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BMS165 > Blood Clotting WK7 L3 > Flashcards

Blood Clotting WK7 L3 Flashcards

1
Q

WHAT IS HAEMOSTASIS?

A

BLOOD CLOTTING

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2
Q

WHAT ARE PLATELETS?

A

CELL FRAGMENTS OF LARGE RED BONE MARROW CELLS CALLED MEGAKARYOCYTES

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3
Q

WHAT IS PLATELET PRODUCTION CALLED?

A

THROMBOPOEISIS

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4
Q

WHAT IS A NORMAL PLATELET COUNT?

A

130,000 TO 4000,000 PER MICRO LITRE

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5
Q

WHAT ARE PLATELETS ALSO CALLED?

A

THROMBOCYTES

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6
Q

WHAT IS LOW PLATELET COUNT CALLED?

A

THROMBOCYTOPENIA

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7
Q

HOW LONG DO PLATELETS LIVE FOR?

A

3 DAYS

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8
Q

WHAT ARE PLATELET FUNCTIONS?

A

SECRET SERETONNG FOR VASODIALATION - SEAL BREAKS IN INJURED BLOOD VESSELS - SECRET CHEMICALS THAT STIMULATE REPAIR AND GROWTH - SECRET CHEMICALS TO BREAT CLOTS DOWN - SECRETE CLOTTING FACTORS - SECRETE ADENS\OSINE TO DRAW OTHER CELLS TO DAMAGED AREA

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9
Q

HOW MANY MECHANISIMS FOR HAEMOSTASIS IS THERE?

A

3

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10
Q

WHAT IS THE FIRST MECHANISIM OF HAEMOSTASIS?

A

VASCUALR SPASAM - SMOOTH MUSCLES IN VESSELS CONSTRICT AND NARROWS DIAMETER AND STOPS BLOOD LOSS

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11
Q

WHAT OS THE SECOND MECHANISIM OF HAEMOSTASIS?

A

PLATELET PLUG FORMATION - ADHERING TO DAMAGED COLLAGEN FIBERS AND SPIKE TO ATTACH TO EACH OTHER AND VESSELL WALLS PLUGGING UP THE VESSEL

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12
Q

WHAT IS THE THIRD MECHANISIM OF HAEMOSTASIS?

A

COAGULATION - TURNING FIBRINOGEN INTO FIBRIN THREADS THAT CATCH RED BOOD CEELS TO MAKE A BLOOD CLOT

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13
Q

HOW MANY PATHWAYS IS THERE TO MAKE FIBRIN?

A

2

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14
Q

HOW DOES FIBRINOGEN TURN INTO FIBRIN?

A

CASCADE OF EVENTS THAT LEAD TP ACTIVE CLOTTING FACTOR 10 WHICH THEN ACTIVATES A PATHWAY WHERE THROMBIN CONVERTS FIBRINOGEN INTO FIBRIN

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15
Q

WHAT IS THE FIRST PATHWAY?

A

EXTRINSIC PATHWAY INITIATED BY THROMBOPLASTIN - CAUSED BY THROMBOPLASTIN BEING RELEASED BY DAMAGED TISSUES AND BLLO VESSELS

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16
Q

WHAT IS THE SECOND PATHWAY?

A

INTRINSIC PATHWAY INITIATED BY CHEMICALS BEING RELEASED BY PLATELETS

17
Q

WHAT CHEMICALS PREVENT PLATELET CLOTTING?

A

PROSTACYLIN AND NITRIC OXIDE

18
Q

WHERE DO PROSTACYLIN AND NITRIC OXIDE COME FROM?

A

ENDOTHELIAL CELLS

19
Q

WHEN MAY BLOOD CLOT?

A

DURING STASIS WHERE THROMBIN IS NOT DILUTED

20
Q

WHAT ARE CONSEQUENCES OF UNWANTED CLOTTING?

A

THROMBOSIS - EMBOLISM - MYOCARDIAL INFARCTION

21
Q

WHY IS VITAMIN K SO IMPORTANT FOR BLOOD CLOTTING?

A

VITAMIN K IS REQUIRED TO SYNTHESIS CLOTTING FACTORS 2,7,9 AND 10

BMS165 (39 decks)

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