Random Flashcards
diagnosis of addisons
shory synacthen test
most ocmmon side effect of gliclazide
weight gain
recommended monitoring glucose in tpe 1
at least 4 times a day
first line investigation in suspected primary hyperaldosteronism
plasma renin/ aldosterone ratio
obesity classes
25-29 overweight
30-35- class 1
35-40- class 2
40>- class 3
definitive management of primary hyperparathyroidism
total thyroidectomy
painful diabetic neuropathy management
duloxetine
what should be used in new cass of graves to control symptoms
propanolol
c peptide levels in diabetes
low in type 1
high/ normal n type 2
what anti diabetic drugs are linked to necrotising fascittis of the genittials
sglt2 inhiitprs
most common cause of cushings syndrome
pituitary adenoma
what causes hyperpigmentation- addisons or cushings
addisons
first line test for acromegaly
serum igf-1
big hands
acromegaly
time difference between hhs and dka
hhs comes on in days and dka comes on within hours
what to switch triple therapy for in type 2 if not improving
glp-1 mimetic
hba1c in prediabetes
42-47
hba1c diagnostic of diabetes
48 or above
drugs which can cause weight gain
zides- sulfonylureas
what are utis a side effect of
sglt2 inhibitors
uncommon yet specific feature of graves
pretibial myxoedeoma
what to give in hypoglycaemia with impaired gcs
iv glucose
what should patients wirth addisons be given in case of an adrenal crisis
hydrocortison IM
electrolyte abnormality in cushings
hypokalaemic metabolic alkalosis
how does cushings cause its electrolyte abnormality
due to increased excretion of potassium and hydrogen ions
abnormal blood test causing gynaecomastia
prolactin
most modifiable risk factor in development of thyroid eye disease
smoking
correct rate of insulin to prescribe in dka
0.1/kg/hr
drug to reduce cerebral oedema
dexamethasone
what is de quervains thyroiditis assoc with
raised esr and tender goitre
complication of fluid resus in dka
cerebral oedema
suscpsion of cerebral oedema test
ct
lipid rich core
benign adenoma
target hba1c with sulfonylureas
53
what type of drug is sitagliptin
ddp 4 inhibitor
primary hyperaldosteronism features
hypokalaemia and hypertension
drug causing thrush
sglt2
addisonian crisis blood results
low sodium
high potassium
low glucose
what does parathyroid hromone cause
excretion of phosphate
thiazides and calcium correlation
causes hypercalcaemia
what does kinefelters syndrome cause
high lh and low testosterone
what does over replacement of levothyroxine enhance
risk of osteoporosis
diagnostic for cushings syndrome
t low dose dexamethasone
what should happen to regular insulin rwgime in dka
continue long acting stop short acitng
management of addisons
hydrocortisone and fludrocortison
exogenous insulin overdose signs
low c peptide high insulin in serum
hypogylcaemia treatment if patient is conscious and able to speak
glucogel orally
purpose of high dose dexamethasone
to loaclise source of excess cortisol
how to treat pituitary adenoma
transphenoidal surgery
treatment for toxic nodule
radio active iodine
hyperthyroidism treatment for first trimester of preg
PTU
causes of raised prolactin
the p’s
pregnancy
prolactinoma
physiological
polycystic ovarian syndrome
primary hypothyroidism
phenothiazines, metoclopramide, domperidone
managemtn of primary hyperaldosteronism
spironolactone
what is assoc with carbimazole use
agranulocytosis
initial management of graves
propanolol
diagnosis of diabetes
fasting glucose 7
random 11.1 or above
if asymptomatic then needs done twice
anti diabetic drug causing hypoglycaemia
sulfonylureas
is fludrocortisone requored for addisonian crisis
no
hypercalcaemia on ecg
shortened qr interval as it speeds up repolarisation
management of hypercalcaemia
fluids , bisphosphonates
pneumonic for primary hyperpara
moans groans bones for hypercalcaemia
another word for addisons
primary hyperaldosteronism
what drug may result in gynaecomastia
GNRH agonists- goseriln
what antidiabetic causes hypogylcaemia
sulfonylureas
hypocalacaemia on ecg
prolonged qt interval
