:) Flashcards

1
Q
A
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2
Q

which is this muscle?

innervation?

movement [2]

A

which is this muscle: sartorius

innervation: femoral nerve

movement [2]: flexes hip AND knee

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3
Q

what are the 3 different muscles that insert at the medial aspect of the knee? [3]

which compartment are they all originally from? [3]

what is name for this meetin of three muscles? [3]

A
  • Sartorius - anterior
  • Gracilis - medial
  • Semitendinosus - posterior

= pes anserinus !!

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4
Q

what are the 3 articulations of knee joint? [3]

which is bone is not part of the knee joint? !!! [1]

A

what are the 3 articulations of knee joint? [3]
–lateral femoral and tibial condyles with corresponding meniscus
–medial femoral and tibial condyles with corresponding meniscus
–patella and femur

fibula not part of knee joint!!

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5
Q

what are the 3 articulations of knee joint? [3]

which is bone is not part of the knee joint? !!! [1]

A

what are the 3 articulations of knee joint? [3]
–lateral femoral and tibial condyles with corresponding meniscus
–medial femoral and tibial condyles with corresponding meniscus
–patella and femur

fibula not part of knee joint!!

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6
Q

what is the Q line?

where is at a line between? [2]

what s the angle in men? (compared to vertical) [1]
whats the angle in women? (compared to vertical) [1]

A

Q line: asis –> centre of patella

what s the angle in men: 14 degress
whats the angle in women: 17 degrees

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7
Q

whats it called when have a small q angle?
whats it called when you have a large q angle?

which condyle does this cause increased presssure on for small q [1] / large q [1]?

A

whats it called when have a small q angle: genu varum - medial condyle
whats it called when you have a large q angle: genu valgum - lateral condyle

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8
Q

whats it called when have a small q angle?
whats it called when you have a large q angle?

which condyle does this cause increased presssure on for small q [1] / large q [1]?

A

whats it called when have a small q angle: genu varum - medial condyle
whats it called when you have a large q angle: genu valgum - lateral condyle

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9
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A
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10
Q

what is a sesamoid bone? [1]

A

what is a sesamoid bone: bone that develops in ligament

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11
Q
A
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12
Q

reflex test of patella: tests which nerve? [1] & which spinal segments[1]

A

reflex test of patella: tests femoral nerve and spinal segments L2-L4

causes contraction of quads

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13
Q

what are the names of the collateral ligaments of knee? [1]
how do they differ? [1]
which one is more prone to damage? [1]

A

what are the names of the collateral ligaments of knee? [2]
medial/ tibial collateral ligament & fibula collateral ligament

how do they differ? [1]

  • *fibula collateral ligament:** seperate to the knee capsule
  • *tibial collateral ligament:** part of the knee capsule

which one is more prone to damage? [1]
tibial collateral ligament bc if damage the knee it also damages the ligament

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14
Q
A
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15
Q

what is the role of the cruciate ligaments?

A

connecting the tibia and the femur to prevent displacement of the tibia relative to the femur

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16
Q

what is the medial menisci attached to [2] (anteriorly / posteriorly)

what is the lateral menisici attached to? [1]

A

what is the medial menisci attached to [2]

  • *anteriorly: ACL
    posteriorly: tibial collateral ligament**

what is the lateral menisici attached to? [1]
pcl
NOT ATTACHED TO LATERAL COLLATERAL LIGAMENT

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17
Q

which structures make up the unhappy triad? [3]

A
  • Medial meniscus
  • ACL
  • Tibial collateral ligament
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18
Q

lower motor neurons innervate skeletal muslces (alpha-motor neurones)

where do you find the cell body of the motor unit of the NMJ? [1]

one alpha motor neuron innervates: [1]

  • one muscle fibre
  • a number of muscle fibres
A

where do you find the cell body of the motor unit of the NMJ? [1]
ventral (anterior) horn of the spinal cord

one alpha motor neuron innervates: [1]

  • one muscle fibre
  • *- a number of muscle fibres:** forms the motor end plate (presynaptic NMJ)
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19
Q

label A

A
20
Q

what happens after Ach binds to Ach receptor?

A
  • Ach binds to Ach-receptor
  • opens Ach-receptor
  • allows Na channel to open & Na goes through
  • causes depolarisation of muscle membrane
  • causes release of Calcium from sarcoplasmic reciticulum
    … other steps…
  • muscle contraction
21
Q
A
22
Q

Where do you find:

L type calcium channels [2]

N type calcium channels [1]

A

L type calcium channels [2]
heart
vascular smooth muscle

N type calcium channels [1]
pre-synaptic terminals - very close to the vesicles

23
Q

explain mechanism of docking at NMJ and release of Ach occurs at presynaptic vesicle

A

vesicles docks by:

  • synaptobrevin interacts with syntaxin and SNAP25: holds the vesicle close to pre-synaptic membrane (but doesnt fuse) = docking.
  • Ca2+ binds to synaptotagmin: interacts with SNAP25/ syntaxin complex and tightens interaction between the vesicle and presynaptic membrane complexs & causes it to merge & release of Ach = confirmational change occurs.
24
Q

what does quantal release of Ach mean? [1]

A

every vesicle contains same amount of ACh: same amount of NM is released with each AP. get a 1:1 transmission of nerve & muscle.

