Pig Repro 2 Flashcards
Duration of Farrowing?
1 to 5 hours
Duration of more than ____ between piglets indicates trouble
1.5 minutes
Benefits to induction:
1. 2. 3. 4. 5. 6. 7.
- Allows farrowing during working hours
- Ensures all-in/all-out management
- Allows cross-fostering piglets between litters
- Piglet size will be more uniform
- improved labor efficiency.
- Improved piglet survival
- Improved facility utilzation
What drug class is more recommended for induction of parturition, prostaglandins or corticosteroids? Why?
Prostaglandins, corticosteroids will have too much lag and poor piglet survival if given too early.
Prostaglandins as parturition inducers:
- Two options?
- 10-50 mg Lutalyse or 175ug Estrumate (closprostanol)
Prostaglandins as parturition inducers:
- Administer on what day of gestation?
- Farrowing will occur how soon after admin?
- 112-113
2. 18-36 hours later
Two most common causes of porcine dystocia?
Uterine inertia and fetal maldisposition
Medical Therapy fo Dystocia:
- 2.
3.
- Manual extraction of piglets after manipulation
- Oxytocin 20-30 IU IM every 30 min.
- Calcium
Cesarean Section:
- Vertical incision location?
- Horizontal incision location?
- left flank
2. parallel to underline
C-section:
- Usually need to incise (one/two) horns?
- Closing pattern?
- two (both)
2. inverting patterns
Complications during parturition?
1.
2.
3.
- Prolapsed vagina, uterus and/or bladder
- Vulvar hematomas/tears
- Vaginal discharge/metritis
Prolapses during Parturition:
Prepartum Vaginal Prolapse:
- Tx?
- (is/is not) likely to recur?
- repair with a suture after placing back in
2. is
Prolapses during Parturition:
Uterine Prolapse:
- Prognosis?
- Occurs when?
- ____ may be needed
- GRAVE
- postpartum or intrapartum
- amputation (if necrotic or torn)
Vulvar hematomas and tears during Parturition:
- Etiology?
- (can/cannot) suture back in?
- mechanical damage to flaccid vulva near parturition
2. cannot.
Vaginal discharge and metritis during parturition:
- Don’t confuse with what?
- Appearence?
- See with what two pregnancy complications?
- CxS in sow?
- normal lochia 1-5 days postpartum
- necrotic malodorous discharge
- dystocias and/or RFMs
- Anorectic and febrile
Methods to reduce piglet mortality:
1. 2. 3. 4. 5. 6 .
- Supervised farrowings
- Cros-foster within 24-48 hours postpartum
- Vaccinate females pre-farrowing
- Feed females according to BCS
- Choose appropriate farrowing crate design
- Cull gilts that savage their piglets
What day range would be considered:
- Resorption?
- Mummification?
- Stillbirths?
- 0-35 days
- 35-114
- 114 - farrowing
Early Embryonic death:
- Generally, how many embryos suffer EED prior to implantation?
- If > ___ embryos, pregnancy remains with decrease in litter size
- 2 - 2.5
2. > 4 embryos
Early Embryonic Death:
- Fetal death < ___ days = regular returns
- Fetal death > ___ days = irregular returns
- < 14 days
2. > 35 days
Mummification:
- What will happen if entire litter is mummified?
- will progress to psuedopregnancy > 114 days
Stillbirth:
- Full term piglets born dead, but grossly normal except for…
- un-inflated lungs
Noninfectious causes of infertility:
- 2.
- Hydrosalpinx
2. Cystic Ovarian disease
Most common cause of infertility?
Second most common?
Hydrosalpinx
Cystic Ovarian Disease
Hydrosalpinx etiology?
secondary to bursitis or peritonitis that result in adhesions
Cystic Ovarian Disease:
- (single/multiple) cysts?
- Clinical Presentation?
- multiple
2. irregular cycles or anestrus
Cystic ovarian disease
- ___-___% of infertile cows have it?
- Do not respond to ___ therapy.
- Can be caused by…
- 20-50%
- hormonal
- estrus induction agents
Other non-infectious causes of infertility:
1.
2.
3.
- Anatomical or congenital abnormalites
- Environmental causes
- Nutritional causes
Anatomical or congenital abnormalities that can cause infertility:
1.
