Pulmonary stuff Flashcards

1
Q

Muscles of Inspiration

A

Diaphragm and external intercostals (principles m of inspiration)

Accessory m of inspiration:
SCM, scalenes, pec major, pec minor, serratus anterior

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2
Q

Muscles of Expiration

A

Passive exhale - passive recoil from lungs and rib cage.

Forceful exhale - rectus abdominus, external oblique, internal oblique, and TA

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3
Q

Lobes of lungs

A

R: 3 lobes (upper, middle, lower)

L: 2 lobes (upper and lower)

Main bronchi branch – lobar bronchi – segmental bronchi – bronchioles – terminal bronchioles

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4
Q

Lung innervation

A

lungs, trachea, and bronchi innervated by both sympathetic and parasympathetic

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5
Q

Ankle-Brachial Index (ABI)

A
  • Compares systolic BP at the ankle and arm to check for peripheral artery disease
  • Systolic BP in both brachial arteries and both tibialis posterior arteries
  • divide the ankle BP by the brachial BP
    (A/B) = ABI
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6
Q

Interpretation of the ABI

A

> 1.40 = indicates rigid arteries and the need for an US test to check for peripheral artery disease

1.0-1.40 = normal; no blockage

.8 - .99 = mild blockage; beginnings of peripheral artery disease

.4-.79 = Moderate blockage; associated with intermittent claudication during exericse

<.4 = severe blockage (suggests sever peripheral artery disease); may have claudication at rest.

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7
Q

Which of the signs or symptoms would most likely lead a health care provider to perform an ankle-brachial index measurement on a patient?

A

claudication

The ankle-brachial index is an objective measurement that can be used to determine the presence of peripheral arterial disease. Claudication is a symptom commonly reported by patients with peripheral arterial disease due to diminished perfusion to the lower extremities.

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8
Q

Which diagnostic test would be most beneficial to compare the relative perfusion pressure in the upper extremities compared to the lower extremities?

A

ankle-brachial index

The ankle-brachial index (ABI) can be used to provide a ratio of systolic blood pressure of the lower extremity compared to the upper extremity.

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9
Q

Which arteries are used for ABI?

A

UE – brachial artery is the only artery used for assessing upper extremity blood pressure.

LE – the posterior tibial artery is most commonly used, though the dorsalis pedis artery is an acceptable alternative

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10
Q

Which ankle-brachial index measurement is most consistent with non-compressible vessels?

A

1.40

An ankle-brachial index (ABI) above 1.4 would be indicative of non-compressible or rigid arteries. An ultrasound may be recommended to further assess for peripheral artery disease.

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11
Q

Physiological control of breathing (how is it controlled?)

A

through central respiratory center in the brainstem (medulla) and peripheral receptors in the lungs, airways, chest wall, and blood vessels

  • Central chemoreceptors (in medulla)
  • Peripheral chemoreceptors (in cartoid bodies)
  • Mechanoreceptors (inhibit m activity when the force of the contraction reaches injurious levels)
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12
Q

Central chemoreceptors

A
  • In medulla
  • Respond to ↑ in the partial pressure of CO2 and hydrogen ion concentration = ↑ ventilation
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13
Q

Peripheral chemoreceptors

A
  • In carotid bodies
  • Respond to hypoxemia by ↑ ventilation
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14
Q

Tracheal and bronchial sounds

A

Tracheal
* Loud, tubular sounds normally heard over the trachea
* Inspiratory phase is shorter than the expiratory phase and there is a slight pause between them.

Vesicular breath sounds
* High pitched, breezy sounds heard over the distal airways in healthy lung tissue.
* Inspiratory phase is longer than expiratory phase and there is no pause between them.

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15
Q

Crackle (formerly rales)

A
  • An abnormal, discontinuous, high-pitched popping sound heard more often during inspiration. May be associated with restrictive or obstructive respiratory disorders.
  • Typically represents the movement of fluid or secretions during inspiration (wet crackles) or occurs from the sudden opening of closed airways (dry crackles).
  • Crackles that occur during the latter half of inspiration typically represent atelectasis fibrosis, pulmonary edema or pleural effusion.
  • Crackles due to the movement of secretions are usually low-pitched and can be heard during inspiration and/or expiration.
    *Pulmonary edema may produce fine crackles as air bubbles through fluid in the dist small airways.
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16
Q

Pleural Friction Rub

A
  • Dry, crackling sound heard during both inspiration and expiration.
  • Occurs when inflamed visceral and parietal pleurae rub together.
17
Q

Rhonchi

A
  • Continuous low-pitched sounds described as having a “snoring” or “gurgling” quality that may be heard during both inspiration and expiration.
  • Caused by air passing through an airway which is obstructed by inflammatory secretions or liquid, bronchial spasm or neoplasms in the smaller or larger airways.
18
Q

