Self Study 6 Flashcards

1
Q

what does allgx and H dermatoses involve

A

inflm respoonse to multi exogenous/endogenous agents

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2
Q

what is allgx and H deramtoses charcterized by

A

epidermal edieam with seperation of epidermal cells

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3
Q

what dermatitis’ does allgx and H deramtoses involve

A

irritant contact dermatitis, allgx contact dermatits, atopic and nummular eczema, drug induced skin interuptions

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4
Q

contact dematitis

A

common inflm of skin

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5
Q

2 types of contact dermatitis

A

allergic and irritant CD

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6
Q

what does allgx CD result from

A

cell mediated, type4 H rxn

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7
Q

what type of reaction occurs in ACD

A

t cell only. no abs.

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8
Q

2 kinds of ACD reaction

A

direct cytotoxic T cell destr of macrophage

deylaed t cell prod of lymphokines that destroy ag bearing cell days later

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9
Q

primary lesions ACD

A

mild erythema with edema to vesicles or lrage bullae

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10
Q

secondary lesions ACD

A

dt bacterial infc

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11
Q

where do lesions in ACD occur

A

anywhere

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12
Q

ICD cause

A

caused by chemicals that irritate skin, environmental triggers

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13
Q

risk for ICD

A

hx of atopic dermatitis

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14
Q

4 types of ICD

A

subjective, acute, chonic, chemical burns

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15
Q

subjective ICD

A

no clinical mnfts

burning or stinging when exposed to irritant

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16
Q

acute ICD

A

due to single exposures to irritant

dryness, erythema, edmea, inflm, vesicles

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17
Q

chronic ICD

A

results from contd exposure to irritant , even when irritant removed.
dryness, erythema, sclaes, fissures, vesicles

18
Q

chemical burn ICD

A

deep necrosis from contact with cement prod

19
Q

Tx ICD

A

remove source
change lifestyle
minor: wash area, apply anti itch cream, topical corticosteroids?
mod - extreme: tx with wet dressing, oral antihistamine, systemic corticosteroids

20
Q

eczema

A

inflm of skin

21
Q

2 types of eczema

A

atopic dermatits

nummular eczema

22
Q

atopic derm

A

ithcy, inflm skin disorder

23
Q

what is atopic derm characterized by

A
poorly defind erythema with edema, vesicles, weeping (acute)
skin thickening (chronic)
24
Q

how is atopic derm often described

A

IgE mediated H. Similar to Allgx patho.

25
Q

how do mnfts present in atopic derm

A

differently depending on age and race

26
Q

when do 70% of atopic derm cases start

A

peds

27
Q

what is atopic derm characterized with (mnfts)

A

vesicles formation, oozing, cursting

28
Q

where does atopic derm begin (peds)

A

cheeks, progress to scalp, arms, trunk, legs

29
Q

atopic derm mnfts adolsecent (acute)

A

dry, red patches on face, neck, upper trunk-no thickening

30
Q

atopic derm mfnts adolescent (chronic)

A

dry, leathery, lichenfied spin

31
Q

is there a gen predisopostion in atopic derm

A

yes

32
Q

tx for atopic derm

A
avoid temp changes/stress
skin care
avoid triggers
dec contact with awater
meds
wet derssing
probiotics
33
Q

why should a person with atopic derm avoid temp changes

A

minimize vasscular and sweat response

34
Q

what triggers does a person with atopic derm have to avoid

A

evironmental

food

35
Q

what meds are used to tx atopic derm

A

topical corticosteroids

topical immune modulators

36
Q

nummular eczema lesions

A

coin shaped lesions

papulovesicular patches on arms and legs

37
Q

what is normal for NE leision patterns

A

initial leisions heal, followed by secondary outbreak of mirror image leisons on opposite side of body

38
Q

pattern of exacerbation for NE

A

chornic, with weeks and years between exacerbations. happens mostly in winter

39
Q

tx for NE (lifestyle

A

avoid iodides and bromides
avoid frequent bathing and stress
inc environmental humidty
t

40
Q

tx for NE (pharm)

A

topical emollients, corticosteroids, coal tar preperations, uv light