2.
3.
4.
- segmental aplasia
- uterus unicornum
- missing cervix
- hermaphroditic
Three mechanisms by which infectious disease can cause infertility from infecting a boar?
1.
2.
3.
- Direct disruption of spermatogenesis via localization within the parenchyma
- Indirect disruption of spermatogenesis via fever
- Shed in the semen and causes disease in females
Infectious agents cause infertility in a boar by causing direct disruption of spermatogenesis via localization within the parenchyma:
1.
2.
3.
- Brucella
- Chlamydia
- Rubulavirus
Infectious agents that are shed in the semen and cause disease in females:
1. 2. 3. 4. 5.
- Classic swine fever
- Leptospira
- psuedorabies virus
- Parvovirus
- PRRS virus
Bacteria that cause infertility in a sow:
1.
2.
3.
- Lepto
- Brucella
- Erysipelas
Viruses that cause infertility in the sow:
1.
2.
3.
4.
- PRRS virus
- Parvovirus
- Pseudorabies virus
- Classic swine fever
Two means by which bacteria are pathogenic:
1.
2.
- Introduced into the repro tract during breeding or parturition
- Resulting septicemia can cause loss of a pregnancy and subsequent repro problems
Most common cause of porcine pregnancy loss?
Lepto. L. pamona specifically
Epidemiology of Lepto in sows?
Brought in by carrier animals, than penetrates mucous membranes
Clinical Signs of Lepto:
- What happens to pregnancy?
- Clinical signs in sow?
- Immunity after?
- mummified and macerated
- mild
- immunity lasts for 1 year after aboriton
Prevention and Treatment of Lepto:
- Vax?
- ____ in the feed help to reduce shedding, but do not….
- Every 6 months, usually in combo with parvovirus
- Antibiotics (tetracyclines)
DOES NOT ELIMINATE CARRIER STATE
- Lepto (is/is not) a reportable disease?
2. Brucella suis (is/is not) a reportable disease?
- Is not
2. Is
Brucella suis:
- Repro effects in Sow?
- In boar?
- abortions, still birth, infertility. Piglet mortality
2. orchitis
Brucella suis:
- Transmission?
introduced and transmited by boar. They can carry it for > 4 years. Infections occur at coitus or ingestion of infected material
B. Suis:
- End result if sow is infectd between days 30-40?
- (sows/boars) shed large numbers of bacteria?
- Abortion between days 65-80.
2. Sows
B. Suis:
- Lesions in sows?
- granulomatous lesions in uterus, ovaries, testes, ASG, liver, kidney.
B. Suis:
- No ___ immunity.
- Can also be spread by what two other animals?
- durable immunity
2. rabbits and rodents
Prevention of B. Suis:
1.
2.
- Report positive animals
2. Test and slaughter, than buy replacements from certified brucellosis-free herd.
Erysipelothrix rhusiopathiae:
- Clinical significance?
- What happens in acute cases?
- Many pigs carry it on tonsils w/o CxS.
2. organism enters circulation via pharynx and infects blood vessels
Erysipelothrix rhusiopathiae:
- How is it transmitted?
- Clinical course depends on ___-
- Via contaminated feed and water
2. Stress = acute or chronic
Erysipelothrix rhusiopathiae:
Clinical presentation in:
- Little pigs?
- Gilts?
- Sows?
- arthritis
- fever and skin lesions
- abortion secondary to fever and septicemia
Porcine Respiratory and Reproductive Syndrome:
- Organism type?
- Sow mortality rate?
- Transmission?
- Arterivirus.
- 5-10%
- Direct contact w/ semen, aerosols
Porcine Respiratory and Reproductive Syndrome:
Two clinical forms it can take?
1.
2.
- Repro failure
2. Post-weaning respiratory disease
Porcine Respiratory and Reproductive Syndrome:
Repro Failure form has what clinical effects?
1.
2.
3.
4.
- premature farrowing
- Increased stillbirths/mummies
- weak neonates
- increased abortion
Porcine Respiratory and Reproductive Syndrome:
Post-weaning respiratory disease form has what clinical effects?
1.
2.
- decreased average daily gain
2. increased mortality
Diagnosis of Porcine Respiratory and Reproductive Syndrome:
- ____ = preferred
2.
