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Neuroscience > 10 JUNE 2019 > Flashcards

10 JUNE 2019 Flashcards

so end of ch. 6 start of ch. 7

1
Q

when is GABA fast acting?

A

w/ a GABA a receptor

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2
Q

when is GABA slow acting?

A

w/ a GABA b receptor

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3
Q

what type of response is a GABA amino acid?

A

inhibitory : generates an IPSP response

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4
Q

what is the most prevalant inhibitory fast-acting amino acid?

A

GABA

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5
Q

what is the most prevalent excitory fast-acting amino acid?

A

Glutamate

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6
Q

when is gluatmate fast acting?

A

w/ AMPA receptor

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7
Q

when is glutamate slow acting?

A

w/ NMPA receptor

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8
Q

what type of response does glutamate do?

A

excitory: EPSP repsonse

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9
Q

what two things can serotonin affect?

A

changes our perceptio of pain

and adjust our arousal levels

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10
Q

what pathology does serontonin affect?

A

depression - most common anti-depressent drug

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11
Q

what type of fast slow methods does the CNS and PNS have?

A

CNS has BOTH fast/slow = gas pedal + breaks
PNS has ONLY fast = gas pedal
- to slow you dont press the gas

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12
Q

dopamine affects what four things?

A

motor activity
cognition
behavior
pleasure

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13
Q

dopamine is associated with what?

A

addiction

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14
Q

norepinephrine is part of what system?

A

fight or flight which is the

autonomic - sympathetic system

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15
Q

tell me two things about norepinephrine:

A

increases attention to sensory information

apart of autonomic - sympathetic system

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16
Q

what does neuroplasticity mean?

A

the brain can adjust : so it can increase in ability or decrease in ability but there is the ability to change

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17
Q

what are the three big amino acids we need to know?

A

Acetylcholine
gluatmate
GABA

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18
Q

how many other random proteins do we need to know?

A

five

19
Q

what are the five random proteins we need to know?

A 
histamine 
epinephrine 
dopamine 
serotonin 
peptide
20
Q

tell me two things about histamine:

A

it can create an interaction affect

and is part of the neuro inflammatory response

21
Q

norepinephrine increase what?

A

attention to sensory information

22
Q

amines are what?

A

slow acting neurotransmitters

23
Q

what are the two types of peptides?

A
  • endogenous opioids

- substance P

24
Q

endogenous opioids do what?

A

inhibit perception of pain

25
Q

substance p can be

A

a neurotransmitter or modulator

26
Q

substance p are?

A

pain pathways

27
Q

habituation def:

A

decrease response from a repeated begin stimulus

28
Q

a short term habituation occur where?

A

presynaptic facilitation

29
Q

what two ways can you get a presynaptic facilitation habituation:

A

1- more presynaptic inhibition

2 - less presynaptic facilitation

30
Q

name two examples of “pathologies” w/ habituation:

A

tactile defensiveness

vestibular disorders

31
Q

a long term habituation occurs where :

A

post synpatic membrane

32
Q

how do you get a long term habituation:

A

you take away the receptors on the membrane so there can no longer be a facilitation response:
down regulates

33
Q

down regulate def:

A

less receptitive to AP

34
Q

how do you down regulate

A

you INTERALIZE or pull down membrane receptors from the post synaptic terminal making htem
INACTIVE
= flipping the switch permenantly so the lights are off

35
Q

when you down regulate what happens to potential

A

it reduces the chance of AP

36
Q

up regulate def:

A

increase recepitiveness to neurotransmitters

37
Q

how do you up regulate?

A

take receptors inside the cell & stick it up in the membrane = EXTERNALIZE = ACTIVE = unlock the gate to allow ion to flow in

38
Q

when you up regulate what happens to potential?

A

increases the chance of an AP

39
Q

what does myasthentic gravis mean?

A

muscles dont work BIG time

40
Q

what are three bad things that happen with MG?

A
  1. membrane unfolds
  2. receptors move closer to the surface
  3. the amount of acteylchloline receptors decrease
41
Q

what are the consequences of the bad things in the MG?

A

the acteylchloline can’t be recycled so the left over proteins gets gobbled up by closer those receptors closer to the surface

42
Q

what three things do we need to know for neuroplasticity?

A

1 - habituation
2- experience-dependent plasticity
3 - recovery or maladaptive behavior

43
Q

what does LTP (long term potentiation do)

A

converts a silent synapse to an active synapse

Neuroscience (29 decks)

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