Recitation 3 Test 2 Flashcards
fatty acyl coA is transported to mitochondria via
carnitine, cat 1, cat 2
cat 1 is inhibited by blank
malonyl coA
how many redox reactions exist in fatty acid beta oxidation
2?
dehydrogenase is automatically a blank reaction
redox
fatty acid oxidation makes blank atp
5
mcad deficiency causes you to overburn blank which leads to hypoglycemia
glucose from glycolysis
mcad deficiency also causes blank acidosis
lactic
mcad deficiency is managed with blank
carnitine
propionyl coA is produced by oxidation of blank
branched fatty acid, branched aa, odd chain fatty acid
adenosylcobalamine is basically this vitamin in our body
b12
form of b12 used to treat cyanide poisoning
hydroxocobalamine
phytanic acid is metabolized by blank
alpha oxidation
oxidation of polyunsaturated fatty acids require which two additional enzymes
reductase, isomerase
peroxisomal fatty acid beta oxidation takes very long chain/long chain and turns it into blank fatty acid and eventually undergoes blank
C8, mitochondrial beta oxidation
cholesterol synthesis takes place where
cytosol
breakdown products of TAG
fatty acids (3), glycerol
fatty acid carrier in the circulation and is the most abundant protein in circulation
serum albumin
HSL is blank but LPL is blank
extracellular, intracellular
ketone body is activated for oxidation where
outside liver
fuel for ketone body production
lipids, muscle proteins
driving forces of glycogenolysis, glycogenesis
udp glucose, pyrophosphate
adenylate cyclase is activated by blank
glucagon/epinephrine
adenylate cyclase makes blank
cAMP
insulin downregulates cAMP via blank
phosphodiesterase 3
cAMP is degraded by blank
pde 3
allosteric control of glycogen phosphorylase is blank in diagram
vertical
g6p is the only blank regulator in glycogen phosphorylase
reciprocal
which allosteric regulators coordinate glycolysis, and glycogenolysis in muscle…. this one inhibits these
AMP, ATP
calcium activates glycogen breakdown via calmodulin bound to blank
phosphorylase kinase
pp1 is differentially regulated because blank inhibits it but blank activates pp1
glucagon, insulin
fatty acid activation takes place in the blank and requires blank and requires blank atp per fatty acid for activation
cytosol, cofactors (coA, ATP), 2 atp