Retinopathy Flashcards

1
Q

The most common cause of legal blindness between 20 - 65

A

Diabetic retinopathy

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2
Q

Risk factors of Diabetic retinopathy

A
  1. Obesity and hyperlepidimua
  2. Smoking
  3. Poor control
  4. anemia
  5. Age
  6. cataract surgery
  7. Complication (hypertension and nephropathy)
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3
Q

Pathogenesis

A
  1. Microvascular leakage
  2. Microvascular occlusion
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4
Q

Classification of Diabetic retinopathy (DRP)

A
  1. Background DRP
  2. pre-prolifrative DRP
  3. prolifrative DRP
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5
Q

Features of background DRP

A
  1. Micro aneurism
  2. Hard exudate
  3. Intraretinal hemorrhage
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6
Q

Features of pre proliferative DRP

A
  1. Cotton wool spots
  2. Severe retinal haemorrhage
  3. IRMA (intra retinal microvascular abnormalities A-V Shunt)
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7
Q

Features of proliferative Diabetic retinopathy

A
  1. NVD (neovascularization on the disc) & NVE (elsewhere)
  2. Tractional retinal detachment
  3. vitreous hemorrhage
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8
Q

Investigations for Diabetic retinopathy

A
  1. Fluorescent angiography
  2. OCT (optical coherence tomography)
  3. US πŸ‡ΊπŸ‡Έ
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9
Q

Treatment modalities for Diabetic retinopathy

A
  1. PRP pan-retinal photocoagulation
  2. PPV pars plana vitrectomy
  3. antiVEGF (IV)
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10
Q

Pathogenesis of Hypertensive retinopathy

A
  1. VC
  2. arterio sclerosis
  3. Increase permeability
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11
Q

Features of hypertensive retinopathy

A
  1. Attenuation and narrowing
  2. A-V crossing
  3. Coper/silver wiring
  4. Cotton wool spots
  5. Macular star
  6. Micro aneurysm
  7. Flame shaped hemorrhage
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12
Q

Types of arterial and venous occlusion (about retina)

A

artrial
- Central retinal artery occlusion
- Branch retinal artery occlusion
venous
- Central retinal vein occlusion
- Branch retinal vein occlusion
- Hemi retinal veins occlusion

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13
Q

Causes of occlusion

A
  1. Thrombus/embolus
  2. Atherosclerosis
  3. Vasculitis
    special in veins
    - compression by artery
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14
Q

Features of central retinal artery occlusion

A
  1. Sudden painless loss of vision
  2. Marcus gunn pupil (relative afferent pupillary defect)
  3. Colors:
    - White retina.
    - Red macula.
    - pale optic disc (optic atrophy).
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15
Q

Why some patients have good central vision with CRAO

A

Due to presence of Cilioretinal artery

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16
Q

Complications of CRAO and CRVO

A

CRAO
- Optic atrophy 91%
- Retinal a. Attenuation
- Neovascularization
CRVO
- macular edema
- Neovascularization
- vitreous hemorrhage
- 100d glaucoma (neovascular glaucoma)

17
Q

Sites of neovascularization in CRAO and CRVO

A

CRAO
NVD>NVE> NVI
CRVO
NVI>NVD>NVE

NV= Neovascularization
D = disc
E = elsewhere
I = iris (robiosis iridis)

18
Q

Treatment of occlusion

A
  1. VD
  2. Fibrenolytic
  3. Yag Laser embolysis
  4. Low IOP
  5. In case of vetrus hemorrhage Vitrectomy and endolaser
  6. AntiVEGF and Dexamethason for neovascularization
  7. Treat the cause
19
Q

Types of CRVO

A
  1. ischaemic
  2. Non-ishemic
20
Q

What is the different between ischemic and non ischemic CRVO?

A

non ischemic
Common (80%) & better: VF, VA, mild RAPD, 50% improve
ischemic
Less (20%) & worse: VF, VA, marked RAPD, 60% neovascularization