Periodontology tutorials Flashcards
What are the signs of periodontal stability?
- BoP < 10%
- PPD < 4mm
- No bleeding on probing @ 4mm sites
What are the signs of patient engagement in perio treatment?
- BoP < 30%
- Plaque < 20%
or > 50% improvement in both
What are 3 ideal outcomes of periodontal treatment?
- Reduce inflammation and pocket depths
- Regain attachment of long junctional epithelium
- Reducing rate of progression of alveolar bone loss
Based on SDCEP criteria, What proportion of >5mm pocket depths would a patient have following successful periodontal treatment?
0
What is the difference in quality of debridement and efficacy between ultrasonic scalers and hand scalers?
If done with correct technique, there is no difference
What is the definition of a non responding site?
Pocketing that remains the same depth following non-surgical instrumentation
Give 4 treatment options for non responding sites
- Continued non surgical instrumentation - re-attempt step 2
- Surgical debridement through surgical access to pocket
- Resective surgery
- Regenerative surgery
What is periodontal access surgery ?
Surgical debridement of full depth of pocket which cannot be accessed with non-surgical instrumentation
What is resective surgery?
Removal of pocketing by changing archtecture of hard and soft tissues to achieve shallow probing depths
What is regenerative surgery?
Adding material into infrabony or furcation defects to regenerate attachment
What is the rationale behind step 3?
To treat non-responsive sites to step 2 with
* targeted repeated non surgical instrumentation
* To consider the use of invasive surgical techniques to gain access for subgingival PMPR in deep pockets
* Regenerate or resect areas
What is a residual pocket?
- A deep pocket that persists after carrying out initial periodontal therapy ( step 1 and step 2)
What PPD is a deep residual pocket?
> 6mm
What PPD is moderately deep residual pocket?
4-5mm
In a stage III periodontitis patient with residual pockets after the first and second step of periodontal therapy, when is it recommended to take access flap surgery instead of repeated subgingival PMPR?
> 6mm
If referral/expertise is not an option, what is the minimum level of primary care required for management of residual pockets associated with/without infrabony defects or furcation involvement after completion of step 1 and step 2 of periodontal therapy? (2)
- Repeated subgingival instrumentation ( with or without access flaps)
- Regular supportive periodontal care than includes subgingival instrumentation
A patient with poor engagement with OHI wishes to undertake periodontal surgery. What is the recommendation for this?
This is a contraindication as surgical interventions need excellent OH compliance
What is the adequate management of residual deep pockets in Stage III after step 1 and step 2
Resective surgery
What is the adequate management for residual deep pockets associated with infrabony defects (>3mm) after step 1 and step 2 ?
Step 3 - targeted non surgical sub-gingival instrumentation / Refer to secondary care
Management of molars with Class 2 or 3 furcation involvement and residual pockets after step 1 and step 2 ?
- Step 3 - targeted non surgical subgingival instrumentation / Refer to secondary care
- Furcation involvement alone is not an indication for XLA
Management options for maxillary interdental class II furcation involvement after step 1 and step 2 ?
- Step 3 involving non surgical instrumentation
Refer for - regenerative surgery
- root resection
- access surgery
Management options for class III furcation involvement after step 1 and step 2?
Step 3
* non surgical instrumentation
* access surgery
* tunneling
* root resection
What is the choice of regenerative biomaterials for promoting healing of residual deep pockets associated with a deep infrabony defect? or class II molar buccal furcation involvement?
Regenerative surgery with
* barrier membranes (deprotinised bovine bone mineral or collagen)
* enamel matrix derivative
What is gingivitis?
Presence of bleeding on probing, erythema and oedema, with no loss of attachment or bone loss
What is chronic periodontitis?
Loss of gingival and periodontal tissues from current or historic chronic inflammation of the periodontium
What is necrotising ulcerative gingivitis?
Immunosuppression resulting in oral commensal bacteria causing necrosis and ulceration of the gingival tissues with no bone loss
What is necrotising ulcerative periodontitis?
Immunosuppression resulting in oral commensal bacteria causing necrosis and ulceration of the gingival tissues with bone loss
What is a periapical abscess?
A periapical pathology with pus accumulation associated with an infected tooth
What os a periodontal abscess
A pathology associated with localised accumulation of non draining pus within the gingival wall of periodontal pocket associated with rapid tissue destruction
What is an endo-perio lesion?
- Pathologic communication between pulpal and periodontal tissues
- Stage 4 grade C generalised periodontitis
- complaining of pain in LLQ
- History of deep pocketing in posterior quadrants
- Multiple >6mm pockets in LLQ
- You take clinical photographs and a PA of 36
Describe the clinical presentation?
Swelling of region associated with LL6, absence of erythema or bleeding on visual inspection
- Stage 4 grade C generalised periodontitis
- complaining of pain in LLQ
- History of deep pocketing in posterior quadrants
- Multiple >6mm pockets in LLQ
- You take clinical photographs and a PA of 36
Describe the radiographic presentation?
Deep pocketing associated distal root of 36, large >3mm infrabony defect distal of 36
- Stage 4 grade C generalised periodontitis
- complaining of pain in LLQ
- History of deep pocketing in posterior quadrants
- Multiple >6mm pockets in LLQ
- You take clinical photographs and a PA of 36
What special tests could you use to help diagnosis?
- Mobility
- Sensibility testing
- Stage 4 grade C generalised periodontitis
- complaining of pain in LLQ
- History of deep pocketing in posterior quadrants
- Multiple >6mm pockets in LLQ
- You take clinical photographs and a PA of 36
What may have caused the patient issue?
Previous PMPR resulted in reattachment of junctional epithelium which trapped bacteria deep in the pocket, long term proliferation of non-draining pus associated with periodontal infection causing significant localised bony destruction
- Stage 4 grade C generalised periodontitis
- complaining of pain in LLQ
- History of deep pocketing in posterior quadrants
- Multiple >6mm pockets in LLQ
- You take clinical photographs and a PA of 36
What are the underlying risk factors?
- Deep pocketing
- Periodontitis patient
- Poor access to site for cleaning
- Impaction of food
- Diabetes
Using the appropriate classification, what is the differential diagnosis?
Periodontal abscess