Approach to common dermatological presentations Flashcards

1
Q

How does pruritus present in different species?

A

Dogs: Licking feet, scratching, chewing

Cats: Overgrooming (self-induced alopecia)

Horses: Stamping, rubbing

Small furries: Barbering (must differentiate from normal grooming)

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2
Q

What are the major causes of pruritus?

A

Hypersensitivities (atopic dermatitis, food allergy)

Parasites (fleas, mites, lice)

Microbial infections (bacterial pyoderma, Malassezia)

Other causes (neoplasia, immune-mediated diseases)

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3
Q

What tests help rule out parasitic and microbial causes of pruritus?

A

Parasites: Coat brushing, skin scraping, trichogram, acetate tape strips

Microbial Infections: Cytology (bacteria, Malassezia), Wood’s lamp, fungal culture

Consider treatment trials if test sensitivity is low (e.g. fleas, scabies)

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4
Q

What should be done if parasites and infections are ruled out as causes of pruritus?

A
  1. Elimination diet trial:
    - 6-8 weeks with novel or hydrolysed protein (dogs/cats)
    - Limited forage diet (horses)
    - Challenge with old foods to validate results
    - If diet works: diagnose with food-induced allergy
  2. If diet trial fails: Diagnose environmental atopic dermatitis by exclusion
  3. Treatment options:
    - Anti-pruritics &/or anti-inflammatory drugs
    - Prophylactic treatments to control secondary infections
    - Allergen-specific immunotherapy (ASIT) if IgE tests confirm environmental triggers
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5
Q

What is the step-by-step systematic approach to pruritus?

A
  1. Signalment, history & clinical signs → Make ranked d/d list
  2. Investigate & rule out parasites & infections first
  3. If pruritus remains → Conduct elimination diet trial
  4. If pruritus still persists → Diagnose environmental atopic dermatitis

Biopsies used for rare conditions or when presentation is unusual.

Client communication is essential – Process takes time!

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6
Q

What are some important considerations when assessing the signalment in pruritic animals?

A

Species:
- Diseases affect different species with different frequency
- e.g. Farm animals: Environmental atopy rare; ectoparasites more important
- e.g. Prion disease (scrapie) important in sheep

Age:
- Consider not only age, but age of ONSET of first signs of pruritus
- e.g. Parasites → Any age (Demodex in young/old)
- e.g. Atopy → Environmental (young adults); Food-induced (<1 year)
- e.g. Older animals → Rule out neoplasia & secondary infections

Sex:
- e.g. (older) ME dog: Sertoli cell tumour → secondary pyoderma

Breed predispositions:
- Care that these don’t have unconsidered effect on diagnosis

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7
Q

What questions would you ask/what would you want to find out for the history of the pruritic animal?

A

Animal background, details of husbandry (eg housing, bedding, diet)

Routine medical care & when administered (esp re parasites)

History of contagion/zoonosis

Details of skin condition – age of onset/duration, initial clinical signs/progression, seasonality

Evidence of systemic disease

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8
Q

What are the key considerations in eliminating parasites and microbial infections in dermatological cases?

A

Use wide range of tests

Choose effective anti-parasiticides

Consider antibiotic stewardship

Combine parasite control & microbial testing for:
- Better welfare
- Easier interpretation of results
- Clear communication with owners

In large animals, parasite control is main focus due to economic impact

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9
Q

What are the next steps if a pruritic animal improves with parasite control?

A

Continue parasite control

Reassess need for ectoparasitic therapy based on:
- Parasite type
- Environment
- Cost

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10
Q

What are the next steps if a pruritic animal improves with antimicrobial treatment?

A

If pruritus resolves after stopping treatment → monitor closely

If pruritus returns after stopping treatment → investigate further (consider other signs, owner wishes & antibiotic stewardship)

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11
Q

How should parasite control and antimicrobial therapy be assessed in a pruritic animal?

A

If both treatments were given & animal improved → stop antimicrobials first & observe response before deciding on further interventions

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12
Q

Are pustules and papules primary or secondary lesions?

A

Always primary

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13
Q

Define papule

A

Small solid elevation of skin <1cm diameter

Often erythematous

May → crusts of serum, pus or blood

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14
Q

Define pustule

A

Small (<1cm) skin elevation, filled with pus

Often start as papule

Depth in epidermis varies with disease

Fragile, short-lived so often see as crusts/ epidermal collarettes

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15
Q

What are some common causes of pustules & papules?

