La Ortho - Severe Lameness /emergency Flashcards
Types of orthopedic emergencies
Foot abscess
Fracture
Joint lux
Septic synovial structure
Deep soft tissue abscess
Flexor disruption
Navicular bursa nail
Wounds
Mechanical
Neuro
Muscular
Colic
Initial assessment key factors
Severity of lameness matching observed injury
What structures are involved? Soft tissue, open/closed, synovial structures, joints?
Brief cardiovascular/hydration
Twitch /sedation
Imaging modalities
Radiology
Ultrasound
Nuclear scintigraphy
Computed tomography
Magnetic resonance imaging
Radiography
Used to evaluate bone, soft tissue
Limited to 2-d imagine, 30-50% of changes in bone density required to be visible
Superimposition of structures
How many days does no displaced fractures take to become visible on a radiograph ?
10-14 days
Focal demineralization occurs with what pathogenesis
Infection
Osseous cyst like lesions
Chronic pressure
OC defect
Neoplasia
Sclerosis
Response to chronic mechanical stress/inflammation
Types of contrast radiography
Venogram, arthrogram, bursogram, fistulogram
Modalities for laminitis prognosis
Venogram
Usefulness of scintigraphy
Early detection of bone disease
Localizing lesion prior is not important - scintigraphy is useful for localizing lesions
Ct scan
Useful, removing 2-d dimension aspect
Most common causes of severe lameness
Foot abscess
Fracture
Laminitis
Synovial sepsis
Periarticular cellulitis
Foot abscess
If abscess is caused by nail, leave nail in for radiography
Observed heat, pain,m swelling, drainage
Laminitis
Shifting weight, observable stance, elevated DP
Synovial sepsis
Effusion + periarticular edema + heat
- NOT cellulitis
Severe lameness
Synoviocentesis
Considerations with soft tissue /acute lacerations
Blood flow to effected area
Nerve damage
Ligament /tendon damage
which tendons have a worse prognosis if they’re cut or damaged
Flexor tendons - they take the majority of weight bearing
Differentiating lameness from neurological disease
Neuro horses may have
Muscle atrophy
Cranial nerve deficits
Cutaneous sensory deficits
Muscle fasciculations
Ataxia
Abnormal posture
Altered skin temp /sweat patterns
Abnormal head/neck movement
Limb knuckling, dragging, stumbling
Weakness
Racehorse breakdown injuries
Combo of injuries = loss of stability
Equine fracture repair factors
Location/config
Blood supply
Soft tissue damage
Contamination
Key for equine fracture repair
Early recognition
- immediate stabilization, to reduce secondary damage
- compliance from owner & horse
- behavior of animal
Laminitis prevention
Sole support
Rockered toe
Cryotherapy
- can develop due to contralateral limb issues
Importance of timing of surgery
Field radiography
Apply split first