SPECONLEC_S1_L2 - 1st (37-72) Flashcards
Usually as a result of an industrial accident
Chemical Burns
Chemical Burns may occur with _____
househol chemical products
chemical burns are usually as a result of an ____
industrial accident
In chemical burns, ___ is greater than (>) __ products
alkali; acidic
alkali products cause a severe injury called ____
liquefactive necrosis
liquefactive necrosis results in (2) in adipose tissue and proteins
denaturing of proteins & saponification of adipose tissue
Inhalation injuries are classified as __, __, or __
carbon monoxide poisoning, heat, or smoke inhalation injuries
Inhalation injuries account for more than ____ of the burn related deaths per year
half
are classified as carbon monoxide poisoning, heat, or smoke inhalation injuries
inhalation injuries
one of the leading causes of death
inhalation injuries
carbon monoxide is bad because it is associated with ___ that impedes blood’s ability to carry oxygen to body tissues and vital organs to produce ATP
hemoglobin
Signs of inhalation injuries (HARCH FSH)
Facial burn
Singed nasal burn
Harsh cough
Hoarseness
Abnormal breath sounds
Respiratory distress
Carbonaceous sputum
Hypoxemia
burns to the airway can cause ___ that blocks the ___ into the lungs
swelling; flow of air
In pathophysiology, what is under the local response?
- zone of coagulation
- zone of stasis
- zone of hyperemia
Occurs at the point of maximum damage
zone of coagulation
outermost zone
zone of hyperemia
decreased tissue perfusion
zone of tissue
Zone of coagulation has IRREVERSIBLE ___ ___
tissue loss
Goal of zone of stasis
increase perfusion
In zone of hyperemia, tissue will recover unless there is __ __
severe sepsis
Pathophysiological Changes During Hypermetabolic/Hyperdynamic Phase of Burn (>48 hrs) in the brain
- Altered mental status
- Cerebral Edema
- Inc. Pain Response
Pathophysiological Changes During Hypermetabolic/Hyperdynamic Phase of Burn (>48 hrs) in the heart & circulation
- Tachycardia
- Inc. cardiac index
- Subclinical myocardial dysfunction
- Echocardiogram (Altered contractility)
- Inc SvO2
- Dec SVR
Pathophysiological Changes During Hypermetabolic/Hyperdynamic Phase of Burn (>48 hrs) in the kidney
- Inc golmerular filtration rate
- Dec tubular function
Pathophysiological Changes During Hypermetabolic/Hyperdynamic Phase of Burn (>48 hrs) in the lungs + inhalation injury
- Pulmonary Edema
- Bronchospasm, bronchorrhea
- Acute respiratory distress syndrome