Equine Cardio 1 and 2 Flashcards

1
Q

what is the most likely cardiac cause of poor perforamce

A

arrhythmia

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2
Q

what kind of heart issue would require a prolonged halter ECG

A

episodic

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3
Q

what kind of arrhythmias might be considered normal

A

bradyarrhythmia
sinus arrhythmia
AV block

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4
Q

pathological bradycardia < __ bpm

A

24

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5
Q

tx for bradycardia

A

-glucocorticoids
-parasympathomilytic (atropine)
-sympathomimetic (clenbuterol)
-pacemaker

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6
Q

what is the purpose of glucocorticoids in bradycardia

A

reduce inflammation causing it

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7
Q

What is ectopy?

A

premature beats
[either from A, V, or junction]

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8
Q

Which type of ectopy is most concerning? {more likely to cause sudden death}

A

Ventricular

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9
Q

an early beat preceded by a p wave would indicate which type of ectopy

A

atrial

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10
Q

what is the risk of Premature Atrial Complexes

A

triggers AF

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11
Q

which type of ectopy is a premature beat not preceded by p wave

A

Premature Ventricular Complex

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12
Q

morphology of PVC on ECG

A

wide and bizarre

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13
Q

when should a PVC concern you?

A

-Aortic regurg
-Cardiac signs (syncope, collapse)
-PVC are frequent, don’t go away w exercise
-enlarged ventricle

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14
Q

describe atrial fibrillation on ECG (common)

A

-Irregular RR intervals
-Normal QRS
-No p waves

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15
Q

because AF causes not all blood to be emptied from atria, we lose approx __% of cardiac output

A

20

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16
Q

how to tx Atrial fibrillation

A

quinidine sulphate OR
elelctrocardioversion (TVEC)

17
Q

when can you not tx atrial fibrillation

A

if o accept poor performance +
no evidence of ectopy

*most horses get AF bc large hearts w high vagal tone –> decide if its secondary to significant cardiac dx

18
Q

common complications w quinidine sulphate?
(pro: cheap)

A

colic,
D,
tachycardia,
dull

19
Q

describe quinidine sulpahte administration

A

Stomach Tube (indwelling tube) q2hr unti convert

*want IV access

20
Q

Describe TVEC procedure

A

place introducers and catheters
xray
anesthesia
shock

21
Q

name some mechanisms for murmurs

A

Increased velocity
Decreased viscosity
Decreased vessel diameter
Regurg
Abnormal communication

22
Q

Systole vs diastole

A

systole = ventricular ejection
diastole = ventricular filling

23
Q

is aortic regurg a systolic or diastolic murmur?

24
Q

2 murmus for systole L and for R

A

L: aortic ejection, mitral regurg

R: tricuspid regurg, VSD

25
are long or short murmurs more significant?
long (goes all the way thru systole)
26
causes of mitral regurg
-athletic -endocarditis -fibrosis -ruptured chordae tendinae
27
consequence of mitral regurg
LHS volume overload, pulmonary edema/ EIPH
28
what is the most common site for valve pathology
aorta
29
is aortic murmur short or long
long
30
what might lead to aortic regurg
degenerative lesions endocarditis VSD
31
aortic regurg can cause __ sided volume overload
LV
32
does bounding pulse indicate severity in aortic regurg
yes
33
describe pathogenesis of severe aortic insufficiency
-LV overload -increased systolic pressure -decreased diastolic pressure -increased difference --> hyperkinetic pulse
34
other cardiac sequelae to aortic regurg
ventricular arrhythmias mitral insufficiency
35
key difference between pathological and physiolgical murmurs
pathological varies more - gets worse w exercise
36
AV valve regurg - does intensity correlate w degree?
yes
37
VSD - does intensity correlate w degree?
no
38
how to distinguish TR from VSD when you hear heart murmur on RHS
TR in athletic horses VSD in ponies *but scan to check