renal infections exam 1 Flashcards

1
Q

types of Pediatric Genitourinary DysfunctionUrinary Tract Infections?

A

Bladder = Cystitis
Kidney = Pyelonephritis

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2
Q

nursing care for Pediatric Genitourinary DysfunctionUrinary Tract Infections?

A

intake and output
blood pressure
teaching prep of child & family
pain control

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3
Q

UTI in kids?

A

Incidence and type of kidney or urinary tract dysfunction change with age and maturation
Identified with physical exam, family history, child’s history, lab studies, radiographic studies, biopsy, physical assessment
In newborns association between malformation of another system (low-set ears or ear tags)
Uncircumcised males under 3 mo. (20%)
Structure of short urethra = increased bacteremia in females
Stasis (normally sterile)

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4
Q

nursing care for UTI?

A

Fluid intake – flushing = lowered organisms (not prior to collection-dilutes sample)
Cranberry juice prevent attachment of E.Coli to tract walls
UTI in children under 2 yr, associated w/ renal malf. & reflux may have progressive renal injury
Prep family & child developmentally for tests

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5
Q

s/s of UTI?

A

Decreased appetite
Voiding often
Thirsty
Crying with painful void
Fever
Fatigue
Blood in urine
c/o abd pain

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6
Q

how to prevent UTI?

A

Girls should clean from front to back
No restrictive clothing-Cotton underwear only
Avoid bubble baths
Use bathroom when nature calls
Increase fluid intake

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7
Q

CM of Vesicoureteral Reflux?

A

Cystitis moves to pyelonephritis
Residual ureter urine in bladder until next void
High fevers, vomiting, chills

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8
Q

diagnosis of Vesicoureteral Reflux?

A

Cystoscopy or Voiding cystourethrogram

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9
Q

nursing care of Vesicoureteral Reflux?

A

Prevent bacteria reaching kidneys to reduce scarring chance
Low dose abx w/ cultures 2-3 months
Assess for high fevers, vomiting, chills

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10
Q

patho of Obstructive uropathy?

A

Structural or functional = hydronephrosis – acquired or congenital, unilateral or bilateral
With congenital defect consider malformation
Distal nephron damage (chronic uropathy) = urine concentration

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11
Q

nursing care of Obstructive uropathy?

A

Prepare family & child (consider development) {emotional}
Potential discharge home w/ devices
Equipment, fluids (urine flow), clotting, sediment
Possible dialysis or transplant in future

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12
Q

patho and nursing care for external defects?

A

Serious due to psychological impact
Activities & inquisitive nature impede recovery
Surgical repair prior to preschool: children may view procedures as punishment & obsession with genitals (differences and normalcy)

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13
Q

protrusion of abdominal contents into scrotum

A

inguinal hernia

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14
Q

fluid in scrotum

A

hydrocele

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15
Q

narrowing of opening in foreskin

A

phimosis

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16
Q

urethral opening on ventral surface of penis

A

hypospadias

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17
Q

ventral curvature

A

chordee

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18
Q

meatal opening on dorsal surface

A

epispadias

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19
Q

teste(s) undescended

A

cryptorchidism

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20
Q

bladder-open pubic arch-bladder exterior of abd wall

A

exstrophy of bladder

21
Q

genitalia-sex undetermined by visualization (ultrasound to determine)

A

ambiguous genitalia

22
Q

patho of nephrotic syndrome?

A

Predominately 2-7 yrs (rare under 6 mo, uncommon under 1 yr, unusual after 8 yrs)

May be metabolic, biochemical, physiochemical, or immune-mediated disturbance:

basement membrane increasingly permeable to protein - albumin leaks & is lost in urine (reduces serum albumin level), fluid accumulates in interstitial spaces (edema) and body cavities (ascites)

23
Q

etiology of nephrotic syndrome?

A

Massive proteinuria, hypoalbuminemia, hyperlipidemia, and edema
Primary: idiopathic nephrosis, childhood nephrosis, or minimal change nephrotic syndrome (MCNS=80%)
Secondary: after glomerular damage
Congenital: autosomal recessive

24
Q

s/s of nephrotic syndrome?

A

Weight gain
Edema around eyes, scrotum, labia, ankles-especially in the morning
Ascites (massive swelling of abdomen, will look like they are pregnant)
Poor appetite
Elevated blood pressure
Tired
Urine is frothy and decreased in volume