A

Infections
- Superficial bacterial pyoderma/folliculitis! (dog, horse)
- Dermatophytosis (cat)
- Malassezia dermatitis (dog)

Ectoparasites
- Fleas (dog, cat)
- Surface mites
- Burrowing mites (e.g. Sarcoptes)
- Demodicosis (dog)

Hypersensitivities
- Fleas (dog, cat)
- Environmental/food (dog, cat, horse)
- Insect bites (horse, farm animal)

Autoimmune disease (rare)
- Pemphigus foliaceus (dog, cat)

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16
Q

What key factors should be assessed during a dermatological clinical examination of lesions?

A

Follicular lesions? Suggests bacterial folliculitis, demodicosis, dermatophytosis

Lesion distribution:
- Caudal/dorsal trunk (dog) → Fleas
- Ventral abdomen, pinnal margins, elbows, hocks (dog) → Sarcoptic mange
- Lesions on face/pinnae/head? Consider autoimmune disease
- Lesions at different stages? Suggests superficial pyoderma
- Lesions appearing in waves? Consider pemphigus

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17
Q

Describe the diagnostic approach for pustules/papules

A
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18
Q

Are scale and crust primary or secondary lesions?

A

Usually secondary, rarely primary

(Where scale occurs in diseases, its secondary to underlying primary cause: secondary keratinisation disorders)

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19
Q

Define scale

A

Rafts of immature keratinocytes which accumulate at skin surface

Due to hyperkeratosis (increased depth of cornified layer)

Caused by increased or disrupted epidermal turnover

Loose or tightly adherent

Form scurf when desquamate

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20
Q

Define crust

A

exudates (serum, pus or blood) that dried on skin surface

Often also involves surface squames, hair, topical medications

Can be associated with:
- scaling diseases
- pustular/papular diseases
- ulcerative/erosive diseases

Very non-specific finding

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21
Q

What are some common infectious causes of scale and crust?

A

Bacterial
- Pyoderma/folliculitis! (dog, horse)
- Dermatophilosis! (farm, horse)

Fungal
- Dermatophytosis! (cattle, horse, cat)
- Malassezia dermatitis (dog)

Viral
- Viral papillomas (cattle, horse)
- Occult sarcoids (horse)

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22
Q

What are some common parasitic causes of scale and crust?

A

Ectoparasites
- Fleas (dog, cat)
- Lice (farm, horse)
- Surface mites
* Chorioptes (horse, cattle)
* Cheyletiella (rabbit)
* Psoroptes (sheep, rabbit)
- Burrowing mites
* Sarcoptes (dog)
* Trixacarus (guinea pig)
* Cnemidocoptes (bird)
- Demodicosis (dog, hamster)

Endoparasites (horse)

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23
Q

What are some common hypersensitivity causes of scale and crust?

A

Flea (dog, cat)

Environmental/food (dog, cat, horse)

Insect bites (horse, farm)

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24
Q

What are some common nutritional/metabolic causes of scale and crust?

A

Photosensitisation (cattle, horse)

Hypothyroidism (dog)

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25
Q

What are primary keratinisation disorders?

A

Defects in normal keratinisation process
- Abnormal formation of keratinocytes
- Abnormal sebaceous gland function

Often breed-related & tend to occur in younger animals

Diagnosis of exclusion

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26
Q

Describe the diagnostic approach for scale/crust

A
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27
Q

What lesion is this?

A

Crust

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28
Q

What lesion is this?

A

Scale

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29
Q

What lesion is this?

A

Papule

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30
Q

What lesion is this?

A

Pustule

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31
Q

Give examples of swellings of non-dermatologic origins

A

Hernias
Oedema
- E.g. due to right sided heart failure, hypoalbuminaemia
Bursitis
Emphysema
Mammary tumours

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32
Q

How does oedema (swelling of non-dermatologic origin) present?

A

Ill-defined, soft, painless swelling from, e.g. R sided heart failure, hypoalbuminaemia

Pits on pressure

Clear fluid on FNA

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33
Q

What is emphysema and what causes it?

