OB Proper (Normal) Flashcards
Diagnosis of Pregnancy: Presumptive, Probable or Definitive
Cessation of menses
Presumptive
Diagnosis of Pregnancy: Presumptive, Probable or Definitive
Physical outlining of fetus within the uterus
Probable
Diagnosis of Pregnancy: Presumptive, Probable or Definitive
Positve pregnancy test
Probable
Diagnosis of Pregnancy: Presumptive, Probable or Definitive
Perception of Quickening by the mother
Probable
Diagnosis of Pregnancy: Presumptive, Probable or Definitive
Perception of fetal movement by an examiner
Definitive
Diagnosis of Pregnancy: Presumptive, Probable or Definitive
Ballottement
Probable
Diagnosis of Pregnancy: Presumptive, Probable or Definitive
Fetal heart action, recognition of embryo on UTZ
Definitive
HCG measuring, when can it be detected? when does it peak? when is the nadir?
Detected: 8-9 days after ovulation
Peak: 8-10 weeks
Nadir: 14-16 weeks (Williams), 18-20 weeks (topnotch)
Diagnosis of Pregnancy: FHTs
TV-UTZ: 5 weeks
Doppler: 10 weeks
Stethoscope: 17 weeks, almost all by 19 weeks
Complaints in Pregnancy:
Cause & Tx of varicosities
Increased venous pressure in the LE
Relaxing effect of progesterone
Tx: stockings, elevate legs
Complaints in Pregnancy:
Cause & Tx of Hemorrhoids
Increased water absorption -> constipation
Tx: warm soaks, stool softeners
Complaints in Pregnancy:
Cause & Tx of Stress incontinence
Pressure on bladder
Tx: Kegel exercises
Complaints in Pregnancy:
Cause & Tx of Headache
Due to increased estrogen
Tx: massage, ice pack
Complaints in Pregnancy:
Cause & Tx of Pica
Iron deficiency
tx: Treat the IDA
Complaints in Pregnancy:
Cause & Tx of Leukorrhea
Increased secretion of cervical glands, estrogen-induced
Common causes of fundal discrepancy
False discrepancy (more common): measurement error, error in calculation of AOG. True discrepancy: pathology of the fetus, amniotic fluid, placenta, uterine wall
10 Danger signs of pregnancy
Vaginal: Bleeding, fluid leakage
Abdominal: Persistent vomiting, uterine cramping, decreased FM, Epigastric pain
Others: dysuria, edema, headache, BOV
Chills and fever
Obstetric Milestones
NTD and Chromosomal abnormality screening
16-18 weeks
Obstetric Milestones
GDM screening & Rhogam administration
24-28 weeks
Obstetric Milestones
GBS screening and Leopold’s manuevers
35-37 weeks
Obstetric Milestones
FMC q6-q8
Start at 28 weeks
Indications for GBS prophylaxis
Previous infant with GBS infection GBS bacteruria Postive GBS screening Unknown GBS status and: 1) Delivery < 37weeks AOG 2) Membrane rupture > 18 hours 3) Intrapartum temp of > 38C
What makes a reactive NST?
2 or more accels within 20 minutes, peak at 15 bpm, lasting 15 seconds
What does a CST measure?
Uteroplacental function
5 Components of a BPP
FHR / NST Breathing Movements Tone AFI
BPP 8/10 with normal AFV
Normal Repeat weekly (2x/week for GDM Pxs)
BPP 8 w/ abnormal AFV
Chronic asphyxiated fetus
>37 weeks deliver
BPP 4-6
Possible fetal asphyxia
AFV abnormal: deliver
> 36 weeks: deliver
< 36 weeks: repeat after 24 hours
BPP 0-2
Almost certain fetal asphyxia
Deliver.
Doppler Velocimetry Measures
1) Most commonly used
2) Common nonreassuring finding
1) UA systolic-diastolic ratio
2) Absent or Reversed End Diastolic Flow
Single most important indicator of an adequately oxygenated fetus
Beat-beat variability
Etiology of Acceleration
Fetal movement
Etiology of Early deceleration
Head compression (vagal compression)
Etiology of Variable deceleration
Umbilical cord compression
Etiology of Late deceleration
Uteroplacental insufficiency
Cat. I on NST
Baseline FHR: 110-160
Mod. Variability
(-) variability / late decel
Cat. III on NST
Absent baseline FHR and
- Recurrent late/var decel
- Bradycardia
Sinusoidal pattern
Management of Reassuring Tracing
Intrauterine resuscitation: Decrease uterine activity Correct maternal hypotension Change maternal position High flow O2
Amnioinfusion: Treatment of variable or prolonged decelerations
Prenatal Diagnosis and Fetal Therapy
Candidates for prenatal diagnosis
1) elderly primi
2) >31 yo with mult gestation
3) Previous pregnancy with:
- an autosomal trisomy
- Triple X, or Klinefelter
4) Women with chromosomal abnormalities
5) Repetitive 1st trim abortions
6) Fetus with major structural defects
Most Common isolated fetal structural defect, 2nd
1st: Cardiac (VSD)
2nd: NTD (Family hx is most recognized risk factor)
MSAFP screening for NTDs
AFP: synthesized in yolk sac then fetal liver
Reported as multiple of median (MoM)
>3.5 MoM = increased fetal risk for NTD
Cranial Signs of NTD
1) Small BPD
2) Ventriculomegaly
3) Lemon sign - frontal bone scalloping
4) Banana sign - elongation and downward displacement of the cerebellum
5) Obliteration of the Cisterna Magnus
Components of the Quadruple Serum Marker
ACEI AFP Chorioinic gonadotropin Estriol Inhibin
Down’s Quadruple Serum Markers
Dec AFP
Inc Chorioinic gonadotropin
Dec Estriol
Inc Inhibin
Edward’s Quadruple Serum Markers
All DECREASED AFP Chorioinic gonadotropin Estriol Inhibin
Prenatal Test of Choice
Fetal Karyotyping in the second trimester
Second trimester amniocentesis
Prenatal Test of Choice
Associated with postprocedural pregnancy loss
Associated with talipes equinovarum
Early amniocentesis (11-14th weeks)
Prenatal Test of Choice
Need for early karyotyping
Less risks for deformities
Chorionic villus sampling (as early as 9th week)
Prenatal Test of Choice
Diagnostic assessment of Red cell anemia or alloimmunization
Cord blood sampling
4 Phases of Parturition
1) Quiesence
2) Activation
End: Onset of Labor
3) Stimulation
End: Delivery of Conceptus
4) Involution
End: Restoration of fertility
Key Points
Phase 1 Parturition
Myometrial quiesence PROGESTERONE: mediator Unyielding cervix Irregular low intensity contractions Braxton Hicks contractions
Mechanisms for Uterine Quiesence
Calcium sequestration
Inhibition of oxytocin receptor synthesis
Increased enzymatic degradation of uterotonics
Inhibition of contractile signal propagation
Key Points
Phase 2 Parturition
Increased uterine responsiveness to uterotonins
ESTROGEN: mediator
Cervical ripening
Increased frequency of painless contractions
6-8 weeks
Formation of the lower uterine segment
Differentiate between physiologic ring and the pathologic ring of Bandl.
Physiologic: separates lower and upper uterine segment
Pathologic: abnormal junction with extreme LUS thinning, increased risk for uterine rupture, common cause of obstructed labor.
Phase 3 Key Mediator:
Oxytocin (also for phase 4)
Minor: Prostaglandin, serotonin, histamine
Most common fetal lie
Longitudinal
Most common fetal presentation
~98% cephalic ~2.7% breech Transverse Face Brow
Characteristic fetal position
Fetus convex Head flexed, chin on chest Thighs flexed Legs bent on knees Arms crossed over thorax
Most common fetal position
Occiput Anterior (L > R)
Station is expressed as plus-minus values at the level of?
The Ischial spine
Key differences between true labor and false labor?
True labor: (+) cervical effacement and dilatation
Confirmatory tests for rupture of membranes:
(+) collection fluid in the vagina
Pool test
Confirmatory tests for rupture of membranes:
Takes advantage of the alkaline nature of the amniotic fluid
Nitrazine test
Confirmatory tests for rupture of membranes:
(+) crystallization under a microscoped
Fern test
4 parameters of the cervical exam?
Bishop score?
Effacement Dilatation Consistency Position Bishop score: add Station
How does one interpret the Bishop score
> 8 favorable
6-8 equivocal
<6 unfavorable