25
therapy goals of nephrotic syndrome?
Reduce excretion of urinary protein & fluid retention in tissues Prevent infection & minimize therapy complications
26
nephrotic syndrome treatment?
Low salt diet Severe cases fluid restriction Edema=diuretics 25% albumin Corticosteriods
27
CMs of acute glomerulonephritis?
Latent period 10 days post pneumo or streptococcal & viral infections Commonly – acute poststreptococcal glomerulonephritis (APSGN) Proteinuria & hematuria 3-4+
28
acute glomerulonephritis nursing care?
Those with normal BP and urine output=home tx Sodium restriction for those with edema and BP Daily wt most useful means of assessing fluid balance Pts w/ edema, HTN and gross hematuria may seize
29
acute glomerulonephritis etiology?
AGN – manifestation of systemic disorder Peak age 6-7 yrs old, uncommon younger than 2 yrs Occurs in boys 2:1 ASO indicates post strep infection Reduced C3 –improvement of disease=risingC3
30
patho of Hemolytic Uremic Syndrome?
Primary site endothelial lining of the small glomerular arterioles-swell and occluded with deposits of platelets and fibrin clots. Red blood cells are damaged moving through partially occluded blood vessels. Damaged cells removed by spleen causing acute hemolytic anemia = thrombocytopenia
31
Hemolytic Uremic Syndrome triad?
anemia, thrombocytopenia, and renal failure
32
Hemolytic Uremic Syndrome CMs?
Primarily in infants 6 mos to 5 yr old Acquired hemolytic anemia, thrombocytopenia, renal injury/failure, and CNS symptoms Associated with e.coli (undercooked ground beef), unpasteurized milk or fruit juice (apple), sprouts, lettuce, salami or drinking-swimming in sewage-contaminated water Begins with gastro or URI
33
Hemolytic Uremic Syndrome treatment?
Hemodialysis or peritoneal dialysis (for those anuric for 24 hrs, or oliguria w/uremia or HTN & seizures) FFP and plasmapheresis, fresh/washed packed cells, remove plasma from blood and replace with FFP Emergency support for family and child
34
Hemolytic Uremic Syndrome prognosis?
Prompt tx = 95% recovery Residual renal impairment 10-50% Complications: chronic renal failure, HTN, CNS disorders
35
wilms tumor patho?
Nephroblastoma-most common malignant renal & intra-abdominal tumor in childhood Peak age 3 yrs No method of identifying gene carriers at this time Slightly favors left kidney-10% both kidneys 7/1 million Caucasian children, 3x more African-American than Asian
36
wilms tumor CMs?
Associated with aniridia (no iris in eyes), hemihypertrophy (1 side of body larger). Beckwith-Wiedemann syndrome (overgrowth syndrome) Metastasis rare Dx with radiographic studies, abd ultrasound, hematologic & biochemical studies, urinalysis
37
nursing care for wilms tumor?
Surgery asap removing tumor (keep encapsulated), affected kidney, and adjacent adrenal gland Prognosis for localized (stage I-II) 90% success May reoccur in lung DO NOT PALPATE Post op at risk for bowel obstruction Family and child support
38
etiology for acute kidney injury (AKI)?
Inability to excrete waste, concentrate urine, conserve electrolytes AKI in response to inadequate perfusion, kidney disease, UT obstruction Chronic (CKI) long standing kidney disease or anomaly Azotemia-accumulation of nitrogenous waste in blood Uremia-retention of nitrogenous products - toxic
39
AKI patho?
Diminished urinary output and lethargy in a child who is dehydrated, is in shock, or has recently undergone surgery should be evaluated for possible AKI Reduced glomerular filtration rate Elevated BUN Significant reductions in renal blood flow
40
AKI CMs?
Oliguria followed by diuresis Water intoxication and hyponatremia risk
41
AKI treatment?
During oliguria: no Na++, CL, K+ Hyperkalemia: oral or rectal Kayexalate HTN: meds, limit fluids and salt Anemia: Hgb below 6g/dL w/PRBC Seizure and cardiac failure precautions
42
AKI nursing care?
Accurate I&O, body wt, electrolyte measurement Limit fluid intake Nutrition (perhaps IV) tx nausea/vomiting Tx anxiety, stress, discomfort
43
etiology of chronic kidney injury (CKI)?
Diseased kidneys maintain normal chemical structure of body fluids under normal conditions Uremia Caused by recurrent UTI, chronic pyelo/glomerulonephritis hereditary disorders, & glomerulonephropathy assoc. w/ anaphylactoid purpura and lupus erythematosus
44
CKI patho?
Asymptomatic w/ minimal biochemical abnormalities Damaged nephrons=kidneys maintain fluid &electrolyte balance
45
s/s of CKI
Tired Pallor (pale/gray skin) Increased B/P (sometimes) Headache Nausea Muscle cramps Anorexia Itching Neurological involvement
46
diet management for CKI?
Most effective means besides dialysis for reducing quantity of waste requiring renal excretion Low in protein, potassium, sodium, phosphorous High in carbohydrates Unrestricted amount of fat Limit fresh fruit and veggies  hyperkalemic High in calcium
47
supplements for CKI?
Rich in folic acid and iron Water soluble vitamin supplements & D Phosphorus can be controlled by giving phosphorus-binding agents, e.g., calcium carbonate (most common) & aluminum hydroxide
48
complications of CKI?
Anemia-HTN-Infections Dialysis and transplant only tx for ESRD
49
Dialysis for CKI physical and emotional challenges?
Physical challenges: Implanting graft, fistula or peritoneal catheter Accessing with needle each time – goal pain free Emotions challenges: Denial-fear-sadness Adolescent independence Body changes Relentless need for tx comes before all else