25
Q

what is nicotinic Ach receptor structure like? [2]

how many Ach have to bind before the Ach-receptor opens? [2]

what is an end plate potential? [1]
at what point does an AP occur in muscle? [1]

A

what is nicotinic Ach receptor structure like? [2]

- 5 subunits: 2 identical alpha subunits, 1 beta, 1 delta (& 1 gamma or epsilon)

how many Ach have to bind before the Ach-receptor opens? [2]
2

what is an end plate potential? [1]
when Na+ move into the muscle & cause depol

at what point does an AP occur in muscle? [1]
- 40mV

26
Q

what are the two types of NMJ blockers [2] explain them

A

What are the two types of NMJ blockers [2]:

1. D tubocuraine: non-depolarising muscle relaxant
blocks the nicotonic Ach-R & prevents AP occuring

  • Acetylcholinesterase inhibitor works as an antidote to it
  • *2. depolarisng muscle relaxant**
  • stimulate Ach-R like Ach and activate muscle (muscle twitch)
  • but **do not detach: no more Ach can bind (paralysis)
  • even if membrane is repolarised (from Na channels / NaKATPase), the drug causes it to bedesensitised**
  • but breaks down after a while, so not perm. paraylses
27
Q

what are the two types of NMJ blockers [2] explain them

A

What are the two types of NMJ blockers [2]:

1. D tubocuraine: non-depolarising muscle relaxant
blocks the nicotonic Ach-R & prevents AP occuring

  • Acetylcholinesterase inhibitor works as an antidote to it
  • *2. depolarisng muscle relaxant**
  • stimulate Ach-R like Ach and activate muscle (muscle twitch)
  • but **do not detach: no more Ach can bind (paralysis)
  • even if membrane is repolarised (from Na channels / NaKATPase), the drug causes it to bedesensitised**
  • but breaks down after a while, so not perm. paraylses
28
Q

Where is T1 dermatome? [1]

where is T2 dermatome? [1]

A

Where is T1 dermatome? [1]
medial forearm

where is T2 dermatome? [1]
axillary forearm

29
Q
A
30
Q
A
31
Q

what is the cubital fossa’s boundaries? [3]

A
32
Q
A
33
Q
A
34
Q

which muscles bring about flexion of MCP and extension of interphalangeal joints?

which innervation?

A

lumbricals - 1/2 ulnar / 1/2 median

35
Q

what is the order of neurovasculature of femoral things? [3]

A

femoral artery = pulsating just belowing midinguinal point
femoral nerve = lateral to artery
femoral vein = medial

VAN

36
Q

what do you measure to find true length of lower limb [2]

A

ASIS –> distal tip of medial malleolus BOTH SIDES

37
Q
A
38
Q

what is difference between T1 & T2 imaging:

  • which one shows normal anatomy? pathology?
  • what do each show fat [1] & CSF [1] as?
  • which one is more sensitive to water content?
A

what is difference between T1 & T2 imaging?

  • which one shows normal anatomy?
    T1 = normal
    T2 = pathology
  • what do each show fat as?
    T1: fat = white, liquid; CSF = pale grey
    T2: fat = pale grey , CSF & fluid = white
  • which one is more sensitive to water content?
  • *T2 - good 4 oedema**
39
Q
  • which position is most stable for the a) hip b) knee (flex / extension)
  • where does the line of central pressure lead through the body? [4]
A

hip and knee both extended = most stable

•Centre of pressure is:
–in front of ankle / talus
–just in front of knee
–just behind hip
–just behind ear

40
Q

when locomoting:

which muscles prevent the pelvis dropping to the unsupported side? [1]
which muscles help to move the body weight? [1]

A

when locomoting:

which muscles prevent the pelvis dropping to the unsupported side: hip abductors (gluteus minimus & medius)
which muscles help to move the body weight: hip adductors

41
Q

how does is weight distributed in after going through verterbral body? [3]

what specifically enables use to undetake bipedal standing & walking more efficient? [1]

A

how does is weight distributed in after going through verterbral body?
•Weight of upper body transmitted centrally through vertebral column (1)
•Ilium transfers weight to femurs (2-3)
•Pubic rami form ‘struts’ or braces that maintain integrity of arch (4)

what specifically enables use to undetake bipedal standing & walking more efficient? [1]
Diagonal angle of femurs re-centres support directly under the body to make bipedal standing more efficient and to help walking. Quadripeds require simultaneous support from both sides.

42
Q

what is scissor gait:

A

•Thigh swings across body during swing phase

Difficulty in putting heel on ground
–Toe walking
–Unstable

43
Q

what is high stepping gait characterised by? [2]
which nerve damage causes this? [1]

A
  • dorsiflexor paraylsis - foot drop (unless leg is lifted higher by exaggerated knee bend)
  • common peroneal nerve injury
  • whole foot tends to be slapped on ground rather than heel strike
44
Q

what is staccator gait like?

A
  • plantaflexor paraylsis - no forward thrust
  • unaffected limb never advances beyond affected limb. just the knee flexors & hip flexors lift it up and swing it forwa
45
Q

when conducting a gait analysis, what do u look for? [5]

A

Gait analysis:
Balance
Co-ordination
Walking rhythm
Change of speed/direction
Stride length