Cardiac Conduction, Calcium Channel Blockers, Anti-Dysrhythmic Drugs Flashcards

1
Q

What has to happen for the heart to be able to pump

A

Electrical simulation

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2
Q

What does the electrical acticity of the heart cause

A

Contraction and blood flow

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3
Q

What are the 4 properties of cardiac tissue

A

Auto/conduc/exicitab/contractility

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4
Q

What is automaticity

A

Ability to initiate an impulse

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5
Q

Where does the pulse start from

A

SA node

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6
Q

What do cardiac cells do to act as a syncytium

A

Work as one

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7
Q

Can there be spontaneous depolarization of the cardiac cells

A

Yes

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8
Q

What is conductivity

A

Ability for an impulse to move along, SPREAD from cell to cell

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9
Q

What is excitability

A

Ability to be elctriclaly stimulated

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10
Q

What is contractility

A

Ability to respond mechanically to an impulse, STRETCH

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11
Q

What is the polarized state

A

Rest, K inside: Ca and Na outside

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12
Q

What is cell depolarization

A

Contraction

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13
Q

What is cell repolarization

A

going be to polarization

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14
Q

What is the hearts main pacemaker

A

sa NODE

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15
Q

What is the normal bpm from the SA node

A

60-100

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16
Q

What is the AV responsible for

A

The delayed impulse which prevents the ventricle from contracting too quickly, allows for better STRETCH

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17
Q

What is the stretch law

A

Starlings law

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18
Q

What does the bundle of HIS do

A

Repid impulse down to the ventricles

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19
Q

What are the purkinje fibers

A

They depolarize the cells

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20
Q

What are the two types of actoin potential channels

A

Faster and slower

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21
Q

What are ECGs used for

A

Dysrhythmia, MI, effects of MEDS

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22
Q

What should you remeber about lead placment

A

white on right, snow over grass

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23
Q

What is a dysrhythmia (arrhythmia)

A

Abnormality in the rhythm of the heartbeat

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24
Q

What do dycrhythmias arise from

A

Impulse formation disturbances

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25
What are the two fundamental causes of dycrhythmias
Distrunances of automaticity or conduction
26
What are some examples of disturbances of conduction
AV block or reentry (circular motion)
27
What is A-fib
no definded P-wave, irregular ventricle rythm of the QRS complex
28
What is PAC
premature atrial contarction
29
What is V-fib
No pulse, blood is not pumping out because the ventricle is not pulsating correctly
30
What is V-tach
Saw tooth, big issue
31
What are the properties of dysrhythmias
Sustatained, symptomatic, above or in the ventricle, acute or long term
32
What are the symtpoms of a-fib
Increased HR, SOB, feeling of fluttering
33
What is the type of treatment you will use for long term care
Beta-blockers to decrease HR, anti-coag, because with a-fib the blood will not move well through the artria
34
What do beta-blockers end with
olol
35
How do beta blockers work
Decrease automaticity of the SA node and the time of AV node
36
What are the adverse affects of beta blockers
AV block, bronchospasm in ASTHMA pts
37
What do potasisum channel blockers end in
ONE
38
What is the potassium channel blocker
Amiodarone
39
How do potassium channel blockers work
IN emergencys, V-fib/tach
40
What are you going to do when you give your pt amiodarone
MONITOR, AV block
41
What are the drug interactions of potassium channel blockers
Increases levels of diltiazem, digoxin, warfrin, other beta-blockers
42
What should you keep in mind about potassium channel blockers
no grapefruit (increase), or cholestyramines (reduces), careful with dirutectics (potassium levels off)
43
What do calcium channels do
regulate entry of calcium into the cells
44
What will happen if calcium channels are blocked
Vasodilation, contraction will not happen
45
What happens if calcium channels are open
Increases the force of contraction and increases the SA node
46
What are the three subclasses of calcium channel blockers
Phenylakylamines, benzothiazepines, diyhydropyridines
47
What is the drug for phenylakylamines
Verapamil
48
What is the drug for benzothiazepines
Diltiazem
49
What is the drug for dihydropyridines
Nifedipine
50
Which drugs work of the arteriles and the heart
Verapamil and diltiazem
51
What drug jsut works on the arteriles
BNifedipine
52
What is the nomonic to rembere the drugs of calcium channel blockers
Very Nice Drugs Verapamil, Nifedipine, Diltiazem
53
What does verapamil and diltiazem do
Reduce SA and AV nodes, slow ventriclular rate, stop SVT
54
What are verapmil and diltiazem used for
BP and dsrythmias
55
What are the adverse effects of verapamil and diltiazem
Bradycardia, hypotension, heart block, constipation
56
Can dihydropyridines be used for dysrhythmias
No
57
What are the adverse effects of nifedipine and amlodipine
peripheral edema in older women
58
What do verapamil and diltiazem act on
Smooth muscle and the heart
59
What are you going to teach your pts about calcium channel blockers
Monitor for slow HR, swelling, constipation
60
What is adenosin used for
Termination of SVT, slowers conduction of AV node, QUICKLY
61
How fast do you push adenosin
FAST push and FAST flush
62
What are the adverse effects of adenosine
Dyspnea, SOB, facial flushing, chest discomfort
63
What are the nursing implications for use of adenosine
O2, hooker up to everything