Topic 4: Heart Dysrhythmias & ECG/EKG Flashcards

1
Q

What does the P wave represent?

A

atrial depolarization

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2
Q

What does the QRS complex represent?

A

ventricular depolarization and atrial repolarization

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3
Q

What does the T wave represent?

A

ventricular repolarization

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4
Q

P-R interval is how many seconds

A

0.12 - 0.20 sec (3 - 5 small squares)

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5
Q

QRS width is how many seconds

A

0.08 - 0.12 sec (2 - 3 small squares)

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6
Q

Q-T interval is how many seconds

A

0.35 - 0.43 sec

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7
Q

5 lead EKG placement

A

White lead - right sternum / clavicle area
Black lead - Left sternum / clavicle area
Red lead - Left lower thoracic area
Green lead - Right lower thoracic area
Brown lead - just below and to the right of the bottom of the sternum

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8
Q

normal rhytms are considered ______

A

sinus (originating from the SA node)

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9
Q

what conducts first in a “normal” heart

A

SA node

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10
Q

Sinus Rhythm (SR, NSR) characteristics

A

*P waves are present & appear at regular time intervals
*P wave rate is constant
*Each P wave is followed by a QRS complex
*QRS complexes are of normal width (slim)
*The intervals between QRS complexes are equal
*The heart rate is between 60 and 100 beats per minute

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11
Q

Sinus Tachycardia characteristics of ECG and rate

A

same a normal siuns rhythm EXCEPT HR is 101-180 bpm

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12
Q

s/s of sinus tachycardia

A

SOB, dyspnea, diaphoresis
decrease CO, decrease BP
rapid, regular pulse, possible palpitation

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13
Q

cause of sinus tachycardia

A

exercise, fever, pain, stimulants. hyper/hypovolemia, anxiety, and CVD (anemia, HF, or valvular heart diseases can increase the heart’s workload.), hypotension

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14
Q

treatment for sinus tachycardia

A

TREAT cause
VALSALVA MANEUVER
fever- antipyretic
reduce stimulant
reduce pain

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15
Q

drug therapy for sinus tachycardia

A

B-blocker
CCB
adenosine

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16
Q

sinus bradycardia characteristics if ECG and rate

A

same as normal sinus rhythm EXCEPT HR < 60 bpm

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17
Q

s/s of sinus bradycardia

A

fatigue, lightheadedness, syncope, symptomatic

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18
Q

cause of sinus bradycardia

A

vagal maneuver (bearing down)
> responsible for the “rest and digest” activities in the body.
Vagal maneuvers, such as bearing down (as if having a bowel movement), can stimulate the vagus nerve = a reduction in HR
meds (CCB, B-blockers)
> These drugs are often used to treat hypertension and certain heart rhythm disorders. They work by relaxing the muscles of your heart and blood vessels.
vomiting, hypothermia
> Forceful or repeated vomiting can also stimulate the vagus nerve, leading to a reflex decrease in heart rate.
common in trained athletes

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19
Q

treatment of sinus bradycardia

A

try to arouse the patient
head of the bed FLAT
fall preacutions
atropine

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20
Q

drug therapy of sinus bradycardia

A

ATROPINE only if showing decreased perfusion (cool, pale, clammy)

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21
Q

atrial fibrillation

A

rapid, random, ineffective contractions of the atrium

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22
Q

Causes of atrial fibrillation

A

CAD, HTN, HF (any underlying heart disease)
surgery

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23
Q

treatment/drug therapy for atrial fibrillation

A

CCB (Diltiazem!!!), B-Blockers, amiodarone, digoxin (all for rate control)
anticoagulants