A

gas in subcutaneous tissue

crepitant without pain or swelling

caused by:
- Severe respiratory disease or lung puncture
- Introduction of air through cutaneous wound
- Rumenotomy or rumen cannulisation
- Clostridial infections

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34
Q

How can skin masses be classified?

A
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35
Q

Give examples of infectious inflammatory skin masses

A

Abscess/ cellulitis
- e.g. post trauma/FB/ bite (farm animal, cat, also rabbit secondary to dental disease)

Furunculosis
- e.g. staphylococcal deep pyoderma
- e.g. Demodex

Bacterial granulomas
- e.g. Mycobacteria
- e.g. Actinobacillus, Nocardia, Actinomyces

Deep/subcutaneous or systemic fungal granuloma

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36
Q

Give examples of non-infectious inflammatory skin masses

A

Urticaria, angioedema (horse>dog>cat)

Seroma

Haematoma

Others
- e.g. eosinophilic granulomas, tick/insect bite granulomas, sterile panniculitis

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37
Q

What is urticaria/angioedema?

A

Degranulation mast cells –> oedema (painless, pit on pressure)

Can be allergic (type I or III) or non-immunologic cause

Urticaria –> wheals (+/- pruritus)

Angioedema –> large oedematous swelling, usually involving head – can be fatal
- same process but bigger area

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38
Q

What is a seroma?

A

Accumulation of serum under skin

Painless, non-pitting

Frequently occurs post-surgery

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39
Q

What is a haematoma?

A

Loss of blood from damaged/ruptured blood vessels under skin; painless, non-pitting

Usually due to trauma, occasionally due to clotting problems

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40
Q

Give examples of common neoplastic causes of cutaneous skin masses in different species

A

Most common in older animals –> uncommon in farm animals

Horse – esp. melanomas (grey horses), sarcoids

Dogs – skin neoplasms common - e.g. lipomas, sebaceous adenomas, mast cell tumours

Cats – neoplastic skin masses less common

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41
Q

What is a cyst?

A

Epithelial-lined cavity, containing fluid or solid material produced by cells of cyst lining

Smooth, well-circumscribed

Fluctuant/solid – dependent on nature of contents

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42
Q

What key factors influence the differential diagnosis for skin masses?

A

Species
- e.g. abscess in cat, cattle, rabbit; skin neoplasm in old dog

Breed/colour
- e.g. Melanomas in grey horse, mast cell tumours in boxers

Age
- e.g. neonate: umbilical abscess
- e.g. old animal: neoplasia

General & dermatologic history

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43
Q

What are examples of history findings that suggest specific conditions in cutaneous skin masses?

A

Recent trauma, fight, surgery → Abscess, haematoma, seroma

Recent injection → Abscess, panniculitis

Systemic signs (e.g. weight loss, respiratory signs) → Neoplasia, systemic fungal infections

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44
Q

What are key factors to look for in a general clinical examination of a patient with a cutaneous skin mass?

A

Pyrexia → Often seen with systemic or severe cutaneous microbial infections & abscesses

Peripheral Lymphadenopathy → Lymph node enlargement due to metastatic neoplasia or reaction to infection/inflammation

Other Systemic Abnormalities → May influence diagnostic/treatment choices or directly relate to mass
- Animals with lung metastases may have no clinical signs

Non-Dermatological Swelling? → Consider bursa, joint swelling, or hernia

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45
Q

What are we assessing in a dermatological examination of a patient with a cutaneous skin mass?

A

Solitary/multiple lesions?
Area of body? Size?
Well-defined? Ill-defined?
Freely moveable?
Draining tracts/sinuses?
Pits on pressure?
Painful/painless?
Inflammatory?

46
Q

What cytology investigations can be done to a cutaneous skin mass?

A

Fine needle aspirate
1. ‘Needle only’ with no suction
2. ‘Continuous suction’
3. ‘Intermittent suction’

Impression smear of surface

Smears/touch impressions of sinus contents

47
Q

How can cytology help differentiate types of inflammatory cutaneous masses?

A

Sterile vs. Septic – Presence of organisms suggests septic inflammation
- may be difficult to detect, esp. in pyogranulomatous inflammation
- Some organisms (e.g. mycobacteria) require special stains

Chronic vs. Acute – Chronic inflammation may show macrophages & fibrosis, while acute inflammation is often neutrophilic

48
Q

What are the three main categories of neoplastic cells in cutaneous cytology?

