Diabetes- EXAM 2 Flashcards
Type 1 DM
Characterized by hyperglycemia
Lack of insulin into the cells, body doesn’t produce it; genetic—-> NEED INSULIN
Usually onsets in childhood and adolescents
Genetic predisposition and viral exposure
Risk factors to Type 1 DM
Genetic
Occurs to younger population
Thin and unnourished
Manifistations of Type 1 DM
Polyuria, polydipsia, Polyphagia= frequent thirst, urination, hunger
Fatigue
Sudden vision changes
Tingling or numbness of feet
Recurrent infection
Diagnostic tests to determine Type 1 DM
Fasting plasma glucose test: Over 100 is preDM
Over 126 is DM
Hemoglobin A1C, above 5.7 is DM
Urinalysis for ketones and glycosuria
Urine for protein dipstick
Type 1 DM managment
Insulin
Type 2 managment
Oral Hypoglycemic agents
Type 2 DM
90-95%—-> You are the problem
Pancrease produces insulin but the body doesn’t take it, insulin resistence. Too much insulin.
Glucose hangs out in the body
Type 2 DM risks
Obesity, Diet, Genetic components, pre diabetic diagnosis
Type 2 DM manifestations
reoccurring infection
Reoccurring heat infection
Prolonged wound healing
Vision changes
Fatigue
Prediabetic lab levels
A1C and FBG
5.7% or higher
FBG: 100 to 125 mg/dL or higher
Rapid acting insulin and duration
Lispro, Aspart and Glulisine
3-5 hour duration, Onset is 15 min
Same time as you eat
Think move your Ass, Let’s go, Glue dries fast
Short acting Insulin and duration
Regular, Clear
5-8 hour duration, onset in 30-60 min
Before meals
Intermediate Acting Insulin and duration
NPH, cloudy
20-24 hours, onset in 1-2 hours
Type 2 DM managment meds
Metformin is the initial drug of choice but Glipizide swell
M:causes N,V,D, CNS symptoms too, Palpitations and chest discomfort
G: taken 30 min before breakfast, causes: nervousness, anxiety, depression, dizziness and insomnia
Gestational DM
Develops during pregnancy from stress, pre-existing DM, altered metabolism due to hormones
Increases risk for C-section
Hydramnios
Increased amniotic fluid may be result of increased fluid urination due to fetal hyperglycemia
Preclampsia
eclampsia occurs especially when vascular changes are present
Hyperglycemia & SS
HOT AND DRY, SUGAR IS HIGH
Sugar above 100
Increased stress=increased glucose
Skipping or not using enough insulin
Increased hunger/thirst/ urination
High risk factors for gestational DM
Previous history of GDM
Previously elevated BG
Over 40 years old
Previous macrosomia
Taking antiphycosis and corticosteroid use
Tests done to fetal status of GDM
AFP protein done at 16-20 weeks
Monitor fetal activity at 28 weeks
Ultrasound is done at 18 weeks to determine anomalies
Diabetic ketoacidosis
DKA
Associated with Type 1DM
Life threatening, Body produces too many ketones
Insulin deficit, depletion of potassium
Can be triggered by infection, inadequate insulin and CVD
Required immediate medical attention and fluid replacement due to polyuria and vomiting
DKA laboratory findings
Symptoms and considerations
BG of 250 or higher ( up to 800)
blood pH less then 7.30
Serum bicarbonate lower then 16
Dehydration, Lethargy, Polyuria, Blurred vision, Gastro symptoms, Acetone breath aka fruity, Hyperventilation
Hydrate first! D5W
Then slowly lower sugar, Monitor glucose levels
HHS & symptoms
Life threatening
Occurs in type 2 diabetics, When blood sugar levels are too high for long period of time; UNMANAGED DIABETES!!!! Sugar is above 600
Like DKA but without the ketones, there’s enough insulin circulating in the body to prevent DKA
Glucose gets into bloodstream and overflows the kidneys causing electrolyte depletion and extreme dehydration
Develops slower then DKA
Nursing managment of DKA and HHS
Monitor BG levels and ketones
Maintain fluid and electrolyte balance!!!
Increase patient knowledge about diabetes managment
Decrease anxiety
Monitor for fluid overload, hypokalemia and cerebral edema