CVS physiology 6 - presure nd flow in arters and veins Flashcards

1
Q

what are korotkoff sounds

A

sound when the blood is being tourbuald

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2
Q

what are hte disandves of manula blood ressure

A

discountis - can’t be measured constanlty
accuracy - needs regular calibarion and different from person to person
skilled

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3
Q

what are the advantages of manaul blood prssure

A

non invasive and cheap

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4
Q

what happens to blood pressure reading if the cuff is too large

A

underestimate

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5
Q

what happens if the blood pressure cuff is too small

A

over estimate

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6
Q

how does a automatic blood pressure machine work

A

the turbulanet flow sets up vibrations, the transueseer monitors the vibration, the maximum vibration occur at the mean arterla pressure, an algorithm estimate the diastoci and systolic pressures

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7
Q

what happens to the blood in venticalr contation

A

pushes it into aorta and this streches the walls of ti

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8
Q

what happens to blood during venticlur relaxiont

A

the blood leaves the aortora, slow asthe vessel have elastic recoild

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9
Q

what 4 factors effects pressure flow

A

stroke volume, veloicty of ejection, elasticy of arteries, total peripheral resitance

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10
Q

how does stroke volume effect pressure

A

as more blood is being pumped out, greater forces is being required thus increase in pressure

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11
Q

how does veloicty of ejection effect pressure

A

with larger risign phase, less time for the walls of the aeorta to stretch, thus higher systolic volume and lower dystolic volume

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12
Q

how does elasticicty of arteries pressure wave volume

A

loss as age, thus less able to spread pressure evently, thus more flow all at once

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13
Q

how does total resure resant effect

A

if arteoles are contrest, tpr will be higher, then more gradium falling paht and higher disaltoic pressure

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14
Q

what happens to arterail pressure with age

A

incresae

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15
Q

what happens to blood pressure thogu the day

A

it chanes cloisn, lower durign sleep

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16
Q

what happen as blodo goes int thea areis and what is it

A

small drop , but only small as they are low resistance, 95 to 90mmg

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17
Q

what happens toblodo rpessure as they go to teh aeortieols and what is it

A

large drop as they are resianct vessles, 90 to 40mmhg

18
Q

what happens blood pressure when it goes from cappie to veins and what is it

A

only allitle bit left
5- 20mmgh

19
Q

where is teh velocity of blood highest

20
Q

where is teh veloicty of blood the lowest

A

cappilare, veain and arteilies

21
Q

why is the veloicty of blood higher in teh aorta and vena cava

A

it has lower corss sectional area

22
Q

why is the velocity of blood lower in teh cappiles

A

higher cross scional rea

23
Q

what is teh effect of gravity on blood vessls

A

pulls blood down, cauing it to swell in the feat,

24
Q

what is teh effect of gravty on the heart

A

reduced edv, reduced prelaod, reduced sv, reduced co reduced map

25
what does juglar vein mean
high work from heart, possibly rasied jvp
26
what is orthostic hypotension
when there is redced blood pressure when you stand up
27
what is teh function of skelat mules pum
rythmic contracion increase venous blood return and edv
28
what is teh fucntion of the respiatyr pump
when you breathin increas advboap press, this push blod back
29
how to increase respat pump
increased breath and depth
30
what is venomotortone
contraction of the sommt musle that suron the venues and vains, which mobiles capacine and increase edv
31
what is systemic filling presseus
the remain pressues that the venues and veins have left after going through the vascular tree
32
what are the wo ways that a clot can happen (what is a clot made of)
platelte plug and a fibrin clot
33
What produdes prostaycing and no do
inhibits platelete aggregation
34
what does initation of tissue factor pathway inhibitor do
stops thrombin productoin
35
what does experesss thrombomodulin do
binds to thrombin and inactiaves it
36
what does expresses heparin do
inacives trhonbin
37
what does tissue plasminogen activator do
breatk sdown blood cloess
38
what does fibrin originate from
fibringogen
39
what molcues caues fibrinorgen to turn to fibrin
thrombrin
40