A

Round cell
Epithelial cell
Spindle cell

49
Q

How does cytology help identify cystic skin masses?

A

Cyst contents are produced by epithelial lining, leading to different aspirates:
- Sebaceous material, keratinised debris, cholesterol crystals

Granulomatous inflammation may be present if cyst has ruptured

Often amorphous appearance

50
Q

What are the limitations of cytology?

A

Not all cell-types shed easily, so not always representative

May take unrepresentative sample

Gives no info re tissue architecture thus can’t grade neoplasm
- Requires subsequent histopathology if grading required

51
Q

When is tissue biopsy recommended?

A

When FNA is inconclusive, for histopathology confirmation, and for tissue culture if needed

52
Q

What skin mass is this?

53
Q

What skin mass is this?

A

Angioedema of muzzle

54
Q

What skin mass is this?

55
Q

What skin mass is this?

A

Aural heamatoma

56
Q

What skin mass is this?

A

Eosinophilic granuloma (horse)

57
Q

What skin mass is this?

A

Melanoma of perineum

58
Q

What is alopecia, and what are the two main types?

A

Partial or complete hair loss, can be:
- Primary – Failure of normal hair growth
- Secondary – Hair grows normally but is later damaged or lost

59
Q

What is the difference between true and apparent alopecia?

A

True alopecia – Direct damage to hair follicle unit –> loss of whole hair follicle

Apparent alopecia – Hair shafts damaged but not lost from follicle –> hair cropped short

60
Q

What are the key mechanisms leading to primary alopecia?

A

Lack of stimulation of anagen phase → Hair fails to enter growth phase

Abnormal growth factors → Leads to miniaturised/dysplastic hairs that break off or shed

Elongation of telogen phase → Hair remains in resting phase indefinitely, preventing new growth

61
Q

What are the major categories of alopecia causes?

A

Folliculitis!/furunculosis/bulbitis – Inflammatory damage to hair follicle unit

Hair cycle abnormality – Hair stops growing (e.g. endocrinopathy)

Hair morphology defects – Hair is malformed and breaks off

Congenital aplasia (rare) – Hair never grows

62
Q

What factors should be considered when creating a differential diagnosis list for alopecia?

A

Species:
- Ringworm → Cattle, horses, cats, hedgehogs.
- Demodicosis, pyoderma → dogs
- Occult sarcoids → Horses

Age:
- Young → Infections (e.g. demodicosis, dermatophytosis, superficial pyoderma)
- Older → Endocrinopathies, neoplasia, chronic demodicosis

Breed:
- Terriers & Boxers → hyperadrenocorticism
- Staffies → demodicosis
- Dachshunds → Pattern baldness
- Jack Russell Terriers → Trichophyton infections from rodents/hedgehogs

Sex:
- Entire female guinea pigs → Ovarian neoplasia
- Entire male dogs → Sertoli cell tumours

63
Q

How can history help identify the cause of alopecia?

A

Contagious/Zoonotic Causes → Consider dermatophytosis (ringworm) or ectoparasites

Systemic Signs:
- PUPD, polyphagia → HAC? (dog)
- Weight gain, lethargy → Hypothyroidism?

Pruritus?
- If present, investigate as self-trauma (pruritic alopecia), not true alopecia

64
Q

In what cases can alopecia be normal?

A

Preauricular/pinnal alopecia of cats

Sphinx cats

Irish water spaniels

Flank scent glands on hamsters

65
Q

What differentials do different distributions of alopecia suggest?

A

Localized or Multifocal/diffuse patchy alopecia → Dermatophytosis, pyoderma, demodicosis

Symmetrical alopecia → Dermatophytosis, pyoderma, demodicosis, Endocrinopathies, pattern alopecie, follicular dysplasias (+- colour linked), trace element deficiency (cattle)

66
Q

How can we investigate whether an animal has true or apparent alopecia?

A

Trichogram
- Ease of epilation at periphery
- Easily epilated → true
- Requires some effort → apparent
- Broken distal tips → apparent
* NB often seen with trauma/pruritus so if pruritic investigate as per pruritus protocol

67
Q

What lesion is seen here (alongside alopecia) and what condition is it suggestive of?

