Acute Abdomen Flashcards by Elizabeth Sager

Q

What are the 2 main chemicals at work in the lumen of the stomach

A

Hydrochloric Acid from parietal cells

Pepsin (from pepsinogen secreted by chief cells)

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

What do hydrochloric acid and pepsin do in the lumen of the stomach

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

What are the 3 hormones that stimulate parietal cells

A

Gastrin, Histamine, & acetylcholine

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

What do gastrin, histamine, & acetylcholine do

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

What is the site of most small intestine absorption

A

duodenum

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

What is responsible for most of the chemical digestion and regulates the rate of stomach emptying

A

duodenum

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

What are the hormones of the small intestine

A

Secretin & cholecystokinin

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

What to secretin and cholecystokinin do

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

What is responsible for absorbing remaining water from undigested food matter, transporting undigested food, and absorbing vitamins produced by bacteria

A

large intestine

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

List the hormones of the pancreas (islets of langerhans)

A

insulin, glucagon, & somatostatin

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

What is insulin produced by and what does it do

A

produced by beta cells to decrease blood glucose levels

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

what is glucagon produced by and what does it do

A

produced by alpha cells and increases blood glucose levels

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

what is somatostatin produced by and what does it do

A

produced by delta cells and suppresses release of GI hormones

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe conjugated bilirubin

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe unconjugated bilirubin

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the etiology & RFs for esophagitis

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the clinical presentation of esophagitis

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the diagnostic testing of esophagitis

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the treatment for esophagitis

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the etiology of Boerhaave’s syndrome

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the presentation of Boerhaave’s syndrome

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the diagnostic testing for boerhaave’s syndrome

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the treatment for boerhaave’s syndrome

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the etiology of mallory-weiss tear

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the presentation of mallory-weiss tear

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the diagnostic testing for a mallory-weiss tear

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the treatment of a mallory-weiss tear

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the etiology & RFs of esophageal varices

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the presentation of esophageal varices

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the diagnosis & treatment for esophageal varices

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the presentation of distal/diffuse esophageal spasm

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the diagnostic testing for distal/diffuse esophageal spasm

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the treatment for distal/diffuse esophageal spasm

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

list the definition and etiology of gastritis

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

describe the presentation of gastritis

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the diagnostic testing and treatment for gastritis

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the etiology of peptic ulcer disease

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the presentation of peptic ulcer disease

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the diagnostic testing and treatment for peptic ulcer disease

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe zollinger ellison syndrome

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe proton pump inhibitors (list 3, MOA, side effects, interactions)

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe H2 receptor antagonists (list 3, MOA, side effects, interactions)

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe misoprostol use, MOA, CIs

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe antacid MOA

A

neutralizes acid and prevents pepsinogen to pepsin conversion

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe bismuth compounds (pepto bismol)

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe sucralfate

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the etiology and epidemiology of pyloric stenosis

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the clinical presentation of pyloric stenosis

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the diagnosis and treatment of pyloric stenosis

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the etiology & RFs for cholecystitis

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the presentation of cholecystitis

A

How well did you know this?

1

Not at all

2

3

4

5

Perfectly

Q

Describe the diagnostic testing for cholecystitis

A

Q

Describe the treatment for cholecystitis

A

Q

Describe the definition, pathophys, & RFs of acute acalculous cholecystitis

A

Q

Describe the presentation of acute acalculous cholecystitis

A

Q

Describe the diagnosis and treatment of acute acalculous cholesystitis

A

Q

Describe the definition & etiology of acute ascending cholangitis

A

Q

Describe the presentation of acute ascending cholangitis

A

Q

Describe the diagnosis of acute ascending cholangitis

A

Q

Describe the treatment for acute ascending cholangitis

A

Q

Describe choledocholithiasis

A

Q

Describe the presentation of choledocholithiasis

A

Q

Describe the diagnosis & treatment of choledocholithiasis

A

Q

Describe the etiology & risk factors for hepatic vein obstruction (budd chiari)

