lecture 7 Flashcards

1
Q

gene environment interaction

A

environment can change the way genes are expressed

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2
Q

genetic code contains

A

instructions on how to respond to the environment

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3
Q

how do nervous systems change

A

neural activity changes synaptic connections
fire together wire together- strengthen
out of sync lose your link - weaken

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4
Q

synaptic activity - baseline activity

A

neural activity without stimulation

action potentials are generated randomly

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5
Q

neural signalling

A

change in activity relative to baseline level

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6
Q

action potential can either become

A

more forewent than baseline (excitation) or less frequent (inhibition)

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7
Q

persisent signalling

A

causes the synapse to change

increased activity causes short term molecular changes

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8
Q

frequent APS cause an increase in

A

neurotransmitter release and number of receptor molecules

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9
Q

increased neural activity can also cause

A

long term structural changes

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10
Q

long term structural changes

A

sustained activity - frequent AP’S can cause
growth of new synapses
synaptic takeover

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11
Q

how do changes affect behaviour

A

optimising existing behaviour
increased transmission rate
react more quickly , reliably to important changes in the environment

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12
Q

decrease transmission rate

A

makes you more likely to ignore unimportant changes in the environment

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13
Q

acquiring new behaviour growth of new synapses

A

combine info from previously unrelated sources

respond to old stimuli in a new way

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14
Q

synaptic takeover

A

reroute info to new pathways

respond to old stimuli in a new way

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15
Q

damage to CNS can lead to

A

memory failures

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16
Q

lesion studies in cortex conclusion

A

no specific place in cortex where memories are stored

17
Q

loss of memory function is due to the

A

size of lesion

18
Q

certain cortical lesions can destroy

A

types of memories

19
Q

removal of medial temporal lobes

A

hippocampus , amygdala and surrounding cortex

anterograde amnesia - unable to consciously remember any new events

20
Q

diencephalon damage

A

korsakoffs syndrome - degeneration of neurons in nuclei of the thalamus and in the maxillary bodies

21
Q

what are the effects of korsakoffs syndrome

A

suffered from retrograde amnesia - lost past memories
anterograde amnesia
learn new skills

22
Q

inability to forget can cause

A

memory problems just as severe as the inability ti remember

23
Q

all experiences

A

shape our brains

24
Q

post traumatic stress disorder

A

memory of a traumatic experience does not fade overtime but begins to dominate the persons life
flashbacks, concentration problems

25
why do some people develop PTSD
hormones
26
hormones
chemicals generated by the endocrine system | transported in to the brain via the bloodstream
27
specific hormones are associated
with different emotional states | eg stress and arousal - adrenaline and noradrenaline
28
stress hormones affect
memory
29
experimental evidence that stress hormones affect memory
2 groups - pictures presented with exciting story or neutral story pictures were remembered better if pairs with an exciting story when given an noradrenalin antagonist this effect disappeared
30
amygdala
crucial for emotional memories
31
experimental evidence for amygdala
lesion of amygdala - animals appear over sexed but emotionally flat can no longer learn a fear response
32
amygdala is directly connected to the
hypothalamus
33
biological model of PTSD
stress- traumatic experience, processed by amygdala, activates hypothalamus, activates ES, releases adrenaline and noradrenalin improves memory of stress- keeps activating amygdala