Traumatic Brain Injury Flashcards

1
Q

Primary and secondary injuries

A

Primary- immediate trauma to brain parenchyma at moment of insult injury
Secondary- results after primary injurt; hypoxia/ischemia, edema, and increased ICP.

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2
Q

MOIs

A

Contact (open vs closed injury)
Acceleration- Deceleration (compression, tension, shearing)
Rotation (angular acceleration).

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3
Q

4 different pathophysiologies

A

Focal
Diffuse axonal
Hypoxia-ischemic
Increased ICP

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4
Q

ICP- normal and elevated levels

A

normal- 5-20 cm H20
elevated- >20
severe- >25 (usually means brain herniation)

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5
Q

Signs of elevated ICP

A

decreased conciousness
altered vital signs
widened pulse pressure
irregular (cheyne-stokes) breathing
vomiting
headache
non-reacting pupils (CN 3)
papilledema (optic disc or nerve swelling)
progressive impairment of motor function
seizure activity

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6
Q

Treatments of elevated ICP

A

Elevate head of bed 30 degs- works immediately

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7
Q

Neuromuscular Impairments

A

Impairments similar to stroke
UMN injury

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8
Q

Cognitive impairments

A

arousal levels
attention
concentration
memory
learning
executive functions

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9
Q

Arousal levels (different states)

A

coma
vegetative state
minimally conscious state

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10
Q

Other terms to describe consciousness

A

Stupor- almost unresponsive state
Obtunded- decreased alertness. Sleeps often

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11
Q

Memory impairments

A

Anomia
Anterograde amnesia
Retrograde amnesia
Post-traumatic amnesia

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12
Q

Neurobehavioral impairments

A

agititaiton, apathy, emotional liability, mental inflexibility, disinhibition, anxiety, aggression, poor self image, sexual apathy etc

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13
Q

Communication impairments

A

aphasia, auditory processing deficits, disorganized communication

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14
Q

Swallowing Impairments

A

Dysphagia common

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15
Q

Dysautonomia

A

increased SNS activity following TBI
Increased HR, RR, BP. diaphoresis and hyperthermia

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16
Q

Visio-perceptual impairments

A

damage to occipital lobe can result in visual impairments
perceptual impairments: apraxia, spatial neglect, somatagnosis

17
Q

Post-traumatic seizures

A

less than half of people with severe TBI develop post-traumatic seizures

18
Q

Heterotopic ossifications

A

boney growth in muscle after injury. Common in proximal body parts

19
Q

Examinations- key areas

A

arousal, attention and cognition
integumentary integrity
sensory integrity
motor function
ROM
reflexes
ventilation and respiration

20
Q

Glasgow Coma Scale- uses

A

measures level of conciousness
helps to determine severity of injury and track progress

21
Q

GCS- scoring (severe, moderate, mild)

A

total score will be from 3-15
severe: <8
moderate: 9-12
mild: >13

22
Q

The three parts of GCS

A

Eye opening
Motor response
Verbal response

23
Q

The moderate level of TBI:
GCS scale
loss of conciousness
altered consciousness
post-traumatic amnesia
neuroimaging

A

GCS scale; 9-12
loss of conciousness; >30 mins and <24hrs
altered consciousness; >24hrs
post-traumatic amnesia; >1 day and <7 days
neuroimaging; normal or abnormal

*mild and severe can be interpretted from these values

24
Q

LOCF

A

Rancho Los Amigos Levels of Cognitive Functioning
descriptive scale used to track cognitive and behavioural recovery as patient emerges from a coma

25
GOAT- Galveston Orientation and Amnesia Test
questions include name, city, recall of how patient is, where he or she is, day, date, month, year, and event of injury helps determining outcome or prognosis
26
Predictors of poor outcomes
low initial GCS score lower education level very young (<7 yrs old) or older (>40 yrs) longer periords of post-traumatic amnesia - <34 days likely to have a good overall recovery
27
PT interventions for TBI
primary goal is to prevent secondary implications (due to prolonged immobility) patient and family education
28
Special considerations for confused and agitated patients
Consistency Expect no carryover Model calm behavior Expect egocentricity Flexibility/ Options Safety