A

Q

Describe the presentation of hepatic vein obstruction

A

Q

Describe the diagnosis & treatment of hepatic vein obstruction

A

Q

Describe the risk factors for acute viral hepatitis

A

Q

Describe the presentation of acute viral hepatitis

A

Q

Describe the diagnosis & treatment of acute viral hepatitis

A

Q

Describe the definition and risk factors for fulminant hepatitis

A

Q

Describe the presentation of fulminant hepatitis

A

Q

Describe the diagnosis of fulminant hepatitis

A

Q

Describe the treatment for fulminant hepatitis

A

Q

Describe the etiology and risk factors for cirrhosis

A

Q

Describe the presentation of cirrhosis

A

Q

Describe the diagnosis & treatment for cirrhosis

A

Q

Describe the definition & etiology of spontaneous bacterial peritonitis

A

Q

Describe the presentation of spontaneous bacterial peritonitis

A

Q

Describe the diagnosis and treatment of spontaneous bacterial peritonitis

A

Q

Describe the etiology of acute pancreatitis

A

Q

Describe the presentation of acute pancreatitis

A

Q

Describe the diagnosis of acute pancreatitis

A

Q

Describe the treatment of acute pancreatitis

A

Q

what are these indicative of

A

acute pancreatitis

Q

Describe some things that would cause abnormal LFTs

A

Q

Describe the etiology & presentation of anorectal abscess & fistula

A

Q

Describe the treatment and complication of anorectal abscess & fistula

A

Q

Describe the definition and etiology of anal fissure

A

Q

Describe the presentation and treatment of anal fissure

A

Q

Describe the treatment of diarrhea

A

Q

Describe fecal impaction

A

Q

Describe constipation treatments

A

Q

Describe the etiology and presentation of splenic rupture or laceration

A

Q

Describe the diagnosis and treatment of splenic rupture and laceration

A

Q

Describe the toxic ingestion of acetaminophen

A

Q

Describe the toxic ingestion of salicylates

A

Q

Describe the toxic ingestion of bases

A

Q

Describe the toxic ingestion of hydrocarbons

A

Q

Describe the toxic ingestion of iron

A

Q

Describe the toxic ingestion of anticholinergics

A

Q

Describe the toxic ingestion of cholinergics

A

Q

Describe the etiology and presentation of button battery ingestion

A

Q

Describe the diagnosis and treatment for button battery ingestion

A

Q

Describe the etiology & presentation of a small bowel obstruction

A

Q

Describe the diagnosis & treatment of a small bowel obstruction

A

Q

Describe the etiology, RFs, & presentation of duodenal atresia

A

Q

Describe the diagnosis & treatment of duodenal atresia

A

Q

Describe the etiology, RFs, & presentation of intussusception

A

Q

Describe the diagnosis & treatment of intussusception

A

Q

Describe the etiology & presentation of diverticulitis

A

Q

Describe the diagnosis & treatment of diverticulitis

A

Q

Describe the etiology & presentation of volvulus

A

Q

Describe the diagnosis & treatment of volvulus

A

Q

Describe the diagnosis & treatment for appendicitis

A

Q

Describe the etiology of acute mesenteric ischemia

A

Q

Describe the presentation of acute mesenteric ischemia

A

Q

Describe the diagnosis & treatment of acute mesenteric ischemia

A

Q

Describe the etiology & presentation of ischemic colitis

A

Q

Describe the diagnosis & treatment of ischemic colitis

A

Q

Describe the etiology & presentation of toxic megacolon

A

Q

Describe the diagnosis & treatment of toxic megacolon

A

Q

Describe the etiology, presentation, diagnosis, & treatment of ulcerative colitis

A

Q

Describe the etiology, presentation, diagnosis, & treatment of chron’s disease

A

Q

Describe the etiology & presentation of incarcerated hernias

A

irreducible

painful enlargement of hernia that can’t be reduced (n/v if bowel obstruction)

surgical

Q

Describe the etiology & presentation of strangulated hernias

A

compromised blood supply = surgical emergency

ischemic with systemic toxicity

Q

Describe the etiology & treatment of epiploic appendagitis

A

Q

Describe the etiology & presentation of appendicitis

A

Q

Describe the etiology & pathophysiology of distal/diffuse esophageal spasm

A