Exam 3 Changed Color Notes on Slides *Except Bone & Men/Women Health Flashcards
insulin is always needed for treatment
type 1
managing type one diabetics
insulin
symptoms of type 2 diabetics
usually subtle or no symptoms in early stages
glucose monitoring
- fasting blood glucose
126 or more
glucose monitoring
- random glucose
200 or more
glucose monitoring
- HgbA1C level
greater than 6.5%
goals of A1C
less tha. 7%
five components of diabetic care
nutritional therapy
exercise
monitoring
pharm
education
nutritional therapy
maintain the pleasure of eating include personal and cultural preferences
promote exercise and activity
meal planning
must be considerate of patient preferences
diabetic diet should consist of
50-60% carbs
20-30% fat
10-20% protein
25g/day fiber
what do we do for high glycemic index foods
monitor BS after ingestion of certain foods to help ID personal glycemic index to improve glucose control
precautions of exercise
do not exercise if BS >250 and ketones in urine
may continue when ketones are negative and BS normal
rotation of diabetic injections
systematic rotation of sites within an autonomic area recommended
how much should you use that exact site
not more than once in 2-3 weeks
what about injecting before you exercise
do not inject in limb that will be exercised
metformin
contraindicated in impaired kidney or liver function must be discontinued 48 hours prior to and after CT with contrast
sulfonylureas
increased risk for hypoglycemia with elderly, beta blocker use may decrease or mask ss of hypo
basic survival skills
defintion
normal BS ranges
effect of therapy
treatment modalities
complications
sick day rules
take insulin or oral anti diabetic agent as usual
test blood glucose and urine ketones (every 3-4 hours)
report elevated BS levels or ketones
- increased insulin coverage may be required
take liquids more frequently to prevent dehydration
- sports drink, cola, or broth
consume soft foods (gelatin, soup, graham crackers) six to eight times a day if unable to follow normal diet
report nausea, vomiting, diarrhea to provider
- hospitlization maybe required if unable to keep fluids down
severe hypoglycemia
glucose levels less than 40
mild hypoglcyemia symptoms
SNS stimulation
sweating
tremor
tachycardia
palpation
nervousness
hunger
moderate hypoglycemia symptoms
inability to concentrate
headache
lightheadedness
confusion
memory lapse
numbness of lips and tongue
severe hypo symptoms
disorientation
seizure
difficulty arousal
loss of consiousness
who might have variable response to hypoglycemia
elderly
beta blockers
logn term diabetic
management of hypoglycemia
immediate treatment must be given
check blood sugar first step
emergency measures of hypoglycemia
glucagon 1mg IM or SQ (community)
25-50mL dextrose IVP
- careful assessment of IV site prior to administration and after
DKA manifesations
hyperglycemia
severe dehydration
metabolic acidosis
DKA common causes
missed insulin dose
illness
infection
undiagnosed
DKA assessment findings
blood glucose levels (300-800)
low pH, CO2, bicarb levels
ketones in urine and blood
electrolyte abnormalities
elevated BUN/CR and HCT
management of DKA
rehydration
- 6-10 liters
-0.9NS initially, changed to 0.45 after first few hours of hydration (.45 may be used with hypertensive patients or those with risk of heart failure) changed to D5W when the BS is at 250-300
- monitor fluid status
insulin
- continuous infusion of regular insulin (12-24hr)
-frequent blood sugar monitoring hourly
- IV solution D5 when blood sugar 250-300
electrolyte restoration
- cautious but timely replacement of K is vital
HHS defintion
metabolic disorder of type 2 diabetes resulting from relative insulin deficiency intimated by illness that raises the demand for insulin, more common in older adults
assessment and diagnostic findings of HHS
BS level 600-1200
osmolality greater than 320
HHS treatment
similar to DKA
- rehydration with IV fluids
- insulin administration
- electrolyte replacement
macrovascucualr complication
accelerated atherosclerotic changes
neuropathic complication
peripheral neruopathy
autonomic neruopaties
hypoglycemic unawarness
neuropathy
sexual dysfunction
meticulous foot care
have podiatrist check feet once a year
- toenails inquire about best way to manage toenails
check feet daily
wash feet daily
keep skin soft
do not walk barefoot
protect from hot and cold
steps to lower risk of complications in diabetics
A1C less than 7
take care of your feet
get recommended screenings and early treatment for complications
most common non lymphocytic leukemia
acute myeloid leukemia
acute myeloid leukemia
- treatment
aggressive chemo
induction therapy
chronic myeloid leukemia
- manifesations
initially may be asymptomatic
malaise
anorexia
weight loss
confusion or shortness of breath caused by leukostasis
enlarged tender spleen
enlarged liver
acute lymphocytic leukemia
- manifestations
leukemia cell infiltration is more common with this leukemia
symptoms of meningeal involvement
liver/spleen/bone marrow pain
hodgkin disease treament
determined by stage of the disease and may include chemo, radiation therapy or both
non hodgkin lymphoma treatment
