lecture 7 Flashcards

1
Q

gene environment interaction

A

environment can change the way genes are expressed

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2
Q

genetic code contains

A

instructions on how to respond to the environment

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3
Q

how do nervous systems change

A

neural activity changes synaptic connections
fire together wire together- strengthen
out of sync lose your link - weaken

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4
Q

synaptic activity - baseline activity

A

neural activity without stimulation

action potentials are generated randomly

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5
Q

neural signalling

A

change in activity relative to baseline level

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6
Q

action potential can either become

A

more forewent than baseline (excitation) or less frequent (inhibition)

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7
Q

persisent signalling

A

causes the synapse to change

increased activity causes short term molecular changes

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8
Q

frequent APS cause an increase in

A

neurotransmitter release and number of receptor molecules

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9
Q

increased neural activity can also cause

A

long term structural changes

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10
Q

long term structural changes

A

sustained activity - frequent AP’S can cause
growth of new synapses
synaptic takeover

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11
Q

how do changes affect behaviour

A

optimising existing behaviour
increased transmission rate
react more quickly , reliably to important changes in the environment

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12
Q

decrease transmission rate

A

makes you more likely to ignore unimportant changes in the environment

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13
Q

acquiring new behaviour growth of new synapses

A

combine info from previously unrelated sources

respond to old stimuli in a new way

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14
Q

synaptic takeover

A

reroute info to new pathways

respond to old stimuli in a new way

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15
Q

damage to CNS can lead to

A

memory failures

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16
Q

lesion studies in cortex conclusion

A

no specific place in cortex where memories are stored

17
Q

loss of memory function is due to the

A

size of lesion

18
Q

certain cortical lesions can destroy

A

types of memories

19
Q

removal of medial temporal lobes

A

hippocampus , amygdala and surrounding cortex

anterograde amnesia - unable to consciously remember any new events

20
Q

diencephalon damage

A

korsakoffs syndrome - degeneration of neurons in nuclei of the thalamus and in the maxillary bodies

21
Q

what are the effects of korsakoffs syndrome

A

suffered from retrograde amnesia - lost past memories
anterograde amnesia
learn new skills

22
Q

inability to forget can cause

A

memory problems just as severe as the inability ti remember

23
Q

all experiences

A

shape our brains

24
Q

post traumatic stress disorder

A

memory of a traumatic experience does not fade overtime but begins to dominate the persons life
flashbacks, concentration problems

25
Q

why do some people develop PTSD

A

hormones

26
Q

hormones

A

chemicals generated by the endocrine system

transported in to the brain via the bloodstream

27
Q

specific hormones are associated

A

with different emotional states

eg stress and arousal - adrenaline and noradrenaline

28
Q

stress hormones affect

A

memory

29
Q

experimental evidence that stress hormones affect memory

A

2 groups - pictures presented with exciting story or neutral story
pictures were remembered better if pairs with an exciting story
when given an noradrenalin antagonist this effect disappeared

30
Q

amygdala

A

crucial for emotional memories

31
Q

experimental evidence for amygdala

A

lesion of amygdala - animals appear over sexed but emotionally flat
can no longer learn a fear response

32
Q

amygdala is directly connected to the

A

hypothalamus

33
Q

biological model of PTSD

A

stress- traumatic experience, processed by amygdala, activates hypothalamus, activates ES, releases adrenaline and noradrenalin improves memory of stress- keeps activating amygdala