Antidysrythmics Flashcards

1
Q

where is the origin of supra ventricular tachycardias?

A

the atria

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2
Q

what is the most common type of supra ventricular tachycardia?

A

atrial fibrillation

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3
Q

Why is AV block desirable with supra ventricular dysrhythmias?

A

because less of the underside activity would get transmitted to ventricles

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4
Q

ventricular/atrial cardiac muscle cells depend on?

A

sodium and potassium

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5
Q

action potential of ventricular/atrial muscle cells start with ______ and end with _______

A

start with sodium influx and end with potassium efflux

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6
Q

Class 1 antidysrhythmic drugs

A

sodium channel blockers

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7
Q

Class 2 antidysrythmics

A

beta blockers

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8
Q

Class 3 antidysrhythmics

A

potassium channel blockers and others

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9
Q

Class 4 antidysrthymics

A

calcium channel blockers

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10
Q

class 5 antidysrhythmics

A

adenosine and digoxin

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11
Q

All class 1 antidysrhythmics block?

A

sodium channels and slow depolarization

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12
Q

Class 1A prototype?

A

quinidine

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13
Q

quinidine is used for?

A

acute onset atrial fibrillation

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14
Q

quinidine slows atrial and ventricular rates but?

A

does not affect rhythm

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15
Q

wolff-parkinson-white syndrome is associated with?

A

quinidine

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16
Q

Class 1 B prototype?

A

lidocaine

17
Q

lidocaine is used for ______ only

A

ventricular dysrhythmias

18
Q

class II prototype?

A

metoprolol

19
Q

how does metoprolol work?

A

reduces or blocks SNS stimulation and creates AV block

20
Q

Class III prototype?

A

amiodarone

21
Q

how does amiodarone work?

A

it blocks potassium channels therefore prolonging depolarization and cardiac action potential

22
Q

downside of amioderone?

A

can have very serious adverse effects if used for longer than 6 months.

23
Q

amioderone is commonly used in those who are?

A

resistant to other drugs

24
Q

Prototype for class IV?

A

diltiazem and verapamil

25
Q

Where do diltiazem and verapamil act?

A

on the AV node to inhibit Ca entry and reduce conduction velocity

26
Q

Diltiazem and verapamil are not used for?

A

ventricular dysrhythmias

27
Q

diltiazem and verapamil control?

A

rate for atrial fibrillation and flutter

28
Q

what are the unclassified antridysrhythmics?

A

adenosine and digoxin

29
Q

How does adenosine work?

A

slows conduction through the AV node and causes AV block

30
Q

What is the half life of adenosine?

A

20 to 30 seconds

31
Q

adenosine is only administered as?

A

fast IV push