Cerebellum Flashcards

exam 1

1
Q

cerebellum are connected by

A

cerebellar peducles

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2
Q

inferior cerebellum is found in the ______ and mostly contains ____ to cerebellum

A

found in the medulla and mostly contains inputs to cerebellum

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3
Q

Middle cerebellum found in ______

`and contains only ____ to cerebellum

A

found in pons and only input to cerebellum

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4
Q

superior cerebellum found in ______ and contains mostly _____ from cerebellum

A

found in midbrain and mostly contain output from cerebellum

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5
Q

lesions in cerebellar leads to

A

ipsilateral deficits

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6
Q

does the cerebellum contain more neurons than the cerebral cortex?

A

heck yea

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7
Q

what is vulnerable for occlusion?

A

4th ventricle

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8
Q

Mollaret’s triangle

A

is a sel-contained circuit involving the inferior olive, dentate and red nucleus

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9
Q

climbing fibers from the inferior olive go where?

A

direct to the purkinje fibers

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10
Q

what is the order in which cells receives the input in the cerebellum?

A

internal granular (packed with cells) —> Molecular —>Purkinje

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11
Q

most incoming fibers synapse on

A

granular cell dendrites

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12
Q

what are the deep cerebellar nuclei?

A

output nuclei

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13
Q

vermis involved with

A

trunkal control

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14
Q

flocculondular lobe?

A

vestibulocerebellum

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15
Q

vestibulocerebellum receives input t (and its output is to) ________ and when lesioned it affects

A

input from vestibular nuclei and when lesioned it affects balance and gait ataxia

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16
Q

spinocerebellum receives input (and its output is to) _______from and when lesioned it affects

A

receives input from spinocerebellar tract and olives and when lesioned it affects trunkal coordination

17
Q

Neocerebellum input is from _______ and output is ________

A

input: cortex via pontine nuclei
output: back to cortex via VL of thalamus

18
Q

neocerebellum lesioned affect

A

upper extremities dyscoordination

19
Q
vascular syndrome  of the cerebellum that are caused by"
- hemorrhage/infarct
-toxic exposure
- heat stroke
anoxia
A

acute cerebellar disease

20
Q

Subacute cerebellar disease:

  • alcoholic–>
  • Praneoplastic –>
  • Cerebellar tumors –>
  • MS –>
A
  • alcoholic–> vermis
  • Praneoplastic –> antibodies vs. purkinje cells leading to cerebellar ataxia
  • Cerebellar tumors –> occlusion of the 4th ventricle
  • MS –> cerebellum white fibers
21
Q

inherent disease of the cerebellum that can be caused by:

  • congenital cerebellar hypoplasia
  • genetic disorders like spinocerebellar ataxia
  • neurodegenerative like olivopontocerebellar degeneration that has parkisonian like features
  • metabolic diseases
A

chronic cerebellar disease

22
Q

How does the cerebellum differ in its representation of the body?

A

ipsilateral strcuture

23
Q

what does this mean for input and output?

A

remains ipsilateral or crosses twice

24
Q

what is carried in each peduncle?

superior:

middle:

inferior:

A

superior: output
middle: cortico-ponto-cerebellar fibers
inferior: spinocerebellar, vestibulocerebellar

25
Q

How is the cerebellar cortex organized?

A

input to granular, synapse on purkinje cell in molecular

Purkinje cell to deep nuclei

26
Q

what are the functional divisions?

A

lateral, vermis and paravermis, flocculonodular

27
Q

Clinical symptoms of cerebellum issues

A
  • nystagmus
  • incoordination, intention tremor
  • dysdiadochokinesis
  • hypotonia- flaccid tone
28
Q

dysdiadochokinesis

A

rapid, altering movements

29
Q

lesion within the mollaret’s triangle

A

palatal myoclonus