Unit 3 Part 2 Flashcards
what is to be said about tx for liver failure
unlikely unless dt hepatotoxicity
what do we tx in liver failure
mnfts and complications
tx options for liver failure
purgative
non absorbable abx
liver transplant
what is a purgative in liver failure
strong laxative that cleanses bowel
fx of purgative in liver failure
removes all ammonia (proteins) from bowel so the liver doesnt have to deal with it
what is a non absorbable abx (LF)
abx not absorbed by CV. Absorbed by GI to kill bacteria that make ammonia and nitrogenous compounds
cholelithasis
gall stones
choleycstitis
inflm of liver
ET of cholelithasis (5)
bile stasis change in bile comp inflm/debris e.coli/ strep faecelisy genetic susceptibilit
how does bile stasis cause gall stones
inc percipitation formation with stasis
what composition change of bile lt inc of gall stones
inc CHO, dec bile salts
how does inflm/debris lt gallstones
inflm/debris acts as a nuclei for stone, this debris collects percipitate forming around hub
how does E.coli/Strep faecelis lt gallstones
bact alters bile comp (idiopathic?) resulting in inc susceptibility for stones
3 types of gallstones
cholesterol stones
pigment stones
mixed stones
cho stones
large, round, yellow, smooth, glistening, occupy inc amount of bladder volume. dt bile with inc [CHO]
what percent of stones are cho stones
80%
pigment stones
pigmented stones with variety of types
what contents make up pigmented stones
bilirubin (pigment) and inc ca+ salts
what percent of pigmented stones are all bile stone
20%
mixed stones
stones of various components
when do mnfts of cholelithasis come up
it is asymptomatic until stone moves
what happens when a stone migrates in cholelithasis
colicky, spasmodic pain, intermittent + radiating
what is pain in cholelithasis dt
contraction of duct where stone is located
other mnfts of cholelithasis then pain
NV dt GI mfnts + severe pain
