Traumatic Brain Injury Flashcards

1
Q

Primary and secondary injuries

A

Primary- immediate trauma to brain parenchyma at moment of insult injury
Secondary- results after primary injurt; hypoxia/ischemia, edema, and increased ICP.

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2
Q

MOIs

A

Contact (open vs closed injury)
Acceleration- Deceleration (compression, tension, shearing)
Rotation (angular acceleration).

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3
Q

4 different pathophysiologies

A

Focal
Diffuse axonal
Hypoxia-ischemic
Increased ICP

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4
Q

ICP- normal and elevated levels

A

normal- 5-20 cm H20
elevated- >20
severe- >25 (usually means brain herniation)

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5
Q

Signs of elevated ICP

A

decreased conciousness
altered vital signs
widened pulse pressure
irregular (cheyne-stokes) breathing
vomiting
headache
non-reacting pupils (CN 3)
papilledema (optic disc or nerve swelling)
progressive impairment of motor function
seizure activity

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6
Q

Treatments of elevated ICP

A

Elevate head of bed 30 degs- works immediately

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7
Q

Neuromuscular Impairments

A

Impairments similar to stroke
UMN injury

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8
Q

Cognitive impairments

A

arousal levels
attention
concentration
memory
learning
executive functions

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9
Q

Arousal levels (different states)

A

coma
vegetative state
minimally conscious state

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10
Q

Other terms to describe consciousness

A

Stupor- almost unresponsive state
Obtunded- decreased alertness. Sleeps often

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11
Q

Memory impairments

A

Anomia
Anterograde amnesia
Retrograde amnesia
Post-traumatic amnesia

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12
Q

Neurobehavioral impairments

A

agititaiton, apathy, emotional liability, mental inflexibility, disinhibition, anxiety, aggression, poor self image, sexual apathy etc

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13
Q

Communication impairments

A

aphasia, auditory processing deficits, disorganized communication

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14
Q

Swallowing Impairments

A

Dysphagia common

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15
Q

Dysautonomia

A

increased SNS activity following TBI
Increased HR, RR, BP. diaphoresis and hyperthermia

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16
Q

Visio-perceptual impairments

A

damage to occipital lobe can result in visual impairments
perceptual impairments: apraxia, spatial neglect, somatagnosis

17
Q

Post-traumatic seizures

A

less than half of people with severe TBI develop post-traumatic seizures

18
Q

Heterotopic ossifications

A

boney growth in muscle after injury. Common in proximal body parts

19
Q

Examinations- key areas

A

arousal, attention and cognition
integumentary integrity
sensory integrity
motor function
ROM
reflexes
ventilation and respiration

20
Q

Glasgow Coma Scale- uses

A

measures level of conciousness
helps to determine severity of injury and track progress

21
Q

GCS- scoring (severe, moderate, mild)

A

total score will be from 3-15
severe: <8
moderate: 9-12
mild: >13

22
Q

The three parts of GCS

A

Eye opening
Motor response
Verbal response

23
Q

The moderate level of TBI:
GCS scale
loss of conciousness
altered consciousness
post-traumatic amnesia
neuroimaging

A

GCS scale; 9-12
loss of conciousness; >30 mins and <24hrs
altered consciousness; >24hrs
post-traumatic amnesia; >1 day and <7 days
neuroimaging; normal or abnormal

*mild and severe can be interpretted from these values

24
Q

LOCF

A

Rancho Los Amigos Levels of Cognitive Functioning
descriptive scale used to track cognitive and behavioural recovery as patient emerges from a coma

25
Q

GOAT- Galveston Orientation and Amnesia Test

A

questions include name, city, recall of how patient is, where he or she is, day, date, month, year, and event of injury
helps determining outcome or prognosis

26
Q

Predictors of poor outcomes

A

low initial GCS score
lower education level
very young (<7 yrs old) or older (>40 yrs)
longer periords of post-traumatic amnesia
- <34 days likely to have a good overall recovery

27
Q

PT interventions for TBI

A

primary goal is to prevent secondary implications (due to prolonged immobility)
patient and family education

28
Q

Special considerations for confused and agitated patients

A

Consistency
Expect no carryover
Model calm behavior
Expect egocentricity
Flexibility/ Options
Safety