Tenosynovitis Flashcards

1
Q

Lateral epicondylitis results from microtears at the origin of the common extensor tendon mass, specifically
the extensor carpi radialis brevis tendon

A

T

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2
Q

Medial epicondylitis affects the common flexor tendon
mass, most often the pronator teres and flexor carpi raclialis.

A

T

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3
Q

Conservative management is the mainstay of medial
epicondylitis treatment and some of the patients need to be assessed
for possible ulnar neuropathy.

A

F all patients should be assessed
for possible ulnar neuropathy.

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4
Q

Intersection syndrom approximately 4 cm proximal to the radiocarpal joint. It often resolves with nonoperative treatment
such as therapy and splinting.

A

T

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5
Q

Trigger fingers in Diabetics are less likely to
respond to nonoperative treatment

A

T

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6
Q

disorders involving the tendons of
the upper extremity are the result of degenerative or mechanical
stresses, rather than an inflammatory process

A

T

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7
Q

The causes of tendinopathy?

A

Degenerative, mechanical, inflammation,and infection

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8
Q

Lateral epicondylitis occurs in male more than female

A

F It is equally common in men and women

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9
Q

the
classic finding of lateral epicondylitis includes ECRB tendon pathology.

A

T

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10
Q

lateral epicondylitis involve ECRP only

A

F the extensor digitorum communis (EDC), and the ECU.
Although each of these tendons may be involved in the process

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11
Q

s within the ECRB tendon
substance represent the sine qua non for lateral epicondylitis.

A

T

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12
Q

Historically tendinosis rather than tenosynovitis was thought to occur in the lateral epicondylitis

A

F historically was thought it was inflammation rather than degenerative

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13
Q

Inflammatory cell are present in the histopathological study of lateral epicondylitis mainly macrophage

A

F Inflammatory cells, such as macrophages and neutrophils,
are lacking

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14
Q

repetitive contact between
the capitellum and the ECRB tendon may be the causative problem.

A

T

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15
Q

provocative maneuver
that may elicit increased pain

A

resisted wrist extension with the
elbow extended and the wrist pronated

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16
Q

Imaging studies add limited information in the diagnosis of lateralepicondylitis

A

T

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17
Q

Ultrasound (US) and magnetic resonance imaging (MRI) have both
been utilized for diagnosis, to assess disease severity and for purposes ofpreoperative planning

A

T

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18
Q

Ultrasound has been found to have higher sensitivity than MRI

A

F MRI is often the modality of choice, with
a higher sensitivity of 90% to 100%

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19
Q

However, the severity ofdisease
present on MRI does not necessarily correlate with symptoms

A

T

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20
Q

patients with resolved pain may continue to have pathologic findings
present on MRI

A

T

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21
Q

MRI is often reserved for recalcitrant cases in which
the patient fails to improve with 6 months of conservative treatment
or when the diagnosis in unclear

A

T

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22
Q

the condition usually clears up in eight to twelve months without any treatment except
perhaps avoidance of the painful movements

A

T

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23
Q

the mainstay of treatment for
lateral epicondylitis

A

activity modification

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24
Q

up to 40% of people continue to have discomfort after 1 to 5 years of
nonoperative treatment

