27 JUNE 2019 Flashcards

mostly all chapter 13? no theres a lot of chapter 14

1
Q

list five types of involuntary muscle movements:

A
  1. muscle cramps
  2. tremors
  3. fasciculations
  4. myoclonus
  5. fibrillations
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2
Q

what are fasciculations:

A

spontaneous oscillations of muscle fibers

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3
Q

a fasciculations can be two things:

A

benign or pathologic

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4
Q

when is there a benign fasciculation?

A

when there is no cormorbidity w/ s/s of damage

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5
Q

when is there a pathologic fasciculation?

A

when there is cormorbidity w/ s/s of damage

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6
Q

what is myoclonus?

A

the whole boyd ocntracts doesn’t know what caues it

- what happens when you’re on the verge of falling asleep

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7
Q

what are tremors?

A

involvuntary muscle contractions:

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8
Q

what are the two types of tremors?

A

resting

action

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9
Q
resting tremors are: 
def + ie:
A

oscillating back and forth type movement of a limb

ie: parkinson’s dx

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10
Q

action termors that we are concerned about are

name + def + ie:

A

intention
its a tremor whenever you move
ie: cerebellar dysfx

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11
Q

fibrillations are

A

always pathologic

spontaneous depolarization of a 1 muscle fiber

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12
Q

how do you detect fibrillations?

A

w/ a needle = end of need goes into your fiber and will feel a tiny electrode that make it vibrate
= NEEDLE EMG

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13
Q

name the five potential sources of damage to nerves:

A
  1. trauma
    - blunt / sharp
  2. infection
  3. neurogedenerative
  4. vascular dx
  5. tumors
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14
Q

what are the two types of trauma:

A

blunt: compression of proximal end of nerve (squish it) s
sharp: cut peripheral motor neuron

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15
Q

infection def and ie:

A

infections attack cell bodies ie: polio

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16
Q

neurodegenerative ie;

A

ALS

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17
Q

vascular dx def:

A

nerves need rich supply of oxygen so this happens when they do not get enough blood

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18
Q

tumors:

A

bunt trauma

grow and press on nerve then leak gluatamate = excitotoxictiy

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19
Q

name three signs of LMN lesions:

A
  1. denervation
  2. decrease or loss of reflex
  3. paresis or paralysis
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20
Q

difference between sp nerve and peripheral nerve damage:

A

spinal nerve = weakness (paresis)

peripheral nerve = paralysis / no sensory info

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21
Q

when there is a loss of reflex that means:

A

the spinal level N is cut = decrease b/c stretch going into the spinal cord can’t come back and form a response

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22
Q

denervation means:

A

wallerian denervation: where the distal axon dies and the muscle fiber loses its axon

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23
Q

where can LMN lesions happen?

A

ventral root
peripheral root
spinal nerve
***all look differently depending

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24
Q

what neuron does LMN lesions affect?

A

a-alpha

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25
name the two types of atrophy:
disuse | denervation
26
disuse atrophy def:
muscle gets smaller than normal still has bulk though
27
denervation atrophy def:
muscle will wither away from nothing
28
"flaccid paralysis" def:
1. loss of muscle tone 2. inability to contract 3. due to injury of alpha motor neuron
29
hypotonia def:
losses some of alpha motor neurons
30
flaccidity def:
it if losses all muscle tone | completely resistive to passive stretch
31
where does motor planning take place?
the basal ganglia
32
what two places does UMN start?
the cerebral cortex and brainstem
33
where does the cerebral cortex send messages to ?
the LMN (and the brainstem)
34
where does the brainstem send messages to ?
the spinal cord
35
the cerebral cortex is made of what and called what?
grey UMN hemisphere | cortical origin
36
the brainstem is called
subcortical orgin
37
what has greater voluntary control?
the cerebral cortex
38
what does the cortex control do?
DIRECTLY controls LMN and then muscles
39
what doe the brain stem do?
provides direction for the muscle (from info from the cortex) sends messages that go down to the spinal cord
40
the medial group controls the
proximal muscles
41
sub cortical origin:
supports voluntary movements | guided by medially lateral cortical pathway
42
cortical origin:
has direct voluntary control of movement
43
the lateral group controls the
distal muscles
44
where does the reticulospinal (medial group) start
starts in the brainstem in the reticular formation
45
where does the reticulospinal project:
broadly out to the upper and lower extremities
46
what does the reticulospinal produce?
gross limb movement lower degree of direct voluntary control of postuarl muscles
47
the medial vestibulospinal projects to
the core axial - upper bakc/ postural Mm - head and neck
48
what does the medial vesibulospinal do?
maintains upright against gravity | lower degree of direct voluntary control
49
the lateral vestibulospinal
overall still controls core/proximal Mm - axial & LE mm lower degree of direct voluntary control
50
the lateral vestibulospinal is mostly:
unilateral
51
the three medial groups according to what they do:
1. reiculospinal - picks stuff up against gravity 2. medial vestibulospinal - upright against pull of gravity 3. lateral vesitbulopsinal - keeps you upright against gravity
52
the fourth medial group is
medial corticospinal
53
medial corticospinal projects to
projects to the medial motor neuron pools bilaterally
54
medial corticospinal controls
neck shoulder trunk Mm
55
why is the medial cortico-spinal group different
it has direct control of the voluntary movement: because it comes from the cerebral cortex
56
what are the lateral group pathways?
1. rubrospinal | 2. lateral corticospinal
57
rubrospinal:
minor control of distal extensor extermity of UE
58
lateral corticospinal:
directly activates distal Mm | provides "fractinated" movement:
59
what does fractinated mean
any Mm in any combination I want = full voluntary control
60
lateral corticospinal does two things?
1. direct control of LMN | 2. guiding control to brainstem group
61
which pathway is the MOST IMPORTANT PATHWAY FOR VOLUNTARY MOVEMENT?
the lateral corticospinal
62
where is home for all the lateral corticospinal cell bodies?
home of the pre-central gyrus
63
what are the percentages of the medulla?
90% cross | 10% stay