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24
Q

what is a big concern with A-fib

A

BLOOD STASIS!! can lead to clots

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25
what drug is used for clots in A-fib
Warfarin
26
what is important to know about warfarin
monitor INR Vit K is the antidote moderate green leafy veggies
27
what needs to be checked before giving digoxin
apical pulse (60 or greater) toxicity: 0.5-2.0, visual changes, N/V, anorexia potassium below 3.5 increases the risk for toxixity
28
s/s of atrial fibrillation
usually asymptomatic if symptomatic: dizzy, faint, fatigue, SOB, chest pain, fluttering in chest, irregular pulse, change in LOC, dyspnea, asymptomatic or syncope
29
atrial fibrillation characteristics of ECG and rate
-absence or inconsistent (fibrillary) p wave -Appears irregular, often changes speed of the heart rate 350-600bpm
30
Transcutaneous Pacemaker
external pacemaker used as a temporary emergency measure for maintaining adequate heart rate
31
Cardioversion
restoration of a normal heart rhythm by electric shock
32
Transcutaneous Pacemaker & Cardioversion is used with what type of dysrhythmia
atrial fibrillation
33
normal digoxin range
0.5-2ng/mL
34
Premature Ventricular Contraction (PVC) characteristics of ECG and rate
underlying rhythm can be any rate, regualr or irregular, PVCs can occur at variable rates -wide and distorted QRS complex, Pwave barely visible
35
what is Premature Ventricular Contraction (PVC) caused by
*Caused by the hearts demand for O2 -stimulants (caffiene, alcohol, nicotine, epi, etc), electrolyte imbalance, hypoxia, fever, exercise, emotional stress
36
s/s Premature Ventricular Contraction (PVC)
*Almost always *asymptomatic, noticed on tele strip
37
Premature Ventricular Contraction (PVC) treatment:
treat the cause O2 therapy for hypoxia elecrolyte replacement
38
3 or more PVC's occur consecutively
indicates Ventricular Tachycardia
39
Ventricular Tachycardia characteristics of ECG and rate
*Ventricular rate is 150-250bpm *Regular to slightly irregular rhythm p wave and Pr interval not visible, ORS wide and distorted
40
causes of ventricular tachycardia
post MI, CAC, hypoxemia, electrolyte imbalance (decrease K and Mg), drug toxicity
41
s/s ventricular tachycardia
*asymptomatic if it lasts a short amount of time due to anxiety *If it happens for an extended amount of time death can occur
42
a patient in V-tach is a patient that is trying to....
CODE!
43
a patient in V-tach may or may not have
a pulse
44
treatments for a patient in V-tach: IV antidyrhythmics
-Procainamide -Lidocaine -Adenosine
45
what is done if the pulse is present in v-tach
assess VS, call rapid response, prepare to call code, give IV dysrhythmias
46
what is done if no pulse is present in v-tach
CALL CODE, begin CPR, early D-fib
47
v-tach with no pulse
defibrillation
48
v-tach with pulse
cardioversion
49
Torsades de Points
-Lethal heart rhythm. Occurs if QT interval is prolonged greater than 0.50 seconds......this is why it is importnt to monitor for a prolonged QT interval. -Treated with IV *magnesium sulfate*, cardioversion, correct causative factors
50
the quivering of te ventricles in v-tach means
NO CARDIAC OUTPUT, MEDICAL EMERGENCY IF THERE IS NO PULSE
51
cause of ventricular fibrillation
untreated v-tach, MI, hyperkalemia, electric shock, hypoxemia, acidosis, drug toxicity
52
s/s ventricular fibrillation
-Poor to no cardiac output -loss of consciousness -no pulse -BP and respirations leading to brain damage -death if not reversed
53
V FIB
D FIB
54
V tach treatment
*check pulse, Call a CODE, start CPR, & early defibrillation *Amiodarone, Epinephrine, Lidocaine *Best results if ACLS is started in the first 30 seconds
55
if a patient goes into cardiac arrest
-check pulse -Call a Code -Start CPR -Start ACLS when trained ACLS professional arrives (rapid response or code team)
56
defibrillator
a device that delivers an electric shock to the heart to restore its normal rhythm
57
Digoxin toxicity symptoms
nausea, vomiting, diarrhea, vision changes, arrythmias, electrolyte imbalance
58
asystole
flatline
59
rate of asystole
none
60
asystole is ___
FATAL- client is clinically DEAD (unresponsive, no respiration or heart beat)
61
causes of asytole
long cardiac Hx, HF, MI
62
treatment for asystole
NO DEFIBRILLATION (no pulse) -epi, atropone, CPR w/ ACLS