CARDIOVASCULAR SYSTEM Acute Coronary Syndromes MYOCARDIAL INFARCTION/ Unstable Angina Flashcards

1
Q

What’s the diff between stable and unstable angina?

A

stable= u know what brings it on

unstable= u don’t know what brings it on (MI, STEMI, NSTEMI)

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2
Q

Acute Coronary Syndromes MYOCARDIAL INFARCTION/ Unstable Angina is caused by a decreased blood flow to the myocardium that leads to _____, and ____.

A

ischemia and necrosis

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3
Q

Does the patient have to be doing anything to bring on this pain?

A

No; they may be asleep

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4
Q

Will rest or nitroglycerine relieve this pain?

A

NO

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5
Q

S/S OF MI:

____ pain

____skin

____ BP

____ Cardiac output

_____ WBCs

_____ temperature

______ changes can they vomit sometimes? why?

A
  • increased pain
  • cold clammy skin
  • decreased bp
  • decreased CO
  • increased wbc (inflammation)
  • increased temp (inflammation)
  • EKG changes
  • yes vomit; anytime you have someone in tremendous pain, they may start to vomit because when you’re in pain, the vagus nerve can be stimulated which drops your heart rate. when the rate drops= decreased CO= less blood in GI tract and they get nausea and they begin to vomit.
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6
Q

The MOST common sign is ___ in the chest with radiation to the___ side.

A

pressure; left

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7
Q

What are the cardiac enzymes that are drawn?

Which ISOENZYME is the MOST sensitive indicator of an MI?

A

CPK (creatinine phospho kinase) test; enzymes found mostly in the heart, brain, and skeletal muscle.

  • CKMB (creatinine kinase myoglobin blood) test
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8
Q

Why is troponin a better indicator of cardiac damage than CPK?

A
  • Nothing else can make the troponin go up except the heart itself; whereas, a simple I.M shot can make the CPK go up.

*Troponin= cardiac muscle damage * < 0.01

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9
Q

TROPONIN:

Can be detected___ to ___ hours after onset of chest pain can last __ to ___ days after heart damage.

A

3 to 6

6 to 8

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10
Q

Which enzymes or markers that are most helpful when the patient delays seeking care?

A

LDH (Lactase dehydronase…. levels rise in response to cell damage) and troponin

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11
Q

CHEST PAIN TX:

When a client presents to the E.R with any form of chest pain, you give them an ____ right there.

A

-aspirin

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12
Q

If you’re in the middle of an MI, What is the drug of choice for pain? why?

A

IV morphine. pain relief and vasodilation which decreases the workload of the heart immediately.

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13
Q

Who greets every patient that have chest pain?

A

M.O.N.A (Morphine, Oxigen, Nitroglycerine, Aspirin)

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14
Q

Which untreated arrhythmias will put the patient at risk for sudden death?

What drugs do we give to treat these?

A

*V.FIB

Pulseless V.Tach

Asystole —-Lidocaine (treats irregular arrythmias as well as numb skin), amiodarone (anti-arrythmic)

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15
Q

When someone goes into V-FIB, what do you better hurry up and do?

A

Defib them without delay or they can die. The longer you wait to shock someone, the greater the chance it’s not gonna work.

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16
Q

What is a symptom of Lidocaine toxicity?

A

Any neuro change, so you better be doing neuro checks

17
Q

what is an important side effect of amiodarone?

A

HYPOTENSION

18
Q

what are you worried about with other arrhythmias?

A

decreased cardiac output

19
Q

Someones has come into the ER and they’re having an MI and we put them in the “head up” position. why?

A

it will decrease cardiac workload and increase cardiac output.

* remember, when u lay flat, it increases cardiac workload*

20
Q

When you are lying flat, is your heart working harder or less?

A

harder; it ‘s a stress on the heart. when you’re flat, your heart is pumping uphill.