Wheeze

A
  • Continuous “musical” or whistling sound composed of a variety of pitches.
  • Heard during both inspiration and/or expiration, but variable from minute to minute and area to area.
  • Arise from turbulent airflow and the vibrations of the walls of small airways due to narrowing by bronchospasm, edema, collapse, secretions, neoplasm or foreign body.
19
Q

Voice Sounds

A
  • In the normal lung, transmission of spoken sounds is usually muffled; whispered words are faint and the syllables are not distinct, except over the main bronchi.
  • Increases in loudness and distinctness indicate consolidation, atelectasis or fibrosis, all of which improve transmission of vibrations through lung tissue.
  • Whispered and spoken voice sounds are somewhat more valuable than breath sounds in detecting pulmonary consolidation, infarction, and atelectasis.
  • Bronchophony. Increased vocal resonance with greater clarity and loudness of spoken words (e.g., “99*).
  • Egophony. A form of bronchophony in which the spoken long “E” sound changes to a long, nasal-sounding “A.”
  • Whispered pectoriloquy Recognition of whispered words *1, 2, 3.
20
Q

Stridor

A
  • Continuous high-pitched wheeze heard with inspiration or expiration.
  • Indicates upper airway obstruction.
21
Q

Tidal volume (TV):

A

volume of gas inhaled (or exhaled)
during a normal resting breath.

~10% of TLC ~500 mL

22
Q

Inspiratory reserve volume (IRV):

A

volume of gas that can be inhaled beyond a normal resting tidal inhalation.

55-60% TLC

23
Q

Expiratory reserve volume (ERV):

A

volume of gas that can be exhaled beyond a normal resting tidal exhalation.

24
Q

Residual volume (RV):

A

volume of gas that remains in the lungs after ERV has been exhaled.

~25% of TLC or 1,000mL

25
Q

Inspiratory capacity (IRV + TV):

A

the amount of air that can be inhaled from the resting end-expiratory position (REEP).

26
Q

Vital capacity ([RV + TV + ERV):

A

the amount of air that is under volitional control; conventionally measured as forced expiratory vital capacity (FVC).

27
Q

Functional residual capacity (ERV + RV):

A

the amount of air that resides in the lungs after a normal resting tidal exhalation.

~40% of TLC

28
Q

Total lung capacity (IRV + TV + ERV + RV):

A

the total amount of air that is contained within the thorax during a maximum inspiratory effort.

4000-6000mL

29
Q

Forced expiratory volume in 1 second (FEV1):

A

the amount of air exhaled during the first second of FVC. In the healthy person, at least 70% of the FVC is exhaled within the first second (FEV1/FVC × 100%>70%).

30
Q

Forced expiratory flow rate (FEF 25%-75%)

A

is the slope of a line drawn between the points 25% and 75% of exhaled volume on a forced vital capacity exhalation curve. This flow rate is more specific to the smaller airways and shows a more dramatic change with disease than FEV1.

31
Q

Active cycle breathing

A
  • Diaphragmatic breathing for 5-10 sec (aka breathing control)
  • Thoracic expansion
  • Diaphragmatic breathing for 5-10 sec
  • Thoracic expansion
  • Diaphragmatic breathing for 5-10 sec
  • Forced expiration technique (huffing 1-2 time)
  • Diaphragmatic breathing
32
Q

Pursed lip breathing

A

To help decrease the respiratory rate (to improve gas exchange)

Also to help prevent airway (alveolar) collapse due to positive pressure

Dyspnea at rest or with exertion and/or wheezing

wording from PTFE test
(Create back-pressure in the airways to ease airflow)

33
Q

Huffing

A

to mobilize secretions

Exhaling through an open mouth (steam mirror)

34
Q

Inspiratory hold technique

A

prolonged holding of breath with Valsalva. Inflates poor ventilated area of lungs and improved cough

For hypoventilation, atelectasis, and improved effective cough

35
Q

Stacked breathing

A

Take a breath and hold it, breathe in more and hold it, breathe in more and hold it and then let it out when there is no more.

Helps open distal airways

36
Q

Segmental breathing (aka lateral costal breathing)

A

tactile cues to help at the ribs and diaphragm.

Helps with asymmetrical expansion, consolidated lunges or secretions, or poor posturing.

A question on the PTFE said cuing for more exhaling.

37
Q

Paced breathing

A

Low endurance, dyspnea on exertion, fatigue, anxiety, tachypnea

38
Q

Biots (ataxic) breathing

A

Ataxic breathing is characterized by unpredictable irregularity. Breaths may be shallow or deep and stop for short periods

From brain damage typically at the brainstem/medulla level

39
Q

Cheyne Stoke breathing

A

gradual increase in depth of respirations followed by gradual decrease and then a period of apnea

Hypo perfusion of the brain

Seen at end of life.