Lecture 14: Equine Nutrition 2 (Exam 2) Flashcards

1
Q

What are the predisposing factors or Cribbing

A
  • Stall weaning v. pasture weaning
  • Stall housing following weaning
  • Feeding concentrates
  • Lack of time @ posture
  • Lack of straw bedding stall
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2
Q

What are some ways to treat/prevent cribbing

A
  • Increase dietary fiber
  • Remove ledges
  • Electric fence
  • Dog collar
  • Cribbing collar
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3
Q

Who most commonly gets gastric ulcers? Where do gastric ulcers commonly occur on the stomach

A
  • Perfomance horses & foals
  • In the non glandular stratified squamous mucosa
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4
Q

What are the risk factors of gastric factors

A
  • Intense exercise
  • Increased stall time
  • Meal feeding
  • Not having hay ava ad libitum
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5
Q

What are ways to prevent/treat gastric ulcers

A
  • Feeding alfalfa can be a protective measure (has protein & calcium to buffer gastric pH)
  • Feeding aloe vera juice for protection & treatment
  • Corn oil to increase the pH
  • Can use antacids but not very effective
  • Have roughage continuously ava to stimulate saliva
  • Avoid large grain meals
  • Reduce stress & increase turnout
  • Over the counter or prescribed for treatment
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6
Q

What is heaves

A
  • An allergic respiratory dx of horses
  • Recurrent airway obstruction
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7
Q

What should be done when feeding horses w/ heaves

A
  • Avoid dusty moldy hay
  • Feed cubes
  • Soak hay (leaches out nutrients, decreases the sugar content by 30% & potassium w/ >50% reduction)
  • Avoid hay storage above horses in the barn
  • Pasture the horse
  • Use hay gain systems
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8
Q

Describe vitamin E deficiency

A
  • Can be an issue w/ poor quality hay & grain (may be devoid of vitamin)
  • Can cause equine degenerative myeloencephalopathy or equine lower motor neuron dx
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9
Q

What happens in equine degenerative myeloencephalopathy

A
  • Can be hereditary
  • Intracellular damage via oxidation
  • Ataxia & spasticity of all four limbs w/ the hind limbs often most infected
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10
Q

Describe Equine lower motor neuron dx

A
  • Older horses deficient in vitamin E for 18 M or longer (risk peaks @ 16 yo)
  • Neurodegenerative disorder of the horse
  • Progressive weakness, muscle wasting, & weight loss
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11
Q

Describe esophageal obstruction/choke

A
  • Food is stuck in the esophagus around the thoracic inlets or behind the base of the heart
  • Not uncommon if horse doesn’t chew properly
  • sx: extended neck & nasal discharge
  • Commonly seen w/ pelleted feed stuffs, cubs, beet pulp, older horses w/ poor dentition
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12
Q

Describe Leukoencephalomalacia (moldy corne disease)

A
  • Fumonisin toxicity (mycotoxin) causes neuro signs
  • Liquefactive necrosis of the cerebral white matter
  • Causes disruption of BV, progressive ataxia, depression, anorexia, delirium, coma, & death
  • Death can occur 12 H to 1 W after onset of signs
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13
Q

What are some problems associated w/ bermuda pastures

A
  • Can rapidly become fibrous & cause impaction colic
  • Coastal bermuda can cause ileal impactions in horses
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14
Q

What are problems associated w/ endophyte fungus in endophyte infected fescue

A
  • Poor growth
  • Increased foal mortality
  • Prolonged gestation or thickened placenta
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15
Q

What pasture grass is linked to dev of liver disease in horses

A

Alsike clover

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16
Q

What produce slaframine

A

Red clover that is contaminated w/ rhizoctonia leguminicola

17
Q

How does high concentrate diet/grain overload cause laminitis

A
  • High levels of NSC in forages (sugars, fructans, & starch)
  • Up to 30 to 50% of NSC reaches the LI where they are rapidly fermented (AKA to march starch in the LI)
18
Q

What can high starch fermentation lead to

A
  • Diarrhea
  • Colic
  • Laminitis
  • Release of endotoxins which damage the mucosa in the intestines
19
Q

How is laminitis caused by high concentrate diets prevented

A

By limiting concentrate feed to a max of 0.5% of BW

20
Q

How do large amounts of starch reaching the LI change the local gastric envi

A
  • Lowers intestinal pH
  • Lowers pH (more gram neg bacteria dies while gram pos grow & produce lactic acid)
21
Q

What occurs when there is excessive NSC in the LI

A
  • Overgrowth of bacteria that ferment starch & fructans (produces lactic acid & lowers pH)
  • Destruction & inflammation of the gut wall
  • Damages allows absorption of toxins
  • Causes laminitis
22
Q

What should ESC & WSC be when a horse has laminitis

A
  • ESC: should be low
  • WSC: Should be low
  • ESC + starch: Be below 10%
23
Q

What are fructans

A

Storage carbs in growing grasses

24
Q

When do fructans accumulate

A

When the rate of photosynthesis exceeds use of energy for growth in plants

25
Why are fructans dangerous
* Not digested by mammalian enzymes * Escape the SI & are rapidly fermented in the LI * They are more dangerous than starch
26
How many g of fructans can pastures accumulate
> 400 g fructans/ kg DM
27
How can pasture associated laminits be avoided
* Restrict grazing (esp in the spring) * Supplement w/ hay made from mature grasses or legumes containing hay * Reduce the NSC content of hay * Analyze grasses and hay * Avoid obesity
28
How can the NSC content of hay be reduced
* Can be reduced by ~ 30% * Soak in water for 30 to 60 mins
29
@ what % NSC can contribute to issues in problem horses
NSC > 12%
30
What percent of laminitis is associated w/ GIT disease
55.7%