98 - Cold Injuries Flashcards
Y/N: The human capacity for physiologic adaption to cold is minimal
Yes
The body can maintain a constant temperature of approximately 37C over a range of external temperatures between
15C and 54C
Core body temperature is prioritized and maintained largely by controlling
Cutaneous blood flow
Arteriovenous anastomoses are abundant in
Acral areas
In prolonged cold exposures, the skin experiences a paradoxical cyclic vasodilation known as _____, to protect against skin necrosis from prolonged vasoconstriction
Hunting reaction of Lewis
(Slow/Fast) freezing results in extracellular formation of ice whereas (slow/fast) freezing tends to produce intracellular ice
Slow
Fast
In (slow/fast) rewarming, ice crystals become larger and more destructive
Slow
Other biologic factors influence vasoconstriction and tissue damage besides temperature
Painful stimuli
Mental stress
Arousal stimuli
Deep breaths
Individual factors predisposing to cold injuries include
Skin conditions with TEWL (eg, atopic dermatitis) Physical injuries Leanness Low physical fitness level Fatigue Dehydration Previous cold injuries Sickness Trauma Poor peripheral circulation Poor clothing insulation Old age
(Under-/Over-)weight persons are more likely to survive prolonged accidental cold exposure
Over-
Y/N: Habitually cold-exposed skin develops a more efficient system for shunting blood away from the surface
Yes
Y/N: Individuals who have experienced previous severe cold injury may have a profoundly delayed or absent hunting reaction in the affected limbs, making them more susceptible to recurrent cold injury with pain, hyperesthesia, or paresthesia
Yes
Skin cold injuries can be divided into _____ cold injuries
Freezing
Nonfreezing
Freezing cold injuries
Frostbite
Iatrogenic cold injury
Self-inflicted cold injury
Nonfreezing cold injuries
Vasoconstriction Hunting reaction Immersion foot Pulling-boat hands Acrocyanosis Chillblains Cold urticaria Cold panniculitis Erythromelalgia Raynaud phenomenon Sclerema neonatorum Subcutaneous fat necrosis of the newborn Livido reticularis Cryoglobulinemia Cold agglutinins Cryofibrinogenemia
Occurs when tissue freezes after exposure to cold air, liquids, or metals
Frostbite
The clinical presentation of frostbite falls into 3 categories
Frostnip (mild frostbite)
Superficial frostbite
Deep frostbite
Frostbite:
Involves only the skin and damage is reversible
Sensation of severe cold progressing to numbness followed by pain
No edema or bleb formation
Frostnip
Only form of frostbite that can be treated safely in the field with first aid measures
Frostnip
Frostbite:
Involves the skin and immediately subcutaneous tissues
With pain subsiding to feelings of warmth (sign of severe involvement)
Clear blebs form, accompanied by edema and erythema
Superficial frostbite
Frostbite:
Extends to the deep subcutaneous tissue
Affected skin becomes deceptively pain free, and the discomfort of feeling cold vanishes
Tissue is totally numb
Large blisters form
Leads to formation of hard, black gangrene
Deep frostbite
Good prognostic signs of frostbite
Large, clear blebs extending to the tips of the digits
Rapid return of sensation
Rapid return of normal (warm) temperature to the injured area
Rapid capillary filling time after pressure blanching
Pink skin after rewarming
Poor prognostic signs of frostbite
Hard, white, cold, insensitive skin
Cold and cyanotic skin without blebs after rewarming
Dark hemorrhagic blebs
Early evidence of mummification
Constitutional signs of tissue necrosis, such as fever and tachycardia
Cyanotic or dark red skin persisting after pressure
Freeze-thaw-refreeze injury