A

Calcinosis cutis –> Hyperadrenocorticism (dog)

68
Q

What lesion is seen here (alongside alopecia) and what condition is it suggestive of?

A

Comedones, skin thinning –> Hyperadrenocorticism (dog)

69
Q

What lesion is seen here (alongside alopecia) and what condition is it suggestive of?

A

Epidermal collarettes, papules –> pyoderma (dog)

70
Q

What lesion is seen here (alongside alopecia) and what condition is it suggestive of?

A

Draining sinuses, furunculosis –> demodicosis (dog)

71
Q

What tests help differentiate causes of alopecia?

A

Skin scrapings
- esp. Demodex mites

Dermatophyte testing
- Direct microscopy, culture
- Wood’s lamp for ringworm

Trichogram (hair analysis):
- Telogen dominance → endocrinopathies
- Melanin clumps → colour dilution alopecia (dog)
- Arthrospores/hyphae → dermatophyte infection
- Follicular casts → sebaceous adenitis (dog)
- Broken hair tips → trauma/pruritus

72
Q

If no definitive diagnosis is reached in a alopecia case following skin scrapings, dermatophyte testing & trichogram, what further tests can be done?

A

Cytology – Helps diagnose secondary bacterial pyoderma

Endocrine testing

Skin biopsy to see:
- Folliculitis – e.g. bacterial, demodicosis, dermatophytosis
- Atrophic changes - e.g. endocrinopathy
- Follicular dysplasias
- Autoimmune cause - e.g. sebaceous adenitis or alopecia areata

73
Q

What lesion is this?

A

Palatine lesion

74
Q

What lesion is this?

75
Q

What lesions are these?

A

Plaques & ulceration

76
Q

What lesion is this?

77
Q

What lesion is this?

A

Excoriation on face

78
Q

Fred is 11-month-old cat with 3-month history of severe pruritic skin disease. He is systemically well & lives in a multi-cat household.

Based on the history, what test would you want to do?

A

Wet paper tests (fleas) and thorough testing for other parasites

79
Q

Label the cells and what condition does it suggest?

80
Q

11-year-old Cavalier King Charles Spaniel with severe pruritus & redness of skin. He has mild cardiac disease, but this is adequately treated & he is generally well. He is receiving monthly afoxolaner & has good appetite & normal thirst.
Owner has googled allergic skin disease & considers this most likely diagnosis.

Why is cAD unlikely:
Based on history?
Based on lesions?

A

Based on the history?
- Age of onset very late for allergic disease (75% of cases start between 6m & 3y of age)

Based on the lesions?
- Distribution – esp. nose leather & top of head would be very unusual in cAD
- Nature of lesions – erosions, depigmentation, scale, alopecia & small hyperpigmented macules suggestive of disease attacking basement membrane

81
Q

What test is best for diseases of basement membrane?

A

biopsy or cytology from exudative lesions

82
Q

What are examples of diseases of the basement membrane?

A

Epitheliotropic lymphoma, cutaneous lupus, vitiligo

83
Q

Describe these lesion. Are the lesions suggestive of endocrine alopecia?

A

Well demarcated, irregular alopecia affecting lateral flank with hyperpigmentation of exposed skin

Distribution appears geographical rather than generalised, indicating localised process rather than widespread telogenisation

Lesions don’t suggest endocrine alopecia, where hair loss occurs due to wear & tear, often presenting with diffuse & poorly demarcated edge

84
Q

2½ year old MN mastiff cross has 4-month history of alopecia affecting sides. He is non-pruritic & owner reports that he has had no other lesions & is fit & well. His weight is normal & static. Its March at time of consultation.

What history points would you consider helpful in considering endocrine problems?

A

Age
- very young dog for neoplastic endocrinopathy (e.g. HAC or hyperoestrogenism due to Sertoli cell tumour)
- Immune-mediated endocrine problem (e.g. hypothyroidism) more likely, but dog is still quite young for this disease

Normal behaviour is noted & no weight gain

Problem started in winter, if it resolves in summer it would be strong hint that endocrine disease isn’t present

85
Q

What basic tests can help investigate hormonal alopecia?

