Exam 3 - Oral Cavity & Esophageal Disorders Flashcards

1
Q

what are the major similarities between actinomyces bovis & actinobacillus ligniersii in regards to pathogenesis?

A

both are common inhabitants of the oral cavity

an oral wound then allows access to other soft tissue/bony structures in the mouth allowing for spread

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2
Q

what may predispose a cow to developing wooden tongue?

A

rough feedstuff

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3
Q

what is the etiology of wooden tongue?

A

actinobacillus ligniersii - granulomatous infection of the tongue primarily seen in cattle

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4
Q

what is the pathogenesis of wooden tongue?

A

oral wound allows the bacteria access - infection begins at the base of the tongue & moves forward

may infect other soft tissues of the oral cavity

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5
Q

what are the clinical signs associated with wooden tongue?

A

anorexia, abnormal chewing, ptyalism, enlarged firm tongue, & regional lymphadenitis

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6
Q

how is wooden tongue diagnosed?

A

gram stain of aspirate/biopsy - gram negative rods +/- sulfur granules

clinical signs & response to treatment

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7
Q

what is the prognosis of wooden tongue?

A

excellent

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8
Q

what is the treatment used for wooden tongue?

A

sodium iodide IV, antibiotics - oxytetracycline, florfenicol, etc

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9
Q

what agent is seen on this sample?

A

actinobacillus ligniersii

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10
Q

what is the etiology of lumpy jaw?

A

actinomyces bovis, gram positive rod, common inhabitant of the oral cavity

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11
Q

what is the pathogenesis of lumpy jaw?

A

oral wound allows agent access & leads to osteomyelitis of the jaw preferentially affecting the mandible before the maxilla

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12
Q

what are the clinical signs of lumpy jaw?

A

initially - painless bony swelling on the mandible/maxilla

later - periosteal response, fibrous tissue reaction, draining tracts, decreased appetite, abnormal/difficulty chewing, ptyalism, weight loss, & may cause other infections in the head/esophagus/forestomach

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13
Q

what agent likely caused this?

A

actinomyces bovis

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14
Q

how is lumpy jaw diagnosed?

A

gram stain of pus - gram positive filamentous branching rods +/- sulfur granules

clinical signs

radiography - osteomyelitis, tooth root involvement, & pathologic fractures

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15
Q

what agent is seen on this?

A

actinomyces bovis

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16
Q

what is the treatment used for cows with lumpy jaw?

A

consider culling

sodium iodide IV, antibiotics (PPG, florfenicol, isoniazid - animals can’t enter food chain)

curette & flush bony lesions & draining tracts

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17
Q

what is the prognosis for animals with lumpy jaw?

A

prognosis can be good if caught early in infection but becomes poor later in infection

18
Q

tooth root abscesses are most common in what animals? what teeth? when may it occur?

A

camelids - mandibular molars!!!!

after fighting teeth have been removed

19
Q

what are the clinical signs associated with tooth root abscesses in camelids?

A

can be asymptomatic

swelling in mandible/maxilla, +/- sinusitis, & may have draining tracts

20
Q

how are tooth root abscesses diagnosed?

A

oral exam & rads - bony lysis of lamina dura, periodontal sclerosis, & periosteal reaction

21
Q

what is seen in the mandible of this radiograph?

A

tooth root abscess

22
Q

what is the treatment used for tooth root abscesses?

A

antibiotics for facultative anaerobic bacteria - 60% recovery

surgical treatment - tooth extraction

23
Q

what is the etiology of balling gun injury?

A

aggressive use of instruments in the mouth - more likely to occur in cases when the animal isn’t restrained well

24
Q

what are some clinical signs of a balling gun injury?

A

anorexia/dysphagia, fever, hypersalivation, laryngeal edema, peripharyngeal swelling/pain, bad breath, inspiratory dyspnea, & aspiration pneumonia

25
how is a balling gun injury diagnosed?
history, physical/oral exam, endoscopy, & rads
26
what is the treatment used for balling gun injury?
antibiotics, NSAIDS, IV fluids, avoiding coarse feeds, trach & rumenostomy if needed
27
what animals are common offenders of choke?
camelids
28
what is the etiology of choke?
obstruction of the esophagus
29
what are some common anatomical sites of choke?
cranial portion of the cervical esophagus thoracic inlet base of the heart
30
what are the big clinical signs of choke in camelids? what are some other general signs?
anxiety, swinging of the head/staggering, & waves of motility in cervical esophagus free gas bloat, dysphagia, & salivation
31
how is choke diagnosed?
physical exam, pass a stomach tube, endoscopy, & rads
32
what are some major differentials for choke?
RABIES, megaesophagus (camelids), tetanus, botulism, balling gun injury
33
what is the treatment used for choke?
wear gloves!!!!! remove the obstruction - cervical esophagus & distal obstructions, gently massage!!!! bloat should resolve once choke is antibiotics & correct fluid/acid-base abnormalities
34
what treatment can be used for stubborn obstructions in choke?
trochar/temporary rumen fistula, sedation, & anesthesia
35
what clinical signs should you watch for in secondary esophageal dysfunction associated with choke?
anorexia, regurgitation, hypersalivation, & recurrence of choke
36
what should you do with animals with esophageal dysfunction?
cull or consider a rumenostomy
37
what is the etiology of megaesophagus?
idiopathic or sequelae of choke
38
what is the pathophysiology of megaesophagus?
chronic distention leads to poor aboral motility
39
what clinical signs are associated with megaesophagus?
esophageal waves, anorexia, dysphagia, & respiratory signs - increased rate/effort & abnormal sounds
40
how is megaesophagus diagnosed?
history, endoscopy, & rads
41
what condition is seen in this rad?
megaesophagus in a camelid
42
what is the treatment used for megaesophagus?
avoid coarse feeds & pelleted feeds elevate the feed, feed them alone, & slow the intake of food by putting obstructions in their bowls (tennis balls)