Exam 7 Infection Control Flashcards
Bioterrorism
Recognize patterns
Are nurses getting sick?
Similar resistant
When exposed to blood
Wash area w/ soap and water
Clean with soap/alcohol
Call employee health asap
Lab work for you and patient for baseline
fomite
contaminated object that transfers pathogen
Chain of infection
Infectious Agent-Reservoir-Portal of Exit-Mode of Transmission-Portal of Entry- Susceptible Host
Tertiary Defense
Specific Immunity
Cell Mediated Immunity
Humoral Immunity
Secondary Defense
Phagocytosis
Complement cascade
Inflammation
Fever
types of portal of entry
eyes, nares, mouth, urethra, vagina, cuts, scrapes, bites, wounds
Infection Control Nurse
Mini infection in facility
Be current on info on infections
Tracks & reports
Specific Immunity
Lympocytes
T cells/ B cells
Antibodies
direct contact
touching, kissing, sex
portal of entry
opening into body
Surgical Asepsis
Sterile Surgical Scrub Surgical Attire Sterile technique Sterile fields
factors leading to infection
- power to infect
- number of pathogen transmitted
- Host defense
Immune Response
Has Humoral and cellular immunity
susceptible host
person with inadequate defenses such as old or young
CDC Tier Two
Contact, Droplet, & Airborne Precautions
Protective Isolation
reservoir
location of infection
CDC Tier One
Wash hands before/after everthing Never Recap needles Splash precaution with goggles/mask Soil precaution with gown Clean all equipment
Complement Cascade
Blood protein causes pathogen’s cell wall to rupture
Inflammatory Response
Acute-Redness,heat,swelling, pain.
Constrict–>Dilate
Flood with fluid
Phagocytes cover area
Humoral Immunity Antibodies
Phagocytosis
Neutralization
Agglutination
Activation of complete & inflammation
types of pathogens
bacteria, viruses, fungi, protozoa-worms and infectious protein
Support of Immune Defense
Nutrition Hygiene Rest/exercise Stress reduction Immunization
Mode of transmission
direct, animal direct, fomite
Cellular immunity
Inc WBC goes after foreign protein
Normal Flora
Body’s Bacteria - aids in gi, limits growth of bad bacteria, synthesize vit k, b12, thiamine, and riboflavin
Factors that increase risk
Developmental stage Breaks in defense smoking substance abuse illness/injury environment medications procedures multi sex partners
different reservoirs
non-living- soil/water/food/surface
living - animals/insects/humans
carrier
stages of infection
Incubation
Prodromal
Illness
Convalescence
Infection
When micros invade the body
Illness
Appearance of disease; Untreated –>death
Body’s Primary defense
Intact Skin Mucous Cilia Hair in Nose Lysozyme in tears and saliva Acid/Bile Vomiting/Diarrhea
Antigen
Foreign protein
Antibody
Body produces in response to antigen
portal of exit
body fluids, cuts, bites, abrasions
Cell Mediated
T cells Cytotoxic Helper Memory Suppressor
Bodies defenses against infection
Inflammatory and Immune Response
Incubation
Expo to infection
What determines length of illness and convalescence?
Infection
Pathogen
organism capable of disease
Convalescence
s/s begin to subside
tissues getting back to norm
may have temp/perm change
carrier
have pathogen but no s/s of disease
Prodromal
First appearance of symptoms
classification of infection
local- one area systemic-whole body primary-cause secondary-happens after treatment exogenous-other people endogenous-already in body iatrogenic-result from treatment acute-sudden chronic-long term latent-carrier
Prodromal
First appearance of symptoms
Cellular immunity
Inc WBC goes after foreign protein
Normal Flora
Body’s Bacteria - aids in gi, limits growth of bad bacteria, synthesize vit k, b12, thiamine, and riboflavin
carrier
have pathogen but no s/s of disease
Humoral Immunity Antibodies
Phagocytosis
Neutralization
Agglutination
Activation of complete & inflammation
Antibody
Body produces in response to antigen
Chain of infection
Infectious Agent-Reservoir-Portal of Exit-Mode of Transmission-Portal of Entry- Susceptible Host
Incubation
Expo to infection
direct contact
touching, kissing, sex
Pathogen
organism capable of disease
Illness
Appearance of disease; Untreated –>death
CDC Tier Two
Contact, Droplet, & Airborne Precautions
Protective Isolation
Surgical Asepsis
Sterile Surgical Scrub Surgical Attire Sterile technique Sterile fields
susceptible host
person with inadequate defenses such as old or young
Cell Mediated
T cells Cytotoxic Helper Memory Suppressor
Infection Control Nurse
Mini infection in facility
Be current on info on infections
Tracks & reports
Inflammatory Response
Acute-Redness,heat,swelling, pain.
Constrict–>Dilate
Flood with fluid
Phagocytes cover area
fomite
contaminated object that transfers pathogen
Body’s Primary defense
Intact Skin Mucous Cilia Hair in Nose Lysozyme in tears and saliva Acid/Bile Vomiting/Diarrhea
Immune Response
Has Humoral and cellular immunity
Factors that increase risk
Developmental stage Breaks in defense smoking substance abuse illness/injury environment medications procedures multi sex partners
Mode of transmission
direct, animal direct, fomite
Bioterrorism
Recognize patterns
Are nurses getting sick?
Similar resistant
Convalescence
s/s begin to subside
tissues getting back to norm
may have temp/perm change
Secondary Defense
Phagocytosis
Complement cascade
Inflammation
Fever
Antigen
Foreign protein
reservoir
location of infection
What determines length of illness and convalescence?
Infection
stages of infection
Incubation
Prodromal
Illness
Convalescence
portal of exit
body fluids, cuts, bites, abrasions
Support of Immune Defense
Nutrition Hygiene Rest/exercise Stress reduction Immunization
types of pathogens
bacteria, viruses, fungi, protozoa-worms and infectious protein
Bodies defenses against infection
Inflammatory and Immune Response
factors leading to infection
- power to infect
- number of pathogen transmitted
- Host defense
Tertiary Defense
Specific Immunity
Cell Mediated Immunity
Humoral Immunity
CDC Tier One
Wash hands before/after everthing Never Recap needles Splash precaution with goggles/mask Soil precaution with gown Clean all equipment
classification of infection
local- one area systemic-whole body primary-cause secondary-happens after treatment exogenous-other people endogenous-already in body iatrogenic-result from treatment acute-sudden chronic-long term latent-carrier
Specific Immunity
Lympocytes
T cells/ B cells
Antibodies
Complement Cascade
Blood protein causes pathogen’s cell wall to rupture
When exposed to blood
Wash area w/ soap and water
Clean with soap/alcohol
Call employee health asap
Lab work for you and patient for baseline
Infection
When micros invade the body
portal of entry
opening into body
different reservoirs
non-living- soil/water/food/surface
living - animals/insects/humans
carrier
types of portal of entry
eyes, nares, mouth, urethra, vagina, cuts, scrapes, bites, wounds
Infection
When microorganism capable of causing infection invades the body.