A

Haematology & Biochemistry:
- Stress leukogram, increased ALP → Hyperadrenocorticism
- Raised cholesterol → Hypothyroidism

Urine Tests:
- high USG → HAC unlikely
- Urine Creatinine:Cortisol: Useful as rule-out test for HAC

Thyroid Function Tests:
- T4 & TSH → Used to rule out hypothyroidism

86
Q

Give examples of primary causes of feather loss in parrots

A

Hypovitaminosis A
Allergies
Hypothyroidism
Feather cyst
Giardia

87
Q

Give examples of secondary causes of feather loss in parrots

A

Fungal/parasitic
Mites (Knemidocoptes)
Ringworm
Systemic illness

88
Q

Give examples of behavioural causes of feather loss in parrots

A

Stress
Self mutilation
Boredom
Mating behaviours
Overbonding with owner

89
Q

Give examples of primary causes of feather loss in chickens

A

Dermayssus Gallinae (red mite)
Nutritional deficiencies

90
Q

Give examples of behavioural causes of feather loss in chickens

A

Feather pecking (hierarchy)
Poor enrichment
Stress
Overmating
Brooding

91
Q

Give examples of causes of hair loss in guinea pigs

A

Cystic ovarian disease
Ectoparasites
Pyoderma
Post partum alopecia
Overgrooming
Endocrine disease
Vit C deficiency
Dermatophytes!

92
Q

What are the 3 most common equine skin neoplasia?

A

Melanoma
- Black, spherical or plaque like, common around perineum & parotid region of grey horses

Sarcoid
- Multiple types, usually hairless, sometimes ulcerative, common in groin, axilla & around eyes

Squamous cell carcinoma
- Raised, irregular, pink, locally invasive, common around genitals & eyes

93
Q

What are equine sarcoids?

A

Most common skin tumour in horses, caused by Bovine Papillomavirus

94
Q

What are key characteristics of equine sarcoids?

A

Locally invasive, fibroblastic & wart-like

Can be single or multiple lesions

Six different types exist

95
Q

How are equine sarcoids diagnosed?

A

Based on visual appearance, but confirmed by biopsy

Histologically, they show dense dermal fibroblasts in interlacing bundles

96
Q

How are equine sarcoids (Bovine Papillomavirus) transmitted?

A

Flies act as vectors, spreading virus between horses

Common in areas where flies frequently bite (e.g. groin, axilla & around eyes)

97
Q

What type of equine sarcoid is this?

98
Q

What type of equine sarcoid is this?

99
Q

What type of equine sarcoid is this?

100
Q

What type of equine sarcoid is this?

A

Fibroblastic

101
Q

What type of equine sarcoid is this?

102
Q

What type of equine sarcoid is this?

103
Q

What are the treatment options for equine sarcoids?

A

Laser surgical removal

Cryotherapy

Caustic cream application

Elastrator band application (causes hypoxic necrosis in nodular sarcoids)

Radiotherapy (particularly for periocular sarcoids)

104
Q

How can melanomas around the perineum be treated?

A

Removed with a surgical laser

105
Q

What are the key features of Squamous Cell Carcinoma (SCC)?

A

Locally invasive, pink, irregular, sometimes ulcerated lesions

Can grow rapidly

Often affects genitals, eyes, penis & third eyelid

106
Q

What is the standard treatment for SCC?

A

Complete surgical excision is recommended as SCC can progress & spread rapidly

Histological confirmation ensures complete removal

Early intervention is curative in most cases

107
Q

What are eosinophilic granulomas?

A

Small, firm, non-itchy, non-painful raised nodules with normal hair covering

Commonly found on withers & back

Not neoplastic; their exact cause is unknown

108
Q

How are eosinophilic granulomas diagnosed?

A

Based on clinical appearance

Confirmed via fine needle aspirate or biopsy

109
Q

What is the treatment for eosinophilic granulomas?

A

Often not required unless interfering with tack

Can be treated with surgical excision or corticosteroid injections if necessary

110
Q

What are juvenile papillomas?

A

Multiple, small, irregular, verrucose (wart-like) grey proliferative lesions

Commonly found on muzzle, face & sheath of young horses

Caused by equine papillomavirus

111
Q

What is the typical progression and treatment of juvenile papillomas?

A

They are usually self-limiting, so treatment is not required

Cryotherapy may be considered for severe cases

112
Q

What are the key characteristics of equine melanomas?

A

Typically benign, black, nodular & slow-growing

Most common in grey horses, esp. in perineum, sheath & parotid region