Cardiovascular Qs Flashcards
summarize the RAS system (diagram on other side)
MAP = (two variables)
CO x Sys Vasc Resistance
hemorrhagic pericarditis vs cardiac tamponade vs Libman Sacks
_HP- s_een in TB: inflammation with blood into pericardial sac
Cardiac tamponade: no inflammation + blood directly into pericardial sac
libman sacks ENDOCARDITIS: SLE associated, fibrin vegetations growing on both sides of mitral and aortic valves
SLE markers
anti dna, anti smith, anti nuclear abs
in a cross sectional CT scan of the abdomin, how can you distinguish the aorta?
it sits directly against the vertebrae.
drug of choice in a patient with hypertension and diabetic nephropathy or heart failure
ACEI to begin with
exacerbates hyperuricemia and gouty arthropathy, can cause hyperglycemia and hyperlipidemia
thiazide and loop diuretics
starling equation, just the order of it
1) capillary mmHg is always first, oncotic (colloidal) is always substracted
2) keep the pressure types together
3) remember its the “pushing” forces minus the “pulling” forces
4) (Hydrostatic - hydrostatic) - (colloid - colloid)- (capillary hystostatic - interstitial hydrostatic) - (colloid - colloid)
golden rule for evaluating the effects of drugs on the heart: blood pressure’s relationship to heart rate
- look at BLOOD PRESSURE first and then HR
- blood vessels: changes on blood vessels will directly result from direct
- heart rate: changes in the heart will be due to indirect baroreceptor reflex or directly from drug interactions
norepinephrine’s effects
alpha 1 agonist on blood vessels–> vasoconstriction —> results increased blood pressure –> baroreceptor reflex –> decrease in heart rate