Ventricular Dysrhythmias (Exam 2) Flashcards
Ventricular Dysrhythmias
Premature ventricular contraction (PVC)
Ventricular Tachycardia (V-tach)
Ventricular fibrillation (V-fib)
Premature Ventricular Contractions
Contraction coming from ectopic focus in the VENTRICLES
It comes earlier than QRS should come and does not follow normal rhythm or p-wave
Wide and distorted in shape compared to normal QRS
PVC’s causes
Stimulants
Electrolytes
Hypoxia
Fever
Exercise
Emotional Stress
CVD
How do we treat PVC’s
Treat the cause
PVC pharmacotherapy
Beta-blockers
Lidocaine
Amiodarone
If some PVC’s are up and some PVS’s are down
Multifocal PVC. More than one spot in the ventricle
Ventricular Tachycardia
Consists of 3 or more PVS together
Ectopic focus within the ventricles takes control and fires repeatedly –> no atrial contraction occurring
SERIOUSLY decrease cardiac output (not good perfusion)
Ventricular Tachycardia is associated with
Myocardial infarction
CAD
Electrolyte abnormalities
Heart failure
Drug toxicity
V-tach
Rate = 150 - 200 bpm regular
No p-wave evident
PR not measurable
How do we treat a patient with V-Tach?
ACLS— depend on pulse, patient will be symptomatic very quickly unless converts back to other rhythm
May need antidysrhythmic
-BB
-CCB
-Amiodarone
Electrolyte replacement
When a patient goes into Ventricular Tachycardia what is the next best action?
Check pulse
If V-tach isn’t treated the patient can slip into
Ventricular Fibrillation
Ventricular Fibrillation
Irregular waveforms of varying shapes and sizes
Ventricles just quivering
NO effective contraction = NO cardiac output
How do we treat ventricular fibrillation
CPR and ACLS / defibrillation
What is the difference between cardioverting a defibrillation
Cardiovert
-Elective procedure
-Awake and sedated
-Synchronized with R
-50-200 Joules
-Consent form
-EKG monitor
Defibrillation
-Emergency
-V-fib-V-Tach
-Begin with 200 Joules up to 360 Joules
-Clinet unconscious
-EKG monitor
