwhite quail, what wrong and what could have caused

A

SI ulcers, hepatitis
C. Colinum

46
Q

what wrong and what cause

A

Turkish towel
C. Perfringes

47
Q

What are some signs of E. Rhusiopathie

A

widespread hemorrhage, endocarditis, swelling of head, fibrinous arthritis

48
Q

t or f: E. Rhusiopathie is zoonotic

49
Q

what wrong and what pathogenesis

A

footpad dermatitis
Husbandry issue—> repeated plantar damage—> bacteria invade—> pododermatitis—> osteomyelitis, arthritis, cellulitis, bacteremia

50
Q

what systems does mycoplasmosis affect

A

joints and respiratory

51
Q

which mycoplasma is reportable

A

Mycoplasma galliseptoicum

52
Q

which mycoplasma infects turkeys

A

mycoplasma melagridis

53
Q

what wrong and what could have caused these lesions

A

Swelling of infraorbital sinuses, airsacculitis, swelling of joints

Mycoplasma

54
Q

who does avibacterium paragallinarium infect

55
Q

what are some signs of avibacterium paragallinarium

A

decreased egg production, hyporexia, oculonasal discharge, face/eyelid edema, intraorbital sinusiits

56
Q

perform necropsy and thick, fowl smelling pus comes out what is likely cause

A

avibacterium paragallinarium

57
Q

what salmonella causes bacillary white diarrhea

A

salmonella pullorum

58
Q

Salmonella pullorum often fatal in __

A

young birds

59
Q

what are some lesions associated with salmonella pullorum

A

Cecal plugs (fibrinonecrotizing typhilitis)

60
Q

with salmonella disease is reportable

A

S. Pullorum

61
Q

what salmonella causes bronze liver in adults

A

salmonella gallinarium

62
Q

what wrong and what could have caused

A

Cecal plug
Salmonellosis pullorum

63
Q

what wrong and what could have caused

A

bronze liver
Salmonella gallinarium

64
Q

what is cause or “raccoon eye” in turkeys

A

bordetella avium

65
Q

what are some signs of raccoon eye

A

voice change/loss, snickering/flicking, mucous exudate, tracheitis, periocular feathers matted

66
Q

what likely cause

A

Bordetella avium (raccoon eye)

67
Q

Which of the following are more likely to be from mites vs lice

A

right- mites- extremity of feather
Left- lice- base of feather

68
Q

what is ascariasis

A

infection with mites

69
Q

What caused these lesions

A

scales leg mite: knemidocoptes mutants

70
Q

knemidocoptes mutants targets __birds

A

older bites

71
Q

how is knemidocoptes mutants transmitted

A

horizontal transmission via direct contact

72
Q

what are some signs of knemidocoptes mutants

A

lameness, impedes joint flexion, toe amputation

73
Q

What these

A

chigger mites

74
Q

what capillarium requires indirect life cycle via earthworm and therefore less likely to be problem in indoor housing

A

C. Annulata

75
Q

what this

A

capillariasis

76
Q

Trachea- what these

A

syngamus trachea (gape worms)

77
Q

what are some signs of syngamus trachea

A

dyspnea, coughing, gasping, head shaking, death due to suffocation, catarrhal tracheitis

78
Q

what is the most common and most expensive disease of poultry

A

coccidiosis

79
Q

where does eimeria tenella live and what does it cause

A

colon and cecum
Necrohemorrhagic typhilitis/colitis

80
Q

what caused this

A

eimeria tenella

81
Q

heterakis gallinarium is __worm of __ and __

A

cecal worm of chickens and turkeys

82
Q

heterakis gallinarium is important vector for

A

histamines meleagridis

83
Q

what species have high vs low susceptibility to histomonas meleagridis

A

high: turkeys
Low: chickens

84
Q

what does histmonas meleagridis cause

A

necrotizing hepatitis, and fibrinonecrotizing typhilitis

85
Q

what type of inflammation does aspergillosis/ A. Fumigates cause

A

fibrinonecrocaseous granulomatous airsacculitis

86
Q

what is likely cause of these lesions

A

Aspergillus

87
Q

what wrong

A

riboflavin deficiency, hypovitaminosis B2

88
Q

what are some signs of vitamin E deficiency/ crazy chick disease

A
  1. Cerebellar encephalomalacia
  2. Muscle dystrophy
  3. Exudative diathesis
89
Q

what wrong and what could have cause this

A

cherry cerebellum
Cause: vitamin E deficiency

90
Q

what wrong and what pathogenesis

A

ascites syndrome, hydropericardium

Rapid growth of muscle develop but low lung volume: body weight ratio—> large muscles demand O2 and resp. Can’T keep up—> increase cardiac output to increase pulmonary perfusion—> pulmonary hypertension—> increase workload of RV—> RV dilates and fails—> RSHF—> increase venous pressure and hydrostatic pressure—> ascites, hydropericardium, pulmonary edema, cyanosis—> death by suffocation

91
Q

What wrong and what pathogenesis

A

deep pectoral myopathy/ green muscle dz

Birds flapping wings a lot—> swelling of supracoracoideus muscle—> increase pressure on fascial sheath and dorsal sternum—> vascular occlusion—> decrease vascular perfusion—> ischemic necrosis

92
Q

what is wrong and what are two possible pathogenesis

A

Fatty liver hemorrhagic syndrome

Path #1: hepatic lipidosis—> hepatocellular vacuole swelling—> disrupt reticulum structure of hepatic plates—> sinusoidal hemorrhage—> hepatic rupture—> fatal hemocoleom

Path #2: hepatocellular swelling—> excessive lipid metabolism—> free radical mediated destruction of reticulum—> sinusoidal hemorrhage—> hepatic rupture—> fatal hemocoleom