Adult hip conditions and surgery Flashcards

1
Q

can you make more hyaline cartilage

A

no

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2
Q

what is femoroacetabular impingement syndrome (FAI)

A

when altered morphology of the femoral neck and/or acetabular causes impingement of the femoral neck on the edge of the acetabulum during movement

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3
Q

what movement usually cause FAI

A

flexion, adduction and internal rotation (pulling on your shoe)

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4
Q

what is a CAM type impingement in FAI

A

femoral deformity- asymmetric femoral head with decreased head:neck ratio

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5
Q

who gets a CAM type FAI

A

usually young, athletic males

can be related to previous SUFE

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6
Q

what is a pincer type inpingement in FAI

A

acetabular deformity- acetabular overhang (extra bit of bone or acetabular tilted forward)

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7
Q

what does FAI cause in the joint

A

damage to the labrum and tears
damage to the cartilage
osteoarthritis in later life

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8
Q

what is the usual presentation of FAI

A

activity related pain in the groin- particularly flexion and rotation
difficulty sitting
C sign positive
FADIR provocation test positive

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9
Q

what is C sign positive

A

when patients with FAI are asked to describe their pain they will make a c shape with their hand and place it around hip joint

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10
Q

what is the FADIR provocation test

A

flexion, adduction, internal rotation- if maneuver produces pain suggestive of hip impingement

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11
Q

what can you diagnose FAI

A

radiographs, CT, MRI (better for visualising damage to labrum and bony oedema)

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12
Q

what is the management for FAI

A

observation in asymptomatic patients

arthroscopic or open surgery to remove CAM/ debride labral tears

periacetabular osteotomy/ debride labral tears in pincer impingement

arthroplasty older patients with secondary OA

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13
Q

what is avascular necrosis

A

failure of the blood supply to the femoral head resulting in subsequent necrosis

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14
Q

what causes AVN

A

idiopathic:

  • coagulation of the intraosseous microcirculation
  • venous thrombosis causes retrograde arterial occulsion
  • intraosseous hypertension
  • decreased blood flow to the femoral head
  • necrosis of the femoral head
  • chondral fracture and collapse

AVN associated with trauma
-injury of femoral head blood supply (medial femoral circumflex)

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15
Q

who gets avascular necrosis and in which hip?

A

males more than females
typical age 35-50
80% of cases bilateral

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16
Q

what are the risk factors for AVN

A

irradiation,
trauma,
haematologic diseases (leukemia, lymphoma), sickle cell,
hypercoagulable staes,
dysbaric disorders (decompression sickness ‘the bends’ aka the bends)
alcoholism,
steroid use,

most cases idiopathic

17
Q

what is the usual presentation of AVN

A

insidious onset of groin pain,
exacerbated by stairs or impact,
examination is usually normal unless disease has advanced to collapse/ OA

18
Q

how can AVN be diagnosed

A

radiographs (normal in early disease)

MRI is most sensitive and specific

19
Q

what is the management for AVN dependent on

A

based on the stage of disease:
reversible
irreversible

20
Q

what is the management of avascular necrosis

A
if reversible:
bisphosphonates
core decompression +/- bone grafting 
curettage and bone grafting 
vascularised fibular bone graft 

rotational osteotomy

total hip replacement
(if irreverisble)

21
Q

what is idiopathic transient osteonecrosis of the hip (ITOH)

A

local hyperaemia and impaired venous return with marrow oedema and increase intraedullary pressure

22
Q

how does ITOH usually present

A

progressive groin pain over several weeks
difficulty weight bearing
usually unilateral

23
Q

who gets ITOH

A

males more than females

usually: middle ages females and pregnant women in third semester

24
Q

how do you diagnose ITOH

A

elevated ESR
radiographs: osteopenia of the head an neck, thinning of the cortices, preserved joint space
MRI (gold standard)
bone scan

25
Q

how is ITOH managed

A

self limiting condition that resolves in 6-9 months
analgesia so they can keep moving
protected weight bearing to avoid stress fracture

26
Q

what is trochanteric bursitis

A

repetitive trauma caused by iliotibial band tracking over trochanteric bursitis causing inflammation of the bursa

27
Q

who gets trochanteric bursitis

A

female patients, young runners and older patients (gluteal cuff syndrome- degenerative of)

28
Q

how does trochanteric bursitis

A

pain on LATERAL aspect of the hip

pain on palpation of the greater trochanter

29
Q

how do you diagnose trochanteric bursitis

A

clinical diagnosis
radiographs usually unremarkable
visible on MRI but not usually needed

30
Q

how is trochanteric bursitis managed

A

analgesia, NSAIDs, physio, steroid injection

no benefit from surgery

31
Q

what is osteoarthritis

A

degenerative disease of synovial joints that causes progressive loss of articular cartilage

inflammatory changes in the capsule lead to thickening and tightness

32
Q

who gets OA

A

females more than males, typically in older age, genetic element, pre existing hip disease

33
Q

what is the usual presentation of hip OA

A

groin pain, worse on activity, pain at night, start up pain (after resting for a while- lack of synovial fluid to lubricate the joint)
stiff on testing ROM

34
Q

what determines if a patient gets surgery for OA

A
level of symptoms 
impact of QOL
medical comorbidities 
social history 
do they want surgery
35
Q

what are the radiographic signs of OA

A

loss of joint space
osteophytes
subchondral sclerosis
subchondral cyst formation

36
Q

what is the management for OA

A

analgesia, weight loss walking aids, physio if weakness, ?steroids

THR

37
Q

what is the main indication for THR in OA

A

pain