Magasjúkdómar - Deniz Flashcards
Upper GI system - The upper gastrointestinal tract consists of;
- mouth
- pharynx
- esophagus
- stomach
- duodenum
Gastroesophageal Reflux Disease
(GERD)
- It is chronic symptom of mucosal damage
- Caused by reflux of stomach acid into the lower esophagus
- HCL acid and pepsin cause esophageal irritation and inflammation (esophagitis)
Gastroesophageal Reflux Disease
(GERD) - Etiology (orsök)
- Incompetent Lower Esophageal Sphincter (LES)
- Normally, LES is antireflux barrier
- Incompetent LES lets gastric contents move from stomach to the esophagus
when patient is supine or has an increase intraabdominal pressure - Factor effecting LES pressure
- Alcohol
- Chocolate
- Drugs
- Fatty foods
- Nicotine
- Peppermint
- Tea, coffee
Gastroesophageal Reflux Disease
(GERD) - Symptoms (einkenni)
- Heartburn (pyrosis)—lower sternum—spreads upward to the throat or jaw,
burning sensation - Relieve with antacids
- Dyspepsia and regurgitation
- Dyspepsia (is another word for indigestion); feelings of stomach pain, over-
fullness and bloating during and after eating - Regurgitation; hot, bitter, or sour liquid coming into the throat or mouth
- Respiratory symptoms; wheezing, coughing, and dyspnea
- Disturbed sleep patterns
- Hoarseness, sore throat, hypersalivation, choking
GERD: Nursing Management
1- Lifestyle Modifications
2- Drug therapy
3- Nutritional Therapy
4- Surgical Therapy
5- Endoscopic therapy
1- Lifestyle Modifications
- The head of bed is elevated 30 degrees (with pillow or blocks)
- Pt should not be supine for 2 to 3 hours after a meal
- Encourage patients who smoke to stop
- Stress management– if stress cause symptoms
- Nutritional advices
2- Drug Therapy - the focus of the drug therapy
- Decreasing the volume and acidity of reflux
- Improving LES function
- Increasing esophageal clearance
- Protecting the esophageal mucosa
2 - Drug Therapy - The most common and effective treatments
- Proton pump inhibitors (PPIs)
- Histamine (H2) receptor blockers
3-Nutritional Therapy
- No specific diet is used to treat GERD
- Avoiding some food; chocolate, peppermint, fatty foods, coffee, and tea
- Tomato-based products, orange juice, cola, red wine may irritate the esophagus
- Avoid late dinner, nighttime snacking and milk
- Small, frequent meals and drinking fluids between meals, helpful
- Weight reduction if the pt is overweight
4- Surgical Therapy
- Nissen and Toupet fundoplication—most
common antireflux surgeries - After surgery reflux symptoms should
decrease - Recurrence is possible
- If the pt has mild dysphagia, pt should
report it - LINX Reflux Management System
- LINX system can cause difficulty
swallowing, nausea, and pain when
swallowing food. - Pt Education: should not have MRI—can
cause serious harm
Peptic Ulcer Disease (PUD)
- PUD is characterized by discontinuation in the inner
lining of the gastrointestinal (GI) tract because of
gastric acid secretion (HCL) or pepsin - It extends into the muscular layer of the gastric
epithelium. - It usually occurs in the stomach and proximal
duodenum - It may involve the lower esophagus, distal
duodenum, or jejunum.
Peptic Ulcer Disease
(PUD)
Etiology ( orsök )
1- Helicobacter Pylori
-H. pylorus is a gram-negative bacillus
-Found within the gastric epithelial cells
-This bacterium is responsible for 90% of
duodenal ulcers
and 70% to 90% of gastric ulcers.
-H. pylori infection is commonly acquired during
childhood
2-Medication-Induced Injury
- Nonsteroidal anti-inflammatory drugs (NSAID) use is the second most common cause of
PUD after H. pylori infection.
- non-steroidal anti-inflammatory agents, such as ibuprofen, aspirin, naproxen
- NSAIDs increase gastric acid secretion
- Reduce the integrity of mucosal barrier
3- Lifestyle Factors
High alcohol intake
Smoking
Coffee
Psychologic distress (stress and depression..)
Peptic Ulcer Disease (PUD)
Gastric Ulcers:
- more prevalent: Women and over 50 years of age
- can occur in any part of the stomach
- Less common
- H.pylori, NSAIDs, bile reflux are the main risk factors
Peptic Ulcer Disease (PUD)
Duodenal ulcers:
Duodenal ulcers
* 80% of all the peptic ulcers
* Incidence is high: 35-45 years of age
* H.pylori is most common risk factor
Peptic Ulcer Disease (PUD)
Clinical manifestations - Gastric Ulcer
- Discomfort—after 1-2 hours after meals– epigastrium
- Pain is like—burning, gaseous
- Perforation symptoms
Peptic Ulcer Disease (PUD)
Clinical manifestations -Duodenal Ulcer
- Symptoms occur 2-5 hours after meal—midepigastric region
- Pain is like—burning, cramplike
- Can also cause back pain
Peptic Ulcer Disease (PUD)
Diagnose (greining)
- Endoscopy
- Biopsy– tissue specimens for H.pylori
- Serology, stool and breath test—H.pylori
- Barium contrast study
- Lab. Tests; CBC—shows anemia, liver enzyme – detect liver problems (in terms of
ulcer threatment), stool test—blood
Peptic Ulcer Disease (PUD)
Management - Conservative Care:
Adequate rest, drug therapy, smoking cessation, dietary
modifications, and long-term follow-up
* Drug Therapy: antibiotic therapy, proton pump inhibitors, cytoprotective drug therapy
(sucralfate), adjunct drugs (H2 receptor blockers and antacids)
* Nutritional Therapy:
Peptic Ulcer Disease (PUD)
Management - Surgical therapy:
If the conservative care is not effective or there is complication
Peptic Ulcer Disease (PUD)
Nursing Management - Planning
- Adhere to the prescribed therapeutic regimen
- Reduction or absence of discomfort
- Have no signal of GI complications
- Have complete healing of the peptic ulcer
- Make appropriate lifestyle changes to prevent recurrence
Peptic Ulcer Disease (PUD)
Nursing Management - Nursing Diagnosis:
(example) Acute pain, lack of knowledge, nausea
Peptic Ulcer Disease (PUD)
Nursing Management - Nursing Assessment:
data obtained from pt
Peptic Ulcer Disease (PUD) - Nursing Management - Nursing Implementation:
- Health Promotion: Early diagnose
- Acute Care:
- If NPO-explain importance
- Mouth care is important
- Check analyze results
- Record input and output
- Vitals
- Rest, pain killer (needed)
- Follow complication signs: hemorrhage, perforation, gastric outlet obstruction
(such as; increased nausea or vomiting, increased epigastric pain, bloody emesis or
tarry stool)
Stomach (Gastric) Cancer -
Etiology (orsök)
- Begins with a nonspecific mucosal injury because of infection (H.pylori),
autoimmune-related inflammation, repeated exposure to irritants such as bile or
NSAIDs, and tobacco use. - Diet: smoked foods, salted fish and meat
- H.pylori—early age
- Atrophic gastritis
- Pernicious anemia
- Adenomatous polyps
- Smoking and obesity
- Heredity