Equine Cardio 1 and 2 Flashcards

1
Q

what is the most likely cardiac cause of poor perforamce

A

arrhythmia

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2
Q

what kind of heart issue would require a prolonged halter ECG

A

episodic

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3
Q

what kind of arrhythmias might be considered normal

A

bradyarrhythmia
sinus arrhythmia
AV block

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4
Q

pathological bradycardia < __ bpm

A

24

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5
Q

tx for bradycardia

A

-glucocorticoids
-parasympathomilytic (atropine)
-sympathomimetic (clenbuterol)
-pacemaker

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6
Q

what is the purpose of glucocorticoids in bradycardia

A

reduce inflammation causing it

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7
Q

What is ectopy?

A

premature beats
[either from A, V, or junction]

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8
Q

Which type of ectopy is most concerning? {more likely to cause sudden death}

A

Ventricular

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9
Q

an early beat preceded by a p wave would indicate which type of ectopy

A

atrial

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10
Q

what is the risk of Premature Atrial Complexes

A

triggers AF

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11
Q

which type of ectopy is a premature beat not preceded by p wave

A

Premature Ventricular Complex

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12
Q

morphology of PVC on ECG

A

wide and bizarre

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13
Q

when should a PVC concern you?

A

-Aortic regurg
-Cardiac signs (syncope, collapse)
-PVC are frequent, don’t go away w exercise
-enlarged ventricle

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14
Q

describe atrial fibrillation on ECG (common)

A

-Irregular RR intervals
-Normal QRS
-No p waves

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15
Q

because AF causes not all blood to be emptied from atria, we lose approx __% of cardiac output

A

20

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16
Q

how to tx Atrial fibrillation

A

quinidine sulphate OR
elelctrocardioversion (TVEC)

17
Q

when can you not tx atrial fibrillation

A

if o accept poor performance +
no evidence of ectopy

*most horses get AF bc large hearts w high vagal tone –> decide if its secondary to significant cardiac dx

18
Q

common complications w quinidine sulphate?
(pro: cheap)

A

colic,
D,
tachycardia,
dull

19
Q

describe quinidine sulpahte administration

A

Stomach Tube (indwelling tube) q2hr unti convert

*want IV access

20
Q

Describe TVEC procedure

A

place introducers and catheters
xray
anesthesia
shock

21
Q

name some mechanisms for murmurs

A

Increased velocity
Decreased viscosity
Decreased vessel diameter
Regurg
Abnormal communication

22
Q

Systole vs diastole

A

systole = ventricular ejection
diastole = ventricular filling

23
Q

is aortic regurg a systolic or diastolic murmur?

A

diastolic

24
Q

2 murmus for systole L and for R

A

L: aortic ejection, mitral regurg

R: tricuspid regurg, VSD

25
Q

are long or short murmurs more significant?

A

long (goes all the way thru systole)

26
Q

causes of mitral regurg

A

-athletic
-endocarditis
-fibrosis
-ruptured chordae tendinae

27
Q

consequence of mitral regurg

A

LHS volume overload,

pulmonary edema/ EIPH

28
Q

what is the most common site for valve pathology

A

aorta

29
Q

is aortic murmur short or long

A

long

30
Q

what might lead to aortic regurg

A

degenerative lesions
endocarditis
VSD

31
Q

aortic regurg can cause __ sided volume overload

A

LV

32
Q

does bounding pulse indicate severity in aortic regurg

A

yes

33
Q

describe pathogenesis of severe aortic insufficiency

A

-LV overload

-increased systolic pressure

-decreased diastolic pressure

-increased difference –> hyperkinetic pulse

34
Q

other cardiac sequelae to aortic regurg

A

ventricular arrhythmias

mitral insufficiency

35
Q

key difference between pathological and physiolgical murmurs

A

pathological varies more - gets worse w exercise

36
Q

AV valve regurg - does intensity correlate w degree?

A

yes

37
Q

VSD - does intensity correlate w degree?

A

no

38
Q

how to distinguish TR from VSD when you hear heart murmur on RHS

A

TR in athletic horses

VSD in ponies

*but scan to check