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4/17 Flashcards

1
Q

Most common cause precocious puberty males

A

CAH

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2
Q

Folicular phase

A

begins 1st day of menses

FSH stimulated ovarion follicle which in turn secretes estradiol

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3
Q

What does estradiol do

A

induces endometrial proliferation, further increasing FSH and LH

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4
Q

What causes ovulation in Luteal phase

A

LH surge

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5
Q

Progesterone effect on FSH and LH

A

decreases

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6
Q

what does hCG do

A

Maintains corpus lutteur and progesterone secretion

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7
Q

Amenorrhea first test

A

B-hcg

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8
Q

first question primary amenorrhea

A

is there secondary sex traits?

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9
Q

Secondary sex traits primary amenorrhea final tests

A

US and genetic analysis to determine anatomy and kayitypes

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10
Q

No secondary sex traits primary amenorrhea next test

A

FSH LH

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11
Q

No secondary sex traits primary amenorrhea low FSH and LH next test and results.

A

check prolactin to see if it’s Prolactinoma or HPA dysfunction(GnRH, etc.)

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12
Q

No secondary sex traits primary amenorrhea high FSH and LH dx

A

Gonadal Agenesis or ovarian failure

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13
Q

Secondary Amenorrhea first test

A

thyroid

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14
Q

2nd amenorrhea normal thyroid next test

A

prolactin

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15
Q

2nd amenorrhea normal thyroid, normal prolactin next test

A

progestin challenge

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16
Q

2nd amenorrhea normal thyroid, normal prolactin, - progesterone challenge, next step

A

Estrogen-progesterone challenge

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17
Q

2nd amenorrhea normal thyroid, normal prolactin, - progesterone challenge, - Estrogen-progesterone challenge dx

A

Ashermans

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18
Q

2nd amenorrhea normal thyroid, normal prolactin, - progesterone challenge, +Estrogen-progesterone challenge next test

A

FSH, LH

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19
Q

2nd amenorrhea normal thyroid, normal prolactin, - progesterone challenge, +Estrogen-progesterone challenge, High FSH and LH dx

A

Ovarian failure

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20
Q

2nd amenorrhea normal thyroid, normal prolactin, - progesterone challenge, +Estrogen-progesterone challenge, Low FSH and LH dx

A

HPA dysfuntion

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21
Q

2nd amenorrhea normal thyroid, normal prolactin, + progesterone challenge, next step

A

Is there hirsutism

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22
Q

2nd amenorrhea normal thyroid, normal prolactin, + progesterone challenge, Hirsutism dx

A

PCOS, Adrenal tumor, Cushing’s syndrome

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23
Q

2nd amenorrhea normal thyroid, normal prolactin, + progesterone challenge, NO Hirsutism dx

A

anorexia,HPA dysfunction

24
Q

most common cause of female infertility

A

endometriosis

25
Q

when is endometriosis pain worst

A

1 to 2 weeks before menses

26
Q

symmetric uterine enlargement AUB, 40+

A

adenomyosis

27
Q

What is increased PCOS LH or FSH

28
Q

Testosterone PCOS

29
Q

Which genital ulcers are painless

A

syphylis and lymphogranularum (lymph nodes are painful for this one also buboes)

30
Q

Which genital ulcers are painful

A

Chancroid, herpes

31
Q

Folicular cyst Origin, characteristics,tx

A

Ovarian folicle
granulose cell, may regress over menses period
Observation

32
Q

Corpus luteum cyst Origin, characteristics,tx

A

Corpus luteum
Theca cells large and firm, more common late weeks of cycle
Observation

33
Q

Mucinous or serous cystadenoma Origin, characteristics,tx

A

Epithelial cells
Psammoma bodies(calcification), may be extremely large
unilateral sapling-oophorectomy

34
Q

endometrioma Origin, characteristics,tx

A

Endometrium
generalized symptoms
OCP, GnRH agonist, cystectomy, oophorectomy

35
Q

teratoma/ dermoid cystOrigin, characteristics,tx

A

germ cells
multiple dermal tissues
cystectomy maybe unilateral sapling-oophorectomy

36
Q

stromal Origin, characteristics,tx

A

Granulosa, theca, ledig
Precocious puberty
unilateral sapling-oophorectomy

37
Q

Ductal Carcinoma insitu characteristics and h&p

A

ducts only, no stromal involvement, usually asymptomatic

38
Q

lobular insitu characteristics and h&p

A

lobules only, increased risk of contralateral malignancy

asymptomatic

39
Q

invasive ductal characteristics and h&p

A

micro calcification, fibrotic response ,most common invasive kind. skin dimple, orange, nipple discharger

40
Q

invasive lobular characteristics and h&p

A

less fibrous response, bilateral or multifocal, associated with hormone replacement same as invasive ductal signs and sx skin dimple, orange, nipple discharger

41
Q

pagets characteristics and h&p

A

adenocarcinoma, scaly eczematous or ulcer on nipple, pain, itching, burning

42
Q

inflammatory characteristics and h&p

A

angioinvasive, poor prognosis, pain, erythema, warmth, orange, lymphadenopathy

43
Q

medullary characteristics and h&p

A

well circumscribed, rapid growth, soft

44
Q

mucinous characteristics and h&p

A

well circumscribed, slow growth, gelatinous

45
Q

tubular characteristics and h&p

A

tubular, asymptomatic

46
Q

okay meds for htn pregnancy

A

labetalol
nifedipine
a-methaldopa
hydralazine- fro crisis

47
Q

where is mg excreted

48
Q

placental abruption clinical presentation

A

sudden onset painful vaginal bleeding.
High frequency low intensity contractions
hypertonic, tender uterus

49
Q

Most common complication chronic hypertension pregnancy

A

preterm labor

50
Q

when to avoid NSAIDS in pregnancy

A

1st and 3rd trimester, acetaminophen is okay

51
Q

what is Leggier Calve Perthes dz

A

osteonecrosis of femoral epiphysis- 7 year old

52
Q

besides sickle shaped what other abnormal rbc sickle cell dz

A

Howell- Jolly- bodies

53
Q

3 aortic stenosis auscultation findings

A
  1. delayed, weak, slow rising carotid pulse(pulses parvus and trades.
    2.single and soft S2
    #. Mid to late peaking systolic murmur
54
Q

why are klinefelters infertile

A

dysgenesis of seminiferous tubules

55
Q

strep bovis association

A

colon cancer

step 2 (53 decks)

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