Female Pelvis 2 Flashcards

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1
Q

to be ovarian cyst must be >______ mm

A

> 9mm

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2
Q

larger cysts are likely if mother had (3)

A

Toxemia, diabetes, Rh isoimmunization

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3
Q

4 types of cysts

A

follicular
corpus luteal
theca lutein
paraovarian

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4
Q

follicular cystshave what fluid

A

clear serous fluid

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5
Q

corpus luteal are what fluid

A

serous or hemorrhagic fluid

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6
Q

theca lutein caused by

A

GTD or clomid/clomiphene

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7
Q

where are paraovarian cysts

A

in broad ligament or fallopian tubes

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8
Q

ovarian torsion is usually the result of

A

ovarian cyst or tumor

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9
Q

who is at risk for ovarian torsion

A

pre pubertal girls

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10
Q

3 symptoms of O torsion

A

abdo pain
N&V
leukocytosis

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11
Q

sono apperance of torsion (5)

A
unilateral enlargement
fluid in cul de sac 
cyst/tumor
twisted vascularular medical (wirlpool/target sign)
absence of flow alone is not reliable
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12
Q

hemorrhagic cyst sono appearance (4)

A

heteromass
thick walls
separations
fluid in culldesac

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13
Q

PCOD also called

A

stein leventhal syndrome

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14
Q

clinical features of PCOD (5)

A
Hirsutism
Irregular menstrual bleeding
Associated with obesity and diabetes
increased incidence of endometrial carcinoma
Infertility
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15
Q

mean ovarian volume of PCOD

A

14cc

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16
Q

Follicle size in PCOD

A

0.5-0.8cm

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17
Q

when do most ovarian neoplasms occur

A

at puberty

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18
Q

is ascites common in children with ovarian neoplasms

A

less so than in adults

19
Q

3 catagories of primary tumors

A

germ cell
epithelial cell
stromal cell

20
Q

5 types of germ cell

A
Benign teratoma
Dysgerminoma
Embryonal carcinoma
Endodermal sinus tumors
Choriocarcinoma
21
Q

3 types of epithelial cell

A

Serous and mucinous cystadenoma and cystadenocarcinoma

22
Q

3 types of stromal cell

A

Granulosa theca cell tumor
Arrhenoblastoma
Gonadoblastoma

23
Q

apperance of benign teratoma (4)

A

Predominantly cystic with or without mural nodule
Solid masses
Complex lesions with fat-fluid or hair-fluid levels
Calcifications

24
Q

dysgerminoma description (4)

A

Malignant
Large, solid, encapsulated
Rapidly growing
Hypoechoic areas from hemorrhage, necrosis

25
Q

epithelial group appearance

A

Cystic masses with septa of variable thickness

Often difficult to differentiate on US between these 4 types of tumors

26
Q

granulosa cells are associated with

A

feminizing effects and precocious puberty (estrogen producing)

27
Q

are granulose theca cell tutors malignant or benign

A

benign

28
Q

what cells are gonadoblastomas made out of

A

germ, stromal and sex cord cells

29
Q

are neoplasms of the uterus and vagina common

A

no, but more common in the vagina than uterus

more likely to be malignant than benign

30
Q

highly malignant germ cell tumor of vagina

A

endodermal sinus tumor

31
Q

where do rhabdomyosarcoma arise from and are they malignant

A

uterus or vagina

and yes they malignant

32
Q

clinical presentation of rhabdomyosarcoma (3)

A

Age at presentation 6-18 months old
Vaginal bleeding
Protrusion of polypoid cluster of masses (sarcoma botryoides)

33
Q

4 endocrine abnormalities with primary amenorrhea

A

Gonadal dysgenesis
Chromosomal abnormalities
Decreased hormonal states
Testicular feminization

34
Q

most common form of gonadal dysgenesis

A

turners syndrome

35
Q

turners syndrome description (4)

A

45, XO karyotype
Delayed or absent puberty
Short statue, webbed neck
Renal and CV problems

36
Q

sono of turners syndrome

A

Ovaries may not be seen
Streak ovaries
Prepubertal uterus

37
Q

testicular feminization description

A
Sex-linked recessive abnormality
End-organ insensitivity to androgens
Phenotypic females with 46,XY karyotype
Absent uterus and ovaries
Ectopic testes
38
Q

define precocious puberty

A

Development of secondary sexual characteristics, gonadal enlargement, & ovulation before age 8 yrs

39
Q

other descriptors of precocious puberty

A

Uterus enlarged with postpubertal shape
fundus/cx ratio - 2:1 to 3:1
Prominent endometrium
Ovarian volume > 1cc, with functional cysts

40
Q

central type precocious puberty description

A

True precocious puberty
Gonadotropin dependent
increased FSH & LH
increased Estrogen

41
Q

cause of central type precocious puberty

A

idiopathic

sometime intracranial tumor

42
Q

peripheral type precocious puberty description

A

Pseudoprecocious puberty
Gonadotropin independent
increase Estrogen,
decrease FSH, LH

43
Q

cause of peripheral type precocious puberty

A

ovarian tumor