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NUR 326 > Inflammatory Disorders GI > Flashcards

Inflammatory Disorders GI Flashcards

1
Q

acute gastritis

A

Temporary inflammation of stomach lining only

Lasts 2-10 days

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2
Q

cause of acute gastritis

A

Irritating substances – alcohol
Drugs, NSAIDS– prostaglandin blocker
Infectious agents – H pylori

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3
Q

chronic gastritis

A

Progressive disorder with chronic inflammation of stomach

Lasts weeks to years

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4
Q

complications of chronic gastritis

A

Peptic ulcer disease
Bleeding
Ulcers
Anemia
Gastric cancers

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5
Q

cause of chronic gastritis

A

Auto immune – attacks parietal cells
H pylori infection

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6
Q

T/F chronic gastritis is a rapid process

A

False, progressive disorder

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7
Q

H. pylori infection

A

Helicobacter pylori bacterium
Gram-negative, spiral

Destructive pattern of persistent inflammation

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8
Q

what kind of environment does H. pylori thrive in?

A

Acidic

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9
Q

how is H. pylori transmitted

A

Person to person saliva
Fecal matter
Vomit
Contaminated food/water

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10
Q

s/sx gastritis

A

Sometimes asymptomatic
Anorexia, anemia
N/V
Post prandial discomfort – after eating
Intestinal gas
Hematemesis
Tarry stool

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11
Q

acute gastroenteritis

A

Inflammation of stomach and small intestine

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12
Q

cause of acute gastroenteritis

A

Viral infections – Norovirus, rotavirus
Bacterial infection – E. coli, salmonella, Campylobacter
Parasitic infections

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13
Q

how long can a cute gastroenteritis last?

A

Normally one to three days
But can last up to 10 days

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14
Q

s/sx acute gastroenteritis

A

Watery diarrhea – bloody or bacteria
Abdominal pain
N/V
Fever, Malaise

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15
Q

t/f fluid volume excess is a complication of acute gastroenteritis

A

False, fluid volume deficit

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16
Q

peptic ulcer disease

A

Ulcerative disease of upper G.I.

Can occur in the esophagus, stomach, duodenum

17
Q

how does peptic ulcer disease develop?

A

When GIs exposed to increased acidity in H. pylori infection

18
Q

what is the stomach’s defensive factors?

A

Mucus
Bicarbonate
Blood flow
Prostaglandins

19
Q

what are the aggressive factors?

A

H pylori infection
NSAIDS
Acid/pepsin
Smoking

20
Q

Cause of peptic ulcer disease

A

H. pylori
Injury causing substances – NSAIDS, aspirin, alcohol
Excess secretion of acid
Smoking
Family history

21
Q

can stress cause peptic ulcer disease?

A

No, but stress increase is gastric acid secreted with a stress response

22
Q

risk factors for peptic ulcer disease

A

Increased NSAID use – induced peptic, ulcer disease
Increase age
History of Peptic ulcer disease
corticosteroid and anticoagulants
Serious stomach diseases – auto immune
H. pylori infection

23
Q

pathogenesis of peptic ulcer disease

A

Mucus is damaged
Histamine is secreted – increase acid and pepsin secretion, further damage
vasodilation – causes edema
If blood vessels are destroyed – bleeding

24
Q

duodenal ulcer

A

Most common type
Occurs at any age, early adulthood

25
Q

gastric/peptic ulcer

A

Ages, 50 to 70
Due to increased NSAID use, corticosteroids, anticoagulants, serious systemic diseases

26
Q

s/sx peptic ulcer disease

A

can be asymptomatic
N/V
Anorexia, weight loss
Bleeding
Burning pain

27
Q

what makes the burning pain worse?

A

Occurs in the middle abdomen
Worse with empty stomach

28
Q

gastric symptoms

A

Burning, cramping, gas like
Occurs in epigastrium, back
One to two hours after eating

29
Q

Duodenal symptoms

A

Burning, cramping, gas like
Occurs in epigastrium, back
2 to 4 hours after eating

30
Q

what is the main difference between gastric and duodenal symptoms?

A

Timing of symptoms after eating

31
Q

complications of peptic ulcer disease

A

HOP

hemorrhage – intestines are vascular
Obstruction – scar tissue, strictures
Perforation and peritonitis

NUR 326 (32 decks)

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