peds96 Flashcards

1
Q

exanthem in rubella

A

non-pruritic, maculopap, and confluent; begins on face, spreads to the trunk, and extremities, and lasts 3-4 days

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2
Q

complications of rubella

A

meningoencephalitis; polyarteritis; congenital rubella syndrome (most serious complic)

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3
Q

congenital rubella syndrome

A

maternal infection during the first trimester; fetal anomalies occur in 30-50% of infected fetuses; thrombocytopenia, jaundice, and purpura (blueberry muffin baby”)

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4
Q

blueberry muffin baby

A

congenital rubella syndrome

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5
Q

fetal anomalies in congenital rubella syndrome

A

congenital cataracts and PDA; sensorineural hearing loss and meningoencephalitis

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6
Q

late complications of congenital rubella syndrome

A

mental retardation, type 1 dm, and autoimmune thyroid disease

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7
Q

management of rubella

A

supportive

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8
Q

aspergillosis

A

invasive disease only in severely immunocomp patients;

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9
Q

management of aspergillosis

A

high dose antifungal like amphotericin B and surgery to resect the aspergilloma

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10
Q

allergic bronchopulmonary aspergillosis

A

wheezing, eosinophila, and pulm infiltrates; happens in patients with chronic lung disease (CF)

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11
Q

management of allergic broncopulmonary aspergillosis

A

corticosteroids, and in some cases antifungal therapy

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12
Q

common reason that candida would go

A

use of antibiotics allows it to flourish

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13
Q

invasive candida growth in immunocomp patients may cuase what

A

fungemia, meningitis, osteomyelitis, and endophthalmitis

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14
Q

treatment for invasive candida

A

antifungal

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15
Q

foung in the soil in the southwestern united states fungus

A

coccidiodomycosis

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16
Q

how does coccidio infect you?

A

inhaled into the lungs

17
Q

clinical features of coccidio infection

A

most are asymp or cause a mild pneumonia; but immunocomp may get disseminated disease (sever pneumonia, meningitis, and osteomyelitis)

18
Q

management of coccidio infection

A

none in mild disease; in disseminated disease, systemic antifungal

19
Q

where is cryptococcus found?

A

in the soil

20
Q

how is crypto infection acquired

A

inhaled into lungs

21
Q

clinical features of crypto infection

A

most infections asymp; in immunocomp, spread to the CNS (cryptomeningitis is an AIDS defining illness)

22
Q

management of crypto infection

A

systemic antifungal therapy

23
Q

amebiasis

A

infection by the protozoan entamoaeba histolytica

24
Q

how do you acquire amebiasis

A

ingestion of the cyst in contaminated food or water

25
Q

when do sx appear in amebiasis infection?

A

1-4 weeks after ingestion when the trophozoite emerges from the cyst and invades the colonic mucosa

26
Q

where is amebiasis present?

A

worldwide, but highest incidence in developing nations

27
Q

clinical features of amebiasis

A

most patients are asymp; symp intestinal disease may range from mild colitis to severe dysentery

28
Q

symptoms of the diarrhea of amebiasis

A

cramping abdominal pain, tenesmus, and diarrhea that may contain blood or mucus;

29
Q

complications of amebiasis

A

intestinal perforation, hemorrhage, strictures, and local inflamm mass called ameboma

30
Q

extraintestinal amebiasis

A

abscess (most commonly in the liver), although it may form in the brain, lung, or other organ

31
Q

diagnosis of amebiasis

A

ID of the trophozoites or cyst in the stool; colonoscopy with biopsy or serum antibody assays may also be used

32
Q

management of amebiasis

A

metronidazole, along with a luminal amebicide like iodoquinol

33
Q

how is giardia infection acquired

A

fecal-oral when the cyst is ingested

34
Q

classic patient for giardia

A

drinking contaminated mountain water in the western unitted states; there are also daycare center outbreaks

35
Q

clinical features of giardia infection

A

range from asymp to explosive diarrhea; may persist for 2-6 weeks

36
Q

voluminous, watery, foul-smelling diarrhea

A

giardia