determined by type and stage of disease and may include interferon, chemo, or radiation
multiple myeloma treatment
include chemo, steroids, radiation, bisphosphonates
neutropenia risk
infection
neutropenia nursing management
patient education
- reverse isolation
immune thrombocytopenia purpura
low platelets
don’t know why
ideopathic
high risk bleeding
pin point bleeding
thrombocytopenia
clinical manifesations
increased risk of bleeding
thrombocytopenia
patient safety and education
bleeding
no aspirin
anti platelet
hemophelia, how to get
inherited
hemophilia medical and nursing management
recumbent forms to factor 8 and 10 when bleeding or profilactically
DIC triggers
sepsis
trauma
shock
cancer
abrupto placenta
toxins
allergic reaction
DIC defintion
alerted hemostasis mechanism causes massive clotting in microcirculation
as clotting factors are consumed bleeding occurs
symptoms are related to tissue ischemia and bleeding
DIC treatment
treat underlying cause
correct tissue ischemia
replace fluids and lytes
maintain blood pressure
replace coag factors
use heparin
heparin therapeutic test
aPTT
heparin aPTT time
1.5-2.5 times the lab control
heparin complications
heparin induced thrombocytopenia
warfarin reversal
vit K
warfarin test
INR
warfarin INR therapeutic range
2-3
most common hematological condition
anemia
clinical manifestations of anemia
fatigue
weakness
pallor or jaundice
cardiac and respiratory symptoms
tongue change
nail changes
pica
angular cheilosis
goals of anemia
decreased fatigue
attainment and or maintence of adequate nutrition
maintence of adequate tissues perfusion
compliance with prescribed therapy
absence of complications
most common type of anemia in all age groups
iron deficiencies
iron deficiency’s manifestations
typical presentations
- may have smooth sore tongue
- rigid nails
- angular cheilosis
anemia in renal disease
- occurs in association of a serum CR greater than
3
treatment of anemia in renal disease
recumbent erythropoietin
- epoetin alfa
- Epogen
- Procrit
- Aranesp
what is the best known secondary immunodeficiency in humans
HIV
prevention of HIV
standard precautions
safer sex practices and safer behaviors
do not share drug injection equipment
blood screening and treatment of blood products
prevention of HIV for health care providers
standard precatuions
PPE
post exposure prophylaxis
report it to supervisor
fill out form
labs drawn (baseline)
source testing patients blood
- not for treating patient but for testing for blood borne pathogens
primary infection symptoms
none to flulike symptoms
HIV asymptomatic
- upon reaching the viral set point what state is reached
chronic asymptomatic state begins
AIDS
- CD4 count
less than 200
AIDS
- as levels drop below ________ the immune system is significantly impaired
100
one quarter of people living with HIV are older than
50
treatment and protocols are continually
evolving
PCP if untreated it can progress to
pulmonary impairment and respiratory failure
complications of HIV
PCP
mycobacterium avian complex
tuberculosis
oral candudasis
diarrhea realted to HIV infection or enteric pathogens
wasting syndrome
kaposi sarcoma
B cell lymphoma
HIV encephalopathy
kaposi sarcoma is what
cutaneous lesions that involve multiple organs
lesions cause discomfort, disfigurement, ulcerations and potential for infection
what is included in OTC. herbal, and prescription medications assessment before a transfusion
NSAIDS
ASA/salicylates
steroids
antibiotics
cytotoxic medications
history of transfusions
diagnostic evaluation before transfusion
CBC and coagulation studies
bone marrow aspiration
patient preparation
careful explatinaton
premedication
bone marrow aspiration what to expect
pressure
pain
bone marrow aspiration complications
bleeding and infection
avoid asa products
pre transfusion assessment
history of previous transfusions
history of previous reactions
physical assessment pretransfusion
baseline vitals
lung sounds
JVD
edema
skin assessment (observe for petechiae, rash
s/s of reactions
rash
fever
chills
low back pain
pain at IV site
anything unusual
complications
febrile non hemolytic reactions
acute hemolytic reaction
circulatory overload
how to prevent circulatory overload
administer slowly to high risk patients
circulatory over load S/S
orthopena
JVD
tachycardia
dyspnea
sudden anxiety
crackles in lungs
increase BP
pulmonary edema
- pink frothy sputum
treatment for circulatory overload
upright position
notify MD
oxygen
diuretics
nursing interventions for sickle cell
pain management
infection prevention
generalized seizure
involve whole brain
both sides of body react during seizure
tonic clonic
partial seziures
begin in one part of brain
simple partial
consciousness remains intact
complex partial
impairment of consciousness
aura
portion of seizure that occurs before cosniousness is lost
post ictal
time after seizure event
epilepsy
group of syndromes characterized by unprovoked reoccurring seizures
complications of seizures
injury
aspiration
status epilepticus
seizure
- depletes the energy stores, increases O2 consumption, increases metabolic demands
cerebral anoxia and edema
meds to halt seizure activity
benzos
meds to maintain seizure free state
phenytoin and phenobarbital
seizure assessment and diagnostics
