Abomasal Displacement & Volvulus Flashcards
Define LDA
left displaced abomasum
Define RDA
right displaced abomasum
Define RTA
right torsion of the abomasum (used interchangeable with right abomasal volvulus)
define antro pexy
pexy of the antral portion of the abomasum
define pyloro pexy
pexy of the pylorus (not commonly done due to risk of stricture formation)
define abomasal pexy
pexy of the abomasum
define omental pexy
pexy of the greater omentum
LDAs are a _____ issue.
management
most abomasal displacements are ____s.
LDAs
DAs are most likely to occur… when? why?
at start of lactation, or 45 days after calving. because this is when peri parturient diseases take place
what signalments of cows are more likely to get DAs ?
- holsteins, Ayrshires, guernseys
- large abdominal cavities
- age 4-7 (multiparous)
- start of lactation, after calving
what the risk factors predisposing to DAs?
- spring szn
- rapid change in diet
- decreased exercise
- high concentrate diet
what diseases increase risk of getting DAs? tell me one word for each as to why this predisposes to DAs
- hypocalcemia - motility
- ketosis - appetite
- retained placenta - appetite
- metritis - appetite
- mastitis - appetite/motility
- endotoxemia - appetite
describe the pathophys of DAs
it is a multifactorial syndrome based on ____.
hypomotility
decreased appetite –> small rumen –> decreased motility –> abomasal atony –> microbial fermentation –> gas production –> displaced abomasum
tell me the pathway of the abomasum during an LDA
abomasum fills up with gas and. becomes distended, then slips under the rumen to the left and goes to the left
creates a partial pyloric outflow obstruction
tell me the pathway of the abomasum during a RDA
abomasum becomes gas distended, floats dorsally (it’s already on the R)
creates a partial pyloric outflow obstruction
DAs create what metabolic derangement? why?
metabolic alkalosis w/ paradoxic aciduria
HCl produced in abomasum, because of pyloric outflow obstruction, HCl is trapped in abomasum, so you get metabolic alkalosis.
tell me the pathway of the abomasum during an RTA
abomasum becomes gas distended, floats dorsally (it’s already on the right) – starts as a RDA
twists counterclockwise when viewed from behind
additional counterclockwise turn when viewed from above
RTAs may include the ____.
omasum
why are RTAs so much worse than RDAs?
RTAs create a complete pyloric outflow obstruction, while RDAs create a partial pyloric outflow obstruction
RTAs cause vascular compromise and potential tissue revitalization
true or false: an animal with signs of colic is never alright until morning, and usually requires immediate surgical intervention.
TRUE!!!!!!!
how do you diagnose an LDA/RDA/RTA? general
PE
- auscultation/percussion
- succussion
- rectal
can use BHBA test, stall side Ca test (v expensive), blood lactate
can technically do CBC/chem, but it would be ridiculous to (but values might show up on NAVLE)
tell me about the HR when cow has LDA/RDA/RTA
potentially normal for RDA/LDA
tachycardia for RTA
tell me about the rumination when cow has LDA/RDA/RTA
RDA: usually still present
LDA: usually still present. harder to hear b/c of rumen displacement by distended abomasum
RTA: absent
tell me about temp when cow has LDA/RDA/RTA
usually normal
tell me the C/S for LDA
decreased appetite –> decreased rumen motility
decreased milk production
hyperketonemia
feces normal or slightly softer
rectal temp = N
resp = N
pulse = N
rumen contractions decreased (Hard to hear)
tell me the C/S for RDA/RTA
edit: this is more for RTA!!
decrease appetite
feces absent or scant
sunken eyes
HR=elevated
weak pulse (poor venous return)
bilateral abdominal distention
ruminal stasis & bloat
colic
8th rib –> middle paralumbar fossa ping and succession
tell me about the bloodwork with an LDA
alkalotic
increase bicarb
hypochloremia
hypokalemia
hypoglycaemia
ketonemia
stress leukogram
tell me about the bloodwork with an RDA/RTA
hypovolemia
dehydration
hemoconcentration
hyperglycaemia
metabolic alkalosis
hypochloremia
hypokalemia
hypocalcemia
hyponatremia
paradoxic aciduria
what do you expect to hear upon auscultation and percussion with an LDA/RDA/RTA?
high pitched “hyper resonant” metallic ping (caused by gas)
where do you auscultate and percuss on PE?
above and below the line made from the tuber coxa to just above the olecranon
other than DAs, what conditions cause a “false ping”? how do you rule them out?
gas filled rumen on L, pneumorectum
do a rectal exam
why do we do a rectal exam last?
introduces air