A

T

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25
Despite numerous randomized controlled studies, none of these interventions has been definitively proven to work better than rest and therapy alone.
T
26
Physical and occupational therapy for lateral Epicondylitis patients improved in the short term and in the long term
F short term only
27
Injections into and around the lateral epicondyle remain highly controversial
T
28
Some studies have shown an improvement in pain at 4 to 6 weeks after steroid injection , but this advantage usually is not present at 12 months
T
29
Transient loss of finger extension is a potential side effect of botox injection
T
30
Indication of surgery
patients who fail 6 to 12 months of conservative management and who remain significantly affected by pain
31
release of the ECRB from the lateral epicondyle, open, or arthroscopic debridement of the ECRB tendinosis, denervation of the lateral epicondyle, and anconeus rotation
T
32
Disadvantages of arthroscopic approach
include decreased visualization of the ECRB tendon, resulting in potential incomplete debridement of diseased tissues, and greater possibility of radial nerve injury
33
the arthroscopic approach has similar results to the open approach
T
34
Medial epicondylitis, also known as golfer's elbow, is a common cause of medial elbow pain
T
35
Medial epicondylitis occurs more than lateral epicondylitis
F medial elbow pain. Although less prevalent than lateral epicondylitis (
36
medial epicondylitis is associated with repetitive movements and is often found in patients 40 to 50 years old
T
37
Muscle involved in the medial epicondyle
The common flexor tendon is composed of five muscles including the pronator teres (PT), FCR, flexor carpi ulnaris, palmaris longus, and flexor digitorum superficialis
38
the pathophysiology of the medial epicondylitis
Microtrauma from repetitive motion, specifically repetitive valgus loads, may result in mucoid degeneration of the tendons resulting in the histopathologic findings of angiofibroblastic hyperplasia, fibrosis, and calcification
39
DDX of medial epicondylitis?
elbow arthritis, osteochondral defects, medial collateral ligament instability, cervical radiculopathy, pronator syndrome, and ulnar neuritis/cubital tunnel syndrome
40
Ulnar neuritis is especially important as 23% to 61% of patients with medial epicondylitis also have concurrent ulnar neuropathy
T
41
Ultrasound can be used to assist in the diagnosis of medial epicondylitis, as it has been shown to be both specific and sensitive
T
42
Treatments
Activity modification Physical therapy, with common flexor mass stretching and strengthening Counterforce bracing has shown some success Elbow extension bracing Corticosteroid injections for short term relieve( 6 week )
43
Overall, in 88% to 96% ofcases, nonoperative management and the passage oftime has been shown to result in resolution ofsymptoms.
T
44
Structure at risk in medial epicondylitis surgery
the ulnar nerve, the MCL and the medial antebrachial cutaneous nerves
45
De Quervain disease is a stenosing tendiopathy involving the first dorsal extensor compartment
T
46
radial-sided wrist pain exacerbated by ulnar deviation of the wrist and thumb abduction.
T
47
De Quervain mor common in male
It is most common in the fifth and sixth decade of life and is up to six times more common in women than men
48
It is also more commonly seen in pregnant and lactating women
T
49
(APL) and extensor pollicis brevis (EPB) travel together within the first dorsal compartment, which overlies the radial styloid.
T
50
the EPB can travel through its own separate subsheath within the first compartment
T
51
DDX od deQuervian
CMC joint arthritis of the thumb, scaphotrapezial-trapezoid arthritis, intersection syndrome, scaphoid fracture, and radial sensory nerve pathology
52
Non operative treatment
nonsteroidal antiinflammatory medications along with splint immobilization with or without corticosteroid injection
53
80% had complete and lasting reliefofsymptoms after corticosteroid injection.
T
54
patients who fail to improve with corticosteroid injections have been found to have a higher rate ofEPB subsheath than the general population
T
55
The EPB should be clearly identified and passively tested to ensure thumb metacarpal extension before the surgical release is complete
T
56
The percentage of EPB sub sheath
44% in the normal population and 62% in de Quervain disease patients.
57
Intersection syndrome involves the contents of the second dorsal compartment, the ECRL and ECRB as they cross beneath the first compartment muscles (APL and EPB)
T
58
Erythema and crepitation may be present.
T
59
intersection syndrome is an uncommon cause of wrist pain,
T
60
MRI and ultrasound can be useful modalities for distinguishing between intersection syndrome and other pathologies, such as de Quervain disease.
T
61
Controversy exists as to whether stenosis of the second compartment sheath alone versus friction between the first and second compartments is the underlying etiology
T
62
Nonsteroidal antiinflammatory drugs (NSAIDs) combined with splinting of the wrist in 20° ofextension for 2 to 3 weeks resolves most cases. Refractory cases can be treated with a corticosteroid injection
T
63
Trigger finger, also known as stenosing tenosynovitis
T
64
lifetime risk percentage
of 2.6% in the general population and up to 10% in those with diabetes
65
The Al pulley, located at the level of the metacarpal head is the site of mechanical impingement seen in trigger finger
T
66
The thumb's Al pulley is located near the MCP joint crease and is the site of flexor pollicis longus triggering.
T
67
repetitive motion of the flexor tendons through the Al pulley leads to friction within the flexor sheath, causing both thickening of the pulley and nodule formation within the flexor tendon
T
68
it is not uncommon for patients to localize the problem to the proximal interphalangeal (PIP) joint rather than the Al pulley
T
69
association with specific occupations has not been found
T
70
patients with diabetes, hypothyroidism, rheumatoid arthritis, gout, and renal failure develop trigger fingers at higher rates
T
71
there is no evidence ofinflammation but there is evidence of fibrocartilage metaplasia and upregulation of type 3 collagen
T
72
Splinting of the MCP joint has been shown to be successful in up to 66% of cases
T
73
Splinting of the MCP joint has been shown to be successful in up to 66% of cases
T
74
with a higher success rate (70%) in digits versus thumbs (50%) with splining
T
75
The PIP joint can also be splinted, instead of the MCP joint, with similar effect.
T
76
Splinting is recommended for 6 to 10 weeks both during the day and night, to which some patients can find difficult to adhere
T
77
Corticosteroid has proven effective in treating trigger fingers and can be the first line intervention
T
78
a second injection increases the success rate
T
79
The injections should be intrasheath
F Intrasheath injections are not required for effect, and intratendinous injections should be avoided to reduce the risk of tendon rupture
80
Patients with diabetes do not respond as well to steroid injections; some studies advocated for immediate surgical release or only one attempt at injection before surgical intervention is considered
T
81
number of injections that should be attempted before proceeding to surgical release has not been established
T
82
Percutaneous techniques have been shown to be successful with up to 95% resolution of symptoms in all patients
F except for the diabetic patient sand hypothyroidism
83
statistically significant increase in digital nerve injury with percutaneous release versus open release
T
84
complications of open release
chronic pain, infection, iatrogenic digital nerve injury, incomplete release, and bowstringing
85
trigger finger and de Quervain disease are the most common tendinopathies of the hand and wrist
T
86
treatment of EPL tendinitis is often surgical with complete release of the third dorsal compartment, to prevent rupture of the EPL
T
87
The floor of the ECU tendon sheath is an important contributor to the triangular fibrocartilage complex
T
88
Injecting local anesthesia into the ECU subsheath can be a helpful diagnostic tool
T
89
Almost every tendon in the hand and wrist can cause pain if irritated through mechanical stresses or stenosing forces
T but Atypical conditions has been recorded include tendinopathy of the extensor carpi ulnaris (ECU), flexor carpi radialis (FCR), and extensor pollicis longus (EPL
90
Green classification of the trigger finger
Grade one pain only at the A1 LEVEL Grade 2 catching finger Grade 3 Looked digit passively corrected Grade 4 fixed flexion
91
Percutaneous techniques have been shown to be successful with up to 95% resolution of symptoms
T
92
statistically significant increase in digital nerve injury with percutaneous release versus open release,44 which may be most germane to the thumb
T