Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

Endo/Repro > Wk3 STDs > Flashcards

Wk3 STDs Flashcards

1
Q

Urethritis in male (inflammatory, exudative infection

Ddx:

A

Neiseria gonorrhoeae

Chlamydia trachomatis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Cervicitis

Ddx:

A

Chlamydia trachomatis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Vaginitis Ddx:

A

T. vaginalis

C. albicans

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Vaginosis Ddx:

A

Gardnerella

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

HPV

genome?

capsid?

envelopment?

A

dsDNA group I

circular genome

Icosahedral

Nonenveloped

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Cancer causing factor in HPV:

A

episome E6 –> decreased p53 –> accumulation of mutations

episome E7 –> knocks out Rb

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

typically purulent discharge in male:

A

Neisseria gonorrhea

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

typically clear discharge in male:

A

Chlamydia trachomatis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

N. gonorrhoeae virulence factors

A

pilli – prevents phagocytosis

endotoxin

capsule

IgA protease

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Thayer Martin media

A

N. gonorrhoeae

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Glucose oxidizer

A

N. gonorrhoeae

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

G -

dipplococci

oxidase +

A

N. gonorrhoeae

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Tx for N. gonorrhoeae

A

ceftriaxone

+ doxy for probable concurrent Chlamydia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Prophylaxis in neonates from N. gonorrhoeae mother

A

erythromycin eye drops

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

N. gonorrhoeae

gram ?

A

Gram -

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

N. gonorrhoea

Oxidase ?

A

oxidase +

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Complications of untreated N. gonorrhoeae:

A

PID

ectopic pregnancy

sterility

Fitz-Hugh-Curtis syndrome

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

LIver infx from N. gonorrhoeae

A

Fitz Hugh Curtis synd

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

most common cause of septic arthritis in sexually active people

A

N. gonorrhoeae

20
Q

Prevents lasting immunity after N. gonorrhoeae infx:

A

antigenic variation

21
Q

Painful penile ulcers in men:

A

HSV – small blisters

Chanchroid (Haemohylus ducryei) – ugly red

22
Q

Syphilis real name:

A

T. pallidum

23
Q

Painless ulcers

A

T. pallidum

24
Q

Alpha herpes viruses (HSV 1/2)

genome?

capsid?

envelopment?

A

dsDNA group I

LInear genome

Icosahedral

Enveloped

25
Q

Immune control of HSV

A

cell mediated

26
Q

Tzank smear with giant multinucleated cells:

A

HSV

27
Q

Tzank smear without multinucleated giant cells:

A

H. ducryei — aka Chanchroid

28
Q

Cowdry bodies type A

A

HSV

29
Q

Tx for HSV

A

acyclovir

30
Q

MOA for acyclovir

A

activated by viral thymidine kinase

chain terminator

selective toxicity for active viral infx

31
Q

3 likely causes of vaginal (NOT urethral) discharge

A
  1. Gardnerella – white/grey, SMELLY
  2. Candida albicans – itchy, white, small clumps
  3. Trichomonas vaginalis – itchy, yellowish, large amount
32
Q

fishy odor

A

bacterial vaginosis

Gardnerella

33
Q

clue cells

A

bacterial vaginosis

34
Q

flagellated protozoan

A

Trichomonas

35
Q

Tx for trichomoniasis

A

metronidazole

36
Q

Most common cause of vaginitis

A

C. albicans

“yeast infection”

37
Q

Indication that C. albicans is growing rapidly

A

pseudohyphae

38
Q

Mech of disease for C. albicans

A

opportunistic

ie. post abx

39
Q
  1. What cell types does HIV infect, and why does this have such an impact on the patient’s immune response?
  2. How does the virus replicate?
  3. To what other opportunistic infections is this woman susceptible?
  4. What are the risk factors for infection?
  5. How can the infection be treated?
A

Can infect T cells and macrophages. Need CD4, CXCR4, or CCR5.

Reverse transcription.

Many, list some…

Unprotected sexual intercourse, contaminated blood.

HAART. NNRTI + 2 NRTI or PI + 2 NRTI.

40
Q

rash on palms and soles of feet

A

T. pallidum

41
Q

hard chancre at site of pathogen entry

A

T. pallidum

42
Q

systemic invasion

diffuse rash

hair falls out

3 weeks - 6 mo after infx

A

Secondary syphilis

43
Q

latent period up to 20 years

A

tertiary syphilis

44
Q

gummas

A

tertiary syphillis

45
Q

gram -

spirochete

A

T. pallidum

46
Q

Host antigen that has antibodies indicative of syphilis

A

cardiolipin

47
Q

Tx for syphilis

A

Penicillin G

Endo/Repro (46 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions