foot lesions Flashcards

1
Q

foot lesions are split into what causes? name the 4 most common foot lesions and tell me what their general cause is.

A

Digital dermatitis - infection

sole ulcer - claw horn

sole hemorrhage - claw horn

white line dz - claw horn

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1
Q

what is the cause of digital dermatitis?

A

Treponema spp (3 groups) + opportunistic/secondary bac t

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2
Q

what are the risk factors for digital dermatitis?

A

group of cattle housed together

unsure of transmission, maybe direct and fomites, maybe genetic component too

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3
Q

what is the main and possibly only reservoir of digital dermatitis?

A

lesions on infected animals

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4
Q

what are the C/S of digital dermatitis?

A
  • mild-severe lameness
  • interdigital area behind heel bulbs
  • small circular moist browny grey exudative areas of epidermal liquefaction
  • matting of surrounding hair
  • raw dermal granulation tissue if diphtheritic debris cleaned (painful!)
  • occasionally skin above 1 heel bulb only
  • interdigital space b/t claws, coronary band at front of foot, around accessory digits
  • papilliform or hairy warts
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5
Q

how is digital dermatitis classified? briefly explain it

A

M scale

M0 = healthy, normal
M1 = early, small, <2cm
M2 = acute, active, bright red/granulomatous, >2cm
M3 = healing stage w/i 1-2d after topical tx, where acute lesion has covered itself with a firm scab
M4 = late chronic lesions (dyskeratotic)
M4.1= chronic lesion w/ subacute components

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6
Q

how do you dx digital dermatitis?

A

clean and examine –> C/S

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7
Q

how do you tx digital dermatitis?

A

clean/dry, topical Ab (oxytetracycline) or bandage w/ Abs

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8
Q

what are the other names for sole ulcer?

A

pododermatitis, circumscripta

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9
Q

what are the causes of sole ulcers?

A
  • movement of P3, claw overgrowth, external pressure –> pinching of corium & germinal epithelium b/t P3 and hoof capsule
  • pinching around bony protrusion where flexor tendon attaches (caudal edge of pedal bone)
  • changes in structure/function of digital cushion –> poor/inadequate cushioning and formation of new bone on P3 (exacerbates problem)
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10
Q

tell me about mild, moderate, and severe sole ulcers

A

mild: blood in horn as sole is produced = sole hemorrhage

mod: partial/intermittent interruption of horn production at sole ulcer site

severe: horn production completely stops at sole ulcer site for long time

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11
Q

what are the C/S of sole ulcers/sole hemorrhage?

A
  • slight-severe lamenss
  • often both hind feet (one worse than the other)
  • predominantly on lateral claw of hindfeet, medial claw of forefeet
  • hemorrhagic discolouration to complete cessation of horn prod. at ulcer site
  • possible secondary infection
  • deep digital sepsis in extreme cases
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12
Q

how do you dx sole ulcers?

A

clean/trim foot, C/S are pathognomonic

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13
Q

how do you tx sole ulcer/sole hemorrhage?

A

corrective trimming + reduce height of affected claw and dish site

remove underrun horn and granuloma if present

apply foot block to sound claw + NSAIDs

parenteral ABs if infection

place on deep straw if severe

want to decrease pressure on pinched corium

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14
Q

what is the px of sole ulcers/sole hemorrhage?

A

good if simple, poor if multiple claws affected/severe/deep digital sepsis is present

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15
Q

tell me the pathogenesis of white line dz.

A
  • junction of sole + wall = area of weakness
  • causes similar to sole ulcers –> initial hemorrhage in white line, which may then separate
  • separation exacerbated by standing on concrete, dirt/stone impaction –> infection through to corium
  • white line seals over –> infection trapped –> pus produced expands within potential space –> under sole pain!
  • abscess forms under sole, infection can tract up wall, eventually pus breaks out, usually at heel or less commonly at coronary band
16
Q

what are the C/S of white line disease?

A
  • mod-severe lameness
  • lat claw more commonly affected
  • swelling = advanced ± large abscess
  • leg abducted to bear weight on medial claw
  • always on white line, more frequently on abaxial wall closest to heel, then going to tip of claw to axial side
17
Q

how do you dx white line dz?

A
  • pain if “tweak” claw
  • white line impaction and abscess when trimming/exploration
  • small pin prick sized holes to large areas of impaction on white line (ALWAYS BLACK)
18
Q

how do you tx white line dz?

A
  • trim/balance both claws
  • explore impacted white line
  • drain abscess
  • remove underrun sole
  • if tracked ip wall, remove entire section of wall
  • Ab dressing (max 48h)
  • foot block sound claw + NSAIDs
  • parenteral Abs if necrotic corium found
19
Q

in bovines, 90% of lameness is in the ____, and 90% of those lesions are in the ____.

A

foot, hind feet

20
Q

what is the other name for foot rot?

A

interdigital necrobacillosis

21
Q

what is the cause of interdigital necrobacillosis?

A

acute necrotising inflammation of interdigital skin –> F. necrophorum ± T. pyogenes, Strep AND damage to skin

22
Q

what are the C/S of interdigital necrobacillosis?

A
  • sudden onset
  • mod-sev lameness
  • swelling of ST above/around coronary band and between digits (digits forced apart)
  • swollen/hot/enflamed/painful
  • split in interdigital space which discharges pus/necrotic tissue
  • F. necrophorum SMELLS
23
Q

how do you dx foot rot?

A

C/S

24
Q

how do you tx root rot?

A

check interdigital space for FBs, wash/flush, debride, topical ABs, parenteral ABs (3-5d)

25
Q

what are the 2 tx options for septic arthritis?

A

amputation
facilitated ankylosis