Vestibular Rehab Flashcards

1
Q

peripheral pathologies

A
  • labyrinth related
  • BPPV
  • vestibular neuritis
  • labyrinthitis
  • acoustic neuroma
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Central vestibular pathologies

A
  • brain related
    -CVA
    -MS
    -Cerebellar disorder
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

anatomy of labyrinth

A

-semicircular canals:
- vertical: anterior and posterior canals
- lateral: horizontal canal
-otolith organs: saccule, utricle

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

BPPV
causes
pathomechanism

A

-benign, paroxismal (comes and goes) positional vertigo
- head spins when change positions
- causes: head trauma, infection, vestibular weakness, advancing age
- otoconia is displaced from the macula of utricle

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

canalithiasis vs cupulothiasis

A
  • canalithiasis: <1min. stuck in canal
  • cupulothiasis: persists in duration >1 min, stuck in cupula
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

BPPV signs and sxs

A

Vertigo:
- when bending or getting up
- when changing positions in bed
- change in head position

Nystagmus:
- nonvoluntary, repetitive and rapid eye movements
- most important symptom!

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Dix Hallpike test

A

(A) The patient’s head is turned (45 degrees)
toward her affected ear while she is in a sitting
position.

(B) The patient is quickly moved into a supine
position with her head extended (20-30 degrees
off the table) and rotated 45 degrees toward her
ear.

Posterior Canal - Upbeating torsional Nystagmus
Anterior Canal - Downbeating torsional Nystagmus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Epley maneuver aka Canalith Repositioning Maneuver

A

A series of four head positions – Maintain each position for 1 to 2 minutes or until the vertigo and nystagmus has
stopped to ensure otoconia low through the canal.
1. (A and B) 45° to the more symptomatic side and 30° below horizontal (the Dix-Hallpike exam position)
2. (C) Rotate 45° to the other side keeping 30° declination
3. (D) Roll to sidelying (uninvolved side), nose down
4. (E) Slowly sit up, maintaining head position flexed (chin tucked) and rotated
Slowly return the head to upright and remain sitting 3-4 minutes, then repeat until no symptoms are seen

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Semont or Liberatory maneuver for cupulothiasis

A

Turn head to opposite side!
The head is
rotated 45° to the left side. (B) With assistance, the patient is then moved from
sitting to right side-lying and stays in this position for 1 minute. (C) The patient is
then rapidly moved 180°, from right side-lying to left side-lying. The head should
be in the original starting position, left rotated (nose down in final position) in
this example. Note that the otoconia have been dislodged from the cupula.
After 1 minute in this position, (D) the patient returns to sitting.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Brandt Daroff Exercise

A
  • generic exercise: for HEP or if pt cant tolerate CRM
  • Start sitting up. Turn head to 1 side (doesnt matter which)
  • Side lye on oppostie side
  • Sit up, turn head other direction, side lye on contra side
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Supine roll test

A
  • For horizontal canal
  • Supine with head in 20deg cervical flexion
  • Turn head to one side
  • Look for nystagmus and sxs
  • Geotrophic beating: canalithiasis
    side of more intense sxs is affected
  • Ageotrophic beating: cupulothiasis
    side with less intense sxs is affectd
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

BBQ roll/canalith repositioning maneuver for Horizontal canal

A
  • for horizontal canalithiasis and cupulothiasis
    -Head in 20deg flexion during test
  • Begin in supine
  • turn head 90deg to affected side
  • back to neutral
  • turn head 90deg to contralat side
  • Roll into prone
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What assessments to do for vestibular function

A

Cranial nerve and coordination
Eye assessments: smooth persuits, saccades, VOR (head impulse test)
- need to refer if central lesion is present

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

smooth persuits

A
  • follow finger with eyes
  • CN3, central
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

saccades

A

-CN3, central
- overshoot/undershoot is abnormal

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

VOR

A
  • vestibulo-ocular reflex
  • responsible for maintaining focus on image during rapid head movements
  • VOR must generate rapid compensatory eye movements in direction opposite head rotation
17
Q

Head impulse (thrust) test

A
  • Testing for VOR
  • turn head while they fixate on your nose
  • if unable to do it while head is turned to 1 side, that side is positive
18
Q

Central vestibular pathology characteristics

A
  • ataxia
    • smooth persuits and saccades
  • diplopia, other red flags, and systemic sxs
  • pendullar nystagmus (eyes oscillate at equal speeds
  • vertical nystagmus
    -Visual fixation doesn’t help
  • ## usually, no hearing loss
19
Q

Peripheral vestibular pathology characteristics

A
  • Visual fixation helps to decrease sxs
  • tinnitus, hearing loss, fullness in ears
  • negative saccades and smooth persuits
  • Jerk nystagmus: (nystagmus will incorporate slow and fast phases) - fast to positive side, and slow to negative side
20
Q

labyrinthitis

A
  • inflammation of labyrinth
  • +HIT
  • sudden onset of nausea, vertigo, and vomiting
  • tinnitis, and hearing loss
  • lasts for days to weeks
  • oscillopsia, disequilibrium, postural instability
21
Q

vestibular neuritis

A
  • inflammation of nerve
    • HIT
  • sudden onset of nausea, vertigo, vomiting
    -lasts days to weeks
22
Q

Meniére’s disease

A
  • overproduction of fluid within inner ear > increase in pressure > vertigo
    -aural fullness (fullnes in ears)
  • tinnitis, hearing loss
  • lasts for minutes to hours
  • vertigo
  • want to use diuretics/decrease salt intake to get fluid out
23
Q

acoustic neuroma/vestibular schwannoma

A
  • slow growing tumor that develops from balance and hearing nerves supplying inner ear
  • gradual sxs
  • facial weakness and sensory loss due to being close to CNVII
  • hearing loss, tinnitus
  • vertigo
  • loss of balance
24
Q

Unilateral vestibular hypofunction interventions

A
  • gaze stability: improves VOR, ex VORx1, VORx2
  • postural stability and balance
  • habituation: fo movement that is hard for them until they can do it
25
Q

VORx1 vs VORx2

A

1: target is stable, and head moves
2: head and target moves

26
Q

bilateral vestibular hypofunction interventions

A
  • gaze stability: only VORx1
  • DONT do VORx2 - too hard for them unless asymmetrical involvement
  • imaginary targets
  • walking