Final 5 Flashcards

1
Q

Breed on ____ heat

A

second or third heat

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2
Q

Who stays in heat longer? Gilts or sows?

A

sows

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3
Q

Why should you avoid continual boar exposure

A
  • they can get bored

- not as exciting as if brought boar in for 10-20 mins each day

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4
Q

Weaning to breeding interval

A
  • should be 4 or 5 days for most sows
  • biggest problem is parity 1
  • sign of problem is parity 2 litter size drop
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5
Q

Two major reasons for problem in weaning to breeding interval

A

poor energy level coming off lactation
-doesn’t cycle from not eating properly/NEB
Missed heat

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6
Q

Semen storage

A
  • swine industry uses fresh semen
  • very delicate
  • needs to be kept at 17 or 18C
  • dilute male sperm to impregnate several females with it
  • might also put antibiotics to keep disease down
  • shelf life of about 4-5 days
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7
Q

Signs of estrus

A
immobility- occurs rapidly in mid-estrus 
ears- erect ears 
lack of vocalization
vaginal viscosity 
vaginal vulval colour
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8
Q

Small litter size

A

Overall genetics
Gilts- bred on 1st heat, nutriton, quality and timing of breeding
Sows- 2nd parity drop- short lactation length
Old sows with drop in live births

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9
Q

Solving Farrowing rate problems

A
regular returns 
-timing of breeding
-semen quality 
-breeding technique
irregular returns (>23)
-infectious disease
-seasonal infertility
-stress such as fighting
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10
Q

Reproductive targets

A

farrowing rates >85%
total born/litter ~12
born alive/litter ~11
Stillborn pigs/litter

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11
Q

Parvovirus

A

Agent: Porcine pavovirus
-ubiquitous virus
Cause of embryonic and fetal death
CLinical signs:
-Increase in number of sows returning to estrus post breeding
-decrease in number of piglets born alive
-increase in number of mummified piglets
mainly gilt litters affected
Pathogenesis
-virus crosses placenta and travels from fetus to fetus along the uterine horn- replicates in cells undergoing mitosis

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12
Q

After about ____ days a fetus can fight off parvo virus

A

70

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13
Q

Control of parvovirus

A
virtually impossible to eliminate
vaccination-many killed vaccines available 
Recommended vaccination program
-gilts-twice- 5 and 2 weeks pre-breeding
-sows-revaccinated 2 weeks pre-breeding
-boars- twice annually
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14
Q

Erysipelas

A
AgentL erysipelothrix rhusiopathiae
diamond skin disease 
resistant to drying 
all age groups susceptible 
Pathogenesis and clinical signs
-systemic vasculitis 
-high fever
-purplish skin on snout, abdomen, ears
-diamond skin lesions- raised, red
-develop arthritis and vegetative endocarditis 
POTENTIAL ZOONOSIS
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15
Q

Prevention/control of erysipelas

A
vaccination
-not the best
-lasts 3-4 months
-every weaning vaccinate
quarantine incoming stock
good sanitation and hygiene
cull animals with joint lesions
treat affected animals with antibiotics/water medication
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16
Q

PRRSV

A

Virus
one of the most significant disease in the swine industry
Proprties of genus
-cause asymptomatic persistent infection
-can cause severe fatal disease
-replicates in macrophages
-exhibit considerable genetic mutation and recombination
Etiology
-considerable strain variation resulting from genetic mutation and recombination
-considerable variation in virulence among strains
-distinct clusters which are antigenically and genetically distinct
Die because of respiratory issues C

17
Q

Clinical signs of PRRSV

A
Reproduction
-pregnant sow- fever, anorexia, sow mortality if virulent, minimal impact in 1st and 2nd trimester
-3rd trimester- transplacental infection (>72 days)= repro failure and infertility 
Respriatory
-congentially infected suckling pigs
-nursery and grower pigs 
-dyspnea "thumping" most common
-cyanosis of extreneties
-minimal coughing in pure PRRS pneumonia
-immunosuppressive