Classifications and Key Features of Dysrhythmias (Exam 2) Flashcards
Different ways to classify dysrhythmias
Early
Slow
Fast
Premature complexes (Early)
Bradydysrhythmias (Slow)
Tachydysrhythmias (Fast)
Another way to classify dysrhythmias (Site of origin in heart)
Sinus
Atrial
Ventricular
Bradydysrhythmias
HR < 60 (sa node)
Bradydysrhythmias can be significant because
-Myocardial oxygenation demand is reduced (good)
-Coronary perfusion time may be adequate because of prolonged diastole (good) (coronary arteries are better perfused)
-Coronary perfusion may decrease if rate is too slow (bad)
Bradydysrhythmias may be tolerated
patients blood pressure is adequate
Bradydysrhythmias with inadequate BP may lead to
-Myocardial ischemia/infarct
-Dysrhythmias
-Hypotension
-Heart Failure
Tachydysrhythmias
HR >100
Tachydysrhythmias may be significant because
-Shortened diastolic time which shortens coronary perfusion time (bad)
-May Increasore CO and BP (good)
but if this is sustained
= decrease ventricular filling and decrease CO / BP (bad)
Increases workload on heart which increases myocardial demand (bad)
Coronary arteries are perfused during
diastole
If tachydysrhythmia is not tolerated it can lead to
-MI
-Dysrhythmias
-Hypotension
-Heart failure
Key Features of sustained tachy and brady dysrhythmias
-Angina
-Restlessness / anxiety / confusion
-Dizziness / syncope
-Palpitations (tachy)
-Pulse deficit (radial pulse less than apical pulse)
-SOA - tachypnea
-Pulmonary crackles
-Orthopnea (cant breath laying)
-S3 or S4 heart sounds (gallops)
-JVD
-Pale cool skin diaphoresis
-N/V
-Decrease urine output (no perfusion)
-Delayed cap refill
-Hypotension