- herd serology
2. virus isolation from aborted fetuses
Porcine Respiratory and Reproductive Syndrome: Prevention and Treatment:
1.
2.
3.
- Symptomatic therapy
- ISOLATION AND ACCLIMATIZATION
- Vax with MLV or killed product
Porcine Parvovirus:
- clinical presentation?
SMEDI - stillbirths, mummies, embryonic death, infertility
Porcine Parvovirus:
Routes on infection?
oral, venereal
Porcine Parvovirus:
Pathogenesis?
viremia –>transplacental crossing —> fetal vasculitis and death
Porcine Parvovirus:
Litters with dead fetuses and embryos may be carried > ____days
114
Porcine Parvovirus:
most commonly affects what type of pregnant animals?
naive replacement gilts
Porcine Parvovirus:
T/F: Sows infected do not usually show signs until farrowing
T
Porcine Parvovirus: Diagnosis methods:
1.
2.
3.
4. ***
- Gross and histological evaluation of lesions on aborted fetuses
- Examine vax program
- Serology
- Clinical signs of increased irregular returns to estrus and increased mummies****
Porcine Parvovirus:
Prevention and treatment:
1.
2.
3.
- Proper acclimatization of replacement gilts**
- Regular vaccination of breeding herd
- No treatment except for suportive care for the aborting sows or gilts
Aujesky’s aka ___
Pseudorabies
Pseudorabies:
- Causative organism?
- Can cause ____, ___, ____
- Herpes virus
2. encephalities, repro failure, reduced litter size
Pseudorabies:
- (can/cannot) be transmitted in semen?
- Can. Especially if boar is under stress
Pseudorabies:
Transmission methods?
1.
2.
3.
4.
None-to-nose contact, fecal oral, aerosol, semen
Pseudorabies:
Clinical signs if:
- Young (< 7 days)?
- Weaned pigs and older?
- Shared between all ages?
- Pregnant?
- CNS signs
- Resp. Signs
- Fever, anorexia, weight loss
- resorption, abortion, increased mummies and stillbirths
Pseudorabies:
Diagnosis:
1.
2.
3.
- Gross +/- histological lesions (pulmonary edema, meningioencephalitis)
- Virus isolation
- Serology
Pseudorabies:
Prevention methods:
1.
2.
- Eradication of whole herd population or positive breeding animals.
- Vax with a G1 gene-deleted product
Hog Cholera:
- Hog Cholera (is/is not) reportable?
- Pseudorabies (is/is not) reportable
- Is
2. Is not
Hog Cholera:
- Current US Status?
- Reportable disease eradicated in the 1970s.
Hog Cholera:
- aka _____
- Natural host?
- classical swine fever
2. Pigs
Hog Cholera:
Clinical Signs:
1. 2. 3. 4. 5. 6.
- Anorexia
- Cyanosis
- Fever
- Diarrhea
- Vomiting
- Abortions
Hog Cholera:
Best diagnostic test to differentiate from other viral infections?
Fluorescent antibody test
Zearalenone
- aka ____
- causes ______
- fusarium roseum
2. implantation failure
Zearalenone
- Clinical presentation in females?
- In prepubertal males?
- signs of estrus, mammary development and vaginal discharge
- reduced testicular development and feminization
Other moldy feed toxins:
1.
2.
3.
- Trichotecene toxin
- Fumonison
- Aflatoxin
Other moldy feed toxin effects:
- Trichotecene toxin?
- Fumonism?
- Aflatoxin?
- toxic to embryos and fetus
- indirect cause of abortion via severe reduction in cardiac output of sows
- poor piglet growth
Carbon monoxide:
- _____ is considered a toxic dose.
- Clinical presentation?
- End result?
- 220 ppm
- cherry red tissues from carboxyhemoglobin
- late-term abortions
Nutritional Factors and their effect on reproduction:
- decreased Ca/P?
- Iodine?
- Selenium?
- farrowing difficulty (inertia)
- increased stillbirths
- MMA, primary uterine inertia
Nutritional Factors and their effect on reproduction:
- low iron?
- low protein?
- low energy?
- high energy?
- piglet anemia and stillbirths
- increased WEI (Weaning to Estrus Interval)
- increased WEI
- decreased litter size