labs
CT
MRI
seizure interventions for seizures
ABC
safe environment
patent IV
observe and record seizure activity and length
seizure meds for prevention
dilantin
depakote
tegretol
medical management for seizures
keto diet
vagus nerve stimulaor
headache medication history
nitrates
vasodilators
histamines
alcohol
types of meningitis
bacterial
viral
fungal
meningitis classic triad
fever
headache
nuchal rigidity
meningitis
kernigs
knee flexion and cannot straighten
meningitis
brudzinskis
neck flexed and knees flexed
meningitis
bedside risk score
increased age
HR >120
+ gram stain
cranial nerve palsy
decreased GCS
meningitis
lumbar puncture
opening pressure increased
leukocyte count is elevated
protein is elevated
meningitis
prevention
meningococcal vaccination
meningitis
meds
antipyretic
antiseziure
IVF
meningitis
treatment is __________ and directed at _________ management
supportive, symptom
meningitis
consult with
epidemilogogy and infection control
meningitis
monitor what status closely
neuro
brain abscess
- higher rate in
immunocompromised
brain abscess
- indicative of
underlying disease or use of immunosuppressive medications
brain abscess
- intracranial brain
progressive symptoms
headache worse in morning
reduced vision
brain abscess
- intracranial epidural abscess
nuchal rigidity
brain abscess
- labs
increased WBC
elevated erythrocyte sedimentation rate
blood cultures
brain abscess
- lumber puncture
elevated opening pressure
increased protein levels
increased lymphocytes
brain abscess
- meds initiated ASAP
antibiotics
brain abscess
- nursing management
monitor vs, Resp, neuro
encourage rest
administer antipyretic/analgesics
coordinate home health care for long term antibiotic therapy
encephalitis is located where
brain tissue
encephalitis what is key for medicaitons
early administration
encephalitis
nursing management
frequenct and ongoing neuro assessment
supportive care
pain meds
seizure prevention
injury prevention
patient safety
multiple sclerosis
-what is happening to myelin
demyelinating
multiple sclerosis
- characterized by
relapses and remission or slow steady progressive dysfunction
multiple sclerosis
- age
young adults
multiple sclerosis
- MRI findings
disruption of blood brain barrier
evidence of inflammation
plaques
axonal loss
brain atrophy
multiple sclerosis
- disease state in every patient
varies in severity
multiple sclerosis
- prediction of symptoms
no
multiple sclerosis
- what occurs in relapsing and remitting course
residual deficits may occur
multiple sclerosis
- s/s
weakness
spasticity
loss of coordination
cognitive changes
fatigue
loss of balance
multiple sclerosis
labs
banding on CSF fluid
multiple sclerosis
cure
none
multiple sclerosis
- meds used for
relapse management and immune suppressants
multiple sclerosis
spasticity
baclofen and benozs
multiple sclerosis
ataxia
beta blockers and anti seizure
multiple sclerosis
nursing intervention of fatigue
recommend energy conservation
recommend that patient avoid overheating
rehab
- Pt/Ot
- pressure ulcer prevention
lifestyle acitivyt level
multiple sclerosis
nursing interventions for spasticity
assess sequelae of spasticity
- difficulty with gait/sitting
hygiene, comfort, energy level, sexual activity
mobile/transfer/safety
use of assistive devices
MG
- autoimmune
yes
MG
- affects
voluntary muscles groups including ocular, oropharyngeal, facial, shoulder girdle, limbs
MG
- bulbar
involves breathing
swallowing
speach
MG
- when intercostal muscles resulting in
decreased vital capacity
respiratory failure
MG
- medical management
treat symptoms up to a level of maximum response
plasma exhange
MG
- nursing interventions
assess respirations
monitor for myasthenic crisis
assist with mobility
assess for swallow and gag
myasthenic crisis
failure of respiratory muscles to maintiain ventilation
priority interventions include airway management typically through intubation
MG
- outcomes
patient is able to maintain own airway
GBS
- weakness
ascending
GBS
- autoimmune?
yes
GBS
- CNS or perhierpal
peripheral
GBS
- what happens to myelin
demyelination
GBS
- gradual or rapid
rapid
GBS
- weakness begins in
legs
GBS
- may result in _________ when diaphragm is impacted
ineffective ventilation
GBS
- progress to peak severity typically in __ weeks
2
GBS
- autonomic dysfunciton
cardiac instability
GBS
- lumbar puncture
increased protein
GBS
- pulmonary function tests
vital capacity
negative inspiratory force
GBS
- medical management
plasma exchange
monitor for hemodynamic compromise
GBS
- nursing inteventions
monitor Resp status, VS, neuro, CN, increased weakness, respiratory failure, DVT
parkinsons
- what type of neurological disorder
degenerative
parkinsons
- caused by a depletion of
dopamine
parkinsons
- characterized by
resting tremor, rigidity, bradykinseas, diminished postural stability
rigidity
resistance of passive limb movement
parkinsons
- what diagnostic testing to confirm
none
parkinsons
- medicaiton
anticholinergics
symmetrel
dopaminergics
- levodopa
levodopa benefits most pronounced for
1-2 years
parkinsons
- surgical treatment
thalamotomy and pallidotomy
deep brain stimuation
parkinsons
- nursing interventions
mobility